Nonetheless, the data reveal that ownership of a cat is not necessary to acquire T. gondii, because it has been shown that
oocyst exposure is not always associated with cat ownership or with recognition of risk factors.
Not exact matches
However, the researchers did find a threefold higher risk of
exposure for otters living near the mouths of rivers and streams, suggesting that storm - water runoff from fields and lawns frequented by cats could be a source of
oocysts.
Lafferty started out believing from review of previous published literature that «infectious disease was preventing the recovery of sea otters, and the most likely source of infection of Toxoplasma gondii was terrestrial runoff containing
oocysts defecated by cats,» but when he and colleagues tagged 135 California sea otters in 2009 and followed them for four years, they found that «Counter to expectations, sea otters from unpopulated stretches of coastline,» around Big Sur, are less healthy and more exposed to parasites than city - associated otters,» from the Monterey Bay area, who have more
exposure to fecal matter from cats.
Indeed, as cats increasingly contaminate public areas with T. gondii
oocysts it will become progressively more difficult to avoid
exposure.»
Most
exposures of humans to
oocysts cause no overt disease.