Sentences with phrase «onset of adolescence»

With the onset of adolescence, teens spend less time with family and more time with peers.

But for Daisy, the middle child, John's absence opens up a world of bewilderment, exposing her at the onset of adolescence to the chaos and instability that hover just beyond the safety of parental love.

The onset of adolescence can play havoc with students» academic performance, as heightened social pressures, bullying and a changing sense of self - identity make it harder for kids to focus on a book report or math homework.

We know the onset of adolescence is a peak time for bullying, when power relationships start to be established and particularly when this change is associated with transition from primary to secondary education and earlier friendships break up and social groups change.

Whether you are child in Baltimore, Beijing, Nairobi or New Delhi, the onset of adolescence triggers a surprisingly common set of rigidly enforced gender expectations that are linked to increased lifelong risks of everything from HIV and depression to violence and suicide.

Whether you are child in Baltimore, Beijing, Nairobi or New Delhi, the onset of adolescence triggers a surprisingly common set of rigidly enforced gender expectations.

Onset of adolescence is usually accompanied with sudden and rapid growth in height, weight and size, with physical strength increasing greatly.

Not exact matches

«Adolescence is a critical risk period for initiation of alcohol use, and earlier onset of drinking is associated with greater risk of developing alcohol abuse and dependence,» said Dr. Gale Burstein, Erie County Commissioner of Health.

Studies have found elevated rates of autism among young people with childhood - onset schizophrenia, in which the features of schizophrenia appear before age 13 rather than in late adolescence.

One current research focus is measuring differences in EEG smoking cue reactivity, as well as neurochemical and morphological alterations in adult brain architecture, related to age of smoking onset during adolescence.

There have been hundreds of theories about schizophrenia over the years, but one of the enduring mysteries has been how three prominent findings related to each other: the apparent involvement of immune molecules, the disorder's typical onset in late adolescence and early adulthood, and the thinning of gray matter seen in autopsies of patients.

Onset of symptoms of CMT is most often in adolescence or early adulthood, however presentation may be delayed until mid-adulthood.

We propose that the discrepancies between the adult ASD and childhood ASD findings with respect to whole - brain functional connectivity may be reconciled by considering critical developmental factors such as the onset of puberty, which signals the beginning of adolescence and has a major impact on brain structure and function.

The peak age of onset for many psychiatric disorders is adolescence, a time of remarkable physical and behavioural changes.

As it happens, both young men and women experience a growth spurt in adolescence that is second only to the rapid growth that occurs in the first year of life.2 With the onset of puberty come increases in height, weight and bone mass; cognitive changes; and reproductive maturation.3 Adolescent boys gain more in bone size and mass than adolescent girls.3 To support this intensive and multifaceted period of growth, the total nutrient needs of adolescents are higher than at any other life stage.

So someone diagnosed at age 65 with late onset diabetes is less of a risk for life insurance companies to insure than a 35 - year - old who was diagnosed during adolescence.

The nature of the co-occurrence of multiple antisocial behaviors, including substance use, during adolescence, and the causal factors which contribute to the early onset and maintenance of youth substance use and substance abuse are discussed.

Adolescence can come with unforeseeable challenges that even the best parents might struggle with: the onset of disturbing and unsociable behaviours, painful self - image issues, unhealthy friendships or addictions, and even mental illness.

Two studies have identified groups of girls exhibiting chronically high levels of antisocial behavior across childhood and early adolescence and having an increased risk for continued antisocial behavior.60 In addition, Odgers and several colleagues found that 7.5 percent of all girls between the ages of seven and fifteen displayed an early - onset of offending that persisted into adolescence and that this pattern was similar to boys of the same age.61 Other studies suggest that although strongly aggressive behavior in girls before the age of seven is rare, continuity of offending for such girls may be stronger than that among comparable boys and that such early problem behavior in girls should be considered a significant warning sign of potential future problems.62

Studies have shown that teaching these social and emotional skills to children at the onset of their depression can prevent it from becoming more serious in adolescence and beyond.

Adolescence is a critical period for the development of depression with prevalence rates rising sharply from childhood to early adulthood.1 Many adult depressive disorders have their first onset in adolescence2 with longer episode duration being the strongest predictor of future problems.3 In addition to increasing the risk of later mental health problems, adolescent depression is associated with significant educational and social impairment and is a major risk factor for suicide.1 Providing effective early interventions to shorten the duration of episodes and potentially reduce the impact on later life is therefore important.3 This study explores this question and compares the effects of...

Some observers have argued that female offenders can, in theory, be either adolescent - limited or life - course - persistent and that the relative scarcity of early - onset aggression in females indicates that they are generally less likely to follow the latter pathway.56 Others, however, have argued that the relative prevalence of adolescent - onset aggression in girls (compared with childhood - onset) indicates that persistent delinquency simply manifests at a later age in girls than it does in boys.57 In Persephanie Silverthorn and Paul Frick's model, girls and boys are influenced by similar risk factors during childhood, but the onset of delinquent behavior in girls is delayed by the more stringent social controls imposed on them before adolescence.

PTSD in these women often had its onset in adolescence, was debilitating and chronic, and was complicated by the presence of other mental health conditions such as depression, psychosis and alcohol and cannabis dependence.

It may be possible to develop improved interventions for individuals who are at high risk for suicide by identifying combinations of risk factors that are associated with the onset of suicidal behavior.2 Prospective epidemiological research can facilitate the identification of such patterns by assessing a wide range of childhood adversities, interpersonal difficulties during middle adolescence, and suicidal behavior during late adolescence and adulthood.

Conclusions About half of Americans will meet the criteria for a DSM - IV disorder sometime in their life, with first onset usually in childhood or adolescence.

Substance use disorders emerged in middle adolescence and increased in frequency through the middle 20s, becoming by far the most common psychiatric problems reported by the study participants.26, 27 We have already shown that early conduct problems predicted the onset of adolescent substance use disorders in this sample, 28,29 and it is not surprising that this is the aspect of behavioral problems that showed the intervention effect in young adulthood.

Sexual abuse and physical abuse were the strongest risk factors for the onset and persistence of suicidal behaviours, with the risk for suicidality being greatest during childhood (age 4 — 12 years) and adolescence (age 13 — 19 years).20

Many parents relax their restrictions regarding R - rated movies during adolescence, but our results suggest that continued restriction is an effective means of reducing adolescents» risk for smoking onset.

Conclusions Childhood adversities, especially sexual abuse, physical abuse and parental divorce, are important risk factors for the onset and persistence of suicidal behaviour, with this risk being greatest in childhood and adolescence.

ATTENTION - DEFICIT / HYPERACTIVITY DISORDER IN ADOLESCENCE PREDICTS ONSET OF MAJOR DEPRESSIVE DISORDER THROUGH EARLY ADULTHOOD.

Children with early - onset conduct problems (CPs) are at high risk for chronic antisocial and aggressive behaviour, and a variety of social and mental health problems in adolescence and adulthood (e.g. delinquency, psychiatric disorders, substance use, school dropout; Fergusson et al. 2005; Kratzer and Hodgins 1997).

The onset of mental illness (typically during adolescence) can amplify symptoms of attachment disorder.

The first year of life is a period of rapid development critical to infants» health, emotional well - being and developmental trajectories.1, 2 The first signs of mental health problems are often exhibited during infancy; however, the symptoms may be overlooked by parents and healthcare providers because they can be less intrusive when a child is young.3 — 8 Early onset of emotional or behavioural problems increases the risk of numerous adverse outcomes that persist into adolescence and adulthood, such as delinquency, violence, substance abuse, mental health problems, teen pregnancies, school dropout and long - term unemployment.1, 2, 4, 9 — 14

Conclusions The multiple disadvantages predicted by childhood ADHD well into adulthood began in adolescence, without increased onsets of new disorders after 20 years of age.

Children with early onset of either internalizing problems such as anxiousness and withdrawn behavior (e.g., [4]-RRB- or persistent externalizing problems such as defiant and disruptive behavior (e.g., [5 — 7]-RRB- are also at higher risk for continued severe and debilitating mental health problems during adolescence and adulthood [8, 9].

Clinical presentation and course of depression in youth: Does onset in childhood differ from onset in adolescence?

«Targeted supports at key points in childhood and adolescence help prevent the onset of those behavioural problems that lead to underachievement and marginalisation.

Furthermore, prospective, longitudinal research has demonstrated that disorganized and avoidant attachment in early childhood, as well as age of onset, chronicity, and the severity of abuse all predicted dissociation in various developmental stages, up to late adolescence (Ogawa et al., 1997).

Given their typical age of onset, a broad range of mental disorders are increasingly being understood as the result of aberrations of developmental processes that normally occur in the adolescent brain.4 — 6 Executive functioning, and its neurobiological substrate, the prefrontal cortex, matures during adolescence.5 The relatively late maturation of executive functioning is adaptive in most cases, underpinning characteristic adolescent behaviours such as social interaction, risk taking and sensation seeking which promote successful adult development and independence.6 However, in some cases it appears that the delayed maturation of prefrontal regulatory regions leads to the development of mental illness, with neurobiological studies indicating a broad deficit in executive functioning which precedes and underpins a range of psychopathology.7 A recent meta - analysis of neuroimaging studies focusing on a range of psychotic and non-psychotic mental illnesses found that grey matter loss in the dorsal anterior cingulate, and left and right insula, was common across diagnoses.8 In a healthy sample, this study also demonstrated that lower grey matter in these regions was found to be associated with deficits in executive functioning performance.

This pathway is characterized by three elements: the onset of conduct problems (such as developmentally excessive levels of aggression, noncompliance, and other oppositional behaviour) in the preschool and early school - age years; a high degree of continuity throughout childhood and into adolescence and adulthood; and a poor prognosis.1, 2 The most comprehensive family - based formulation for the early - starter pathway has been the coercion model developed by Patterson and his colleagues.3, 4 The model describes a process of «basic training» in conduct - problem behaviours that occurs in the context of an escalating cycle of coercive parent - child interactions in the home, beginning prior to school entry.

This is unfortunate, considering that early onset is one of the strongest identified risk factors for substance use problems in later life (Breslau et al. 1993; Chen et al. 2005; De Wit et al. 2000) and these personality predispositions may play a particularly important role in explaining risk behavior and receptivity for substance use during the period of adolescence (e.g., Carver et al. 2009; Malmberg et al. 2010b).

Considering those who develop a SUD, the peak age of onset is in later adolescence or young adulthood, with less common onset after age of 25 [15].

The externalizing pathway highlights the underlying neurological systems of increased reward sensitivity and reactivity combined with decreased effortful control and regulation that are imbalanced and competing during adolescence (i.e., dual systems model), which further contributes to adolescence being a high - risk period for the onset of substance use and comorbid behavior problems [80, 81].

However, some individuals have onset of SUD early in adolescence, develop multiple SUD diagnoses, and have severe persistent courses [1, 2].

IBD onset during adolescence was associated with reports of increased somatic complaints, t (47) = 2.38, p <.05, but no other differences in current adjustment (see Table II).

[jounal] Monroe, S. M. / 1999 / Life events and depression in adolescence: Relationship loss as a prospective risk factor for first onset of major depressive disorder / Journal of Abnormal Psychology 108: 606 ~ 614

The onset of a chronic illness during adolescence likely has very different implications for development than earlier onset (Spirito, DeLawyer, & Stark, 1991).

Previous research has suggested that parental and child functioning factors are the most significant predictors of early onset risk behavior (i.e., prior to adolescence), as opposed to more distal factors such as the neighborhood environment and social status among peers (Kaplow et al. 2002).

In their taxonomy of antisocial behavior, Moffitt (1993) and Moffitt et al. (2002) demonstrated that two prototypes of offenders, the life - course - persistent (LCP: childhood onset and continuing into adulthood) and adolescent - limited group (AL: beginning in adolescence and desisting in young adulthood), account for the bulk of antisocial behavior (two additional groups were identified and labeled abstainers and low - level chronics, but these groups were relatively small, i.e., around 5 % and 8 % respectively).

Early - onset persistent conduct problem (EOP CP) children are defined as having an onset of conduct problems (fighting, lying, stealing) before the age of 10 years, that persist through adolescence (Moffitt 2006).