These childhood externalizing behavioral problems may express an underlying individual vulnerability toward risk behavior and thereby appear to be the most salient indicator for an increased risk of early
onset risk behavior, including substance use.
Previous research has suggested that parental and child functioning factors are the most significant predictors of early
onset risk behavior (i.e., prior to adolescence), as opposed to more distal factors such as the neighborhood environment and social status among peers (Kaplow et al. 2002).
This study demonstrates that the development of substance use is correlated with externalizing problems and underscores the theory that etiologies of early and later
onset risk behavior are different.
Not exact matches
Risk factors for suboptimal infant breastfeeding
behavior, delayed
onset of lactation, and excess neonatal weight loss.
Information about the
onset and patterns of
risk behaviors associated with overweight and obesity during infancy are showcased in the link below.
Risk Factors for Suboptimal Infant Breastfeeding
Behavior, Delayed
Onset of Lactation, and Excess Neonatal Weight Loss.
Potential impacts of climate change on the transmission of Lyme disease include: 1) changes in the geographic distribution of the disease due to the increase in favorable habitat for ticks to survive off their hosts; 85 2) a lengthened transmission season due to earlier
onset of higher temperatures in the spring and later
onset of cold and frost; 3) higher tick densities leading to greater
risk in areas where the disease is currently observed, due to milder winters and potentially larger rodent host populations; and 4) changes in human
behaviors, including increased time outdoors, which may increase the
risk of exposure to infected ticks.
In this direction, some recent studies investigated that children and adolescents who attended educational programs focused on the promotion of self - efficacy in life skills reduced the
onset of at -
risk and maladaptive
behaviors (Griffin et al., 2003; Botvin & Griffin, 2004; Yankah & Aggleton, 2008; Menrath et al., 2012; Jegannathan, Dahlblom, & Kullgren, 2014): it was possible to observe a significant and positive effect for the reduction of health -
risk behaviors in the intervention group, compared to control group (see Menrath et al., 2012), confirming the efficacy of school - based on life skills programs.
Multiple factors reportedly increase the
risk of suicide.44 - 49 Substance abuse has repeatedly been associated with suicidal
behaviors, and depression has as well.1,50 - 62 Moreover, previous reports from the ACE Study have demonstrated strong, graded relationships between the number of adverse childhood experiences and the
risk of alcohol or illicit substance abuse and depressive disorders.23, 24,28 Although a temporal relationship between the
onset of substance abuse or depressive disorders and lifetime suicide attempts in the ACE Study cohort is uncertain, our analysis of the potential mediating effects of these known
risk factors provides evidence that for some persons, adverse childhood experiences play a role in the development of substance abuse or depression.
Notwithstanding these gender - specific
risk and protective factors, in most cases, the same factors — ADHD, negative temperament, impulsivity, compromised intelligence — predict antisocial
behavior in both males and females, as suggested by the substantial overlap shown in figure 4.99 Although some analysts have argued the need to concentrate on the commonalities in predictors of male and female offending, it is also important to note the areas in which
risk factors differ by gender.100 Even if the differences between male and female offenders are confined to only a few key areas, the differences in these areas — for example, sensitivity to victimization, timing of
onset of persistent offending, prevalence of mental health problems — can be substantial and can profoundly influence the effectiveness of
risk assessments and treatment programs.
Two studies have identified groups of girls exhibiting chronically high levels of antisocial
behavior across childhood and early adolescence and having an increased
risk for continued antisocial
behavior.60 In addition, Odgers and several colleagues found that 7.5 percent of all girls between the ages of seven and fifteen displayed an early -
onset of offending that persisted into adolescence and that this pattern was similar to boys of the same age.61 Other studies suggest that although strongly aggressive
behavior in girls before the age of seven is rare, continuity of offending for such girls may be stronger than that among comparable boys and that such early problem
behavior in girls should be considered a significant warning sign of potential future problems.62
Some observers have argued that female offenders can, in theory, be either adolescent - limited or life - course - persistent and that the relative scarcity of early -
onset aggression in females indicates that they are generally less likely to follow the latter pathway.56 Others, however, have argued that the relative prevalence of adolescent -
onset aggression in girls (compared with childhood -
onset) indicates that persistent delinquency simply manifests at a later age in girls than it does in boys.57 In Persephanie Silverthorn and Paul Frick's model, girls and boys are influenced by similar
risk factors during childhood, but the
onset of delinquent
behavior in girls is delayed by the more stringent social controls imposed on them before adolescence.
On the other hand, parental expectations regarding school achievement were associated with lower levels of health
risk behaviors; parental disapproval of early sexual debut was associated with a later age of
onset of intercourse (P <.001).
It may be possible to develop improved interventions for individuals who are at high
risk for suicide by identifying combinations of
risk factors that are associated with the
onset of suicidal
behavior.2 Prospective epidemiological research can facilitate the identification of such patterns by assessing a wide range of childhood adversities, interpersonal difficulties during middle adolescence, and suicidal
behavior during late adolescence and adulthood.
In addition, it may be possible to prevent the development of suicidal
behavior and other psychiatric symptoms by helping parents of at -
risk youths to modify their child - rearing
behavior.50 Because parental psychiatric disorders are associated with maladaptive parenting and offspring suicidality, it may also be possible to prevent the
onset of suicidal
behavior by improving the recognition and treatment of parental psychiatric disorders.
A range of childhood psychosocial
risk factors have been associated with depression, including characteristics of the child (eg, behavioral and socioemotional problems, poor school performance), characteristics of the parents (eg, parent psychopathology, rejecting or intrusive
behavior), and family circumstances (eg, the loss of a parent, physical or sexual violence, family discord).12 - 15 However, it has not been shown decisively whether these
risks distinguish juvenile from adult -
onset MDD.
Psychological characteristics include low IQ, impulsivity, hyperactivity, lack of empathy, and fearlessness.12, 13 Parental
risks include low levels of education, antisocial
behavior, poor parenting skills, maternal early
onset of childbearing, and family discord.14 — 20 There is evidence of an intergenerational transmission of these problems through both genetic and environmental channels.18, 19,21 — 24 Developmental research also shows that the spontaneous
onset of physical aggression in school - aged children is highly unusual.1, 7,25 Instead, the developmental precursors of chronic physical aggression are present before school entry.
Parental abuse,
onset of problem
behavior in early childhood, financial hardship and lack of supervision are all associated with more severe conduct disorder.10, 18 Additionally, a poorer prognosis is associated with an increase in the number and severity of specific DSM - IV criteria.10
Risk also increases with comorbid ADHD and substance abuse.10 These dimensions should guide treatment Subclinical conduct disorder symptoms or those of recent
onset may be amenable to physician - parent counseling.
OBJECTIVE: To test the a priori hypothesis that this genetic variant predicts early -
onset antisocial
behavior in a high -
risk sample and further examine the effects of birth weight, an environmentally influenced index of prenatal adversity previously linked to childhood disruptive
behaviors and genotype x birth weight interaction.
CONCLUSIONS: Early -
onset antisocial
behavior in a high -
risk clinical group is predicted by a specific COMT gene variant previously linked with prefrontal cortical function and birth weight, and those possessing the val / val genotype are more susceptible to the adverse effects of prenatal
risk as indexed by lower birth weight.
The relationship between early age of
onset of initial substance use and engaging in multiple health
risk behaviors among young adolescents
Deficient self - regulation plays a key role in the etiology of early
onset disruptive
behavior disorders and signals
risk for chronic psychopathology.
Children with early
onset of either internalizing problems such as anxiousness and withdrawn
behavior (e.g., [4]-RRB- or persistent externalizing problems such as defiant and disruptive
behavior (e.g., [5 — 7]-RRB- are also at higher
risk for continued severe and debilitating mental health problems during adolescence and adulthood [8, 9].
The inability to make advantageous decisions under conditions of
risk is associated with health -
risk behaviors such as early -
onset alcohol and drug use.
The five
risks at Wave 1 were never used a condom (10 %), drinking during either first or most recent sex (15.5 %), using drugs during either first or most recent sex (7.9 %), ever has sex for drugs or money (2.5 %), and early
onset of sexual activity [< 13 years old, 11.6 % (age from the Center for Disease Control and Prevention's Youth
Risk Behavior Survey, Brener et al., 2002)-RSB-.
This is unfortunate, considering that early
onset is one of the strongest identified
risk factors for substance use problems in later life (Breslau et al. 1993; Chen et al. 2005; De Wit et al. 2000) and these personality predispositions may play a particularly important role in explaining
risk behavior and receptivity for substance use during the period of adolescence (e.g., Carver et al. 2009; Malmberg et al. 2010b).
Further, understanding how baseline sexual
risk interacts with stability and change in the family — peer mesosystem enhances interpretability of the sexual
risk outcome as being either
onset of
risk behavior, termination of
risk behavior, or sustained
risk behavior (Joiner, 1994).
The externalizing pathway highlights the underlying neurological systems of increased reward sensitivity and reactivity combined with decreased effortful control and regulation that are imbalanced and competing during adolescence (i.e., dual systems model), which further contributes to adolescence being a high -
risk period for the
onset of substance use and comorbid
behavior problems [80, 81].
The objective of the present study was twofold: first, using latent transition analysis (Lanza et al. 2009), we tested whether the relevant groups found in previous studies (e.g., Flory et al. 2004; Moffitt et al. 2002), i.e., those with an early
onset, a late
onset, and no
onset or low levels of
risk behavior could be identified, while using a developmental model of a single, underlying construct (constituted by tobacco, alcohol and marijuana use and externalizing
behavior problems).
First, it was tested whether the relevant groups found in previous studies i.e., those with an early
onset, a late
onset, and no
onset or low levels of
risk behavior could be identified, while using a developmental model of a single, underlying construct of
risk behavior.
All research questions were addressed by analyzing data from a large (n = 2,230), population based cohort study of Dutch adolescents, starting at age 11 with two follow - ups at the mean ages of 13.6 and 16.3 years, thereby capturing the majority of
onset and continuation of adolescent
risk behaviors.
The life - course persistent antisocial group was characterized by high -
risk childhood backgrounds (e.g., inadequate parenting, neurocognitive problems, and temperament and
behavior problems) and appeared to be at highest
risk for adverse outcomes in adulthood, whereas the adolescent
onset group was considered to be quite normative, and thus at much lower
risk for such adverse outcomes (Moffitt and Caspi 2001; Moffitt et al. 2002; Odgers et al. 2008).
Risk factors and life processes associated with the
onset of suicidal
behavior during adolescence and early adulthood