Table salt is the number one source of aluminum in the American diet causing
oxidative damage to brain tissues.
Not exact matches
This metabolic demand makes
brain cells particularly vulnerable
to damage from
oxidative stress, in which reactive oxygen species (ROS), sometimes called free radicals, exert toxic effects on cellular components.
Jianguo Fang and colleagues note that mounting evidence suggests that
oxidative damage to neuronal cells contributes
to the development of diseases that originate in the
brain.
In effect, administration of DHA seemed
to shut down the
oxidative damage that binge drinking usually exerts on the
brain.
Yerba mate was even shown in one study
to reduce the amount and length of epileptic seizures, while reducing
oxidative damage in the
brain.
These two plant medicines have been shown
to decrease
oxidative cellular
damage and increase cellular energy, paramount
to overcoming
brain fog.
In fact, a study published in the prestigious
Brain Journal singled out astaxanthin's potential brain - health benefits and its ability to fight oxidative damage that can lead to mental dec
Brain Journal singled out astaxanthin's potential
brain - health benefits and its ability to fight oxidative damage that can lead to mental dec
brain - health benefits and its ability
to fight
oxidative damage that can lead
to mental decline.
Oxidative damage to the... blood vessels causes arterio - and atherosclerosis (hardening of the arterioles and arteries) leading
to heart disease and stroke;
brain causes Alzheimer's and Parkinson's disease; eyes causes cataracts and macular degeneration; skin causes wrinkles, sagging and «age spots»; connective tissue causes faulty collagen and elastin formation, possibly leading
to tendonitis / osis, and other musculoskeletal complications and injuries; etc..
Glycated proteins and cellular structures have altered function, increased vulnerability
to oxidative damage, and reduced degradation and clearance.6 Slowing the entry of glucose into the
brain would delay these processes and possibly give the body's defenses more time
to dispose of the AGEs.
In studies rosmarinic acid has been shown
to increase oxygenation of
brain tissues and help prevent
damage from
oxidative stress.
Scientists are clear that toxic metals
damage brain tissue and lead
to degenerative disease by producing
oxidative stress — and aluminum is one of the worst offenders.
The
brain is vulnerable
to oxidative damage because of its high oxygen consuming metabolic rate.
Homocysteine levels rise with age, and increasing levels can lead
to a
damaging chain reaction of effects — research has revealed that elevated homocysteine levels can disrupt delicate arterial linings, increase inflammation and
oxidative stress, decrease blood flow
to the heart and
brain and set the stage for atherosclerosis and coronary artery disease.
And so when the
brain is using ketones, there is going
to be less free radical
damage and what we call
oxidative stress in the
brain, so less
damage being associated with the various neurons in the
brain therefore better connection, better communication.
It also helped protect against
oxidative damage that leads
to brain cell death, and reduced injury
to brain endothelial cells in mice with high - fat diets.
Researchers from several universities in Australia indicate that curcumin, a natural anti-inflammatory and antioxidant, may help reduce symptoms of MDD by restoring neurogenesis and healthy
brain function, protecting it from
oxidative stress, inflammation, and other types of
damage, which could in turn lead
to improvements in mood.
«Generally,
brain cells have relatively high metabolic activity and use discrete
oxidative damage - repair mechanisms
to remove end - products of intracellular
damage.
Aging in most animals is accompanied by the progressive accumulation of
oxidative (or free radical)
damage to body tissue, including
brain tissue.
This, researchers hypothesized, was consistent with the assumption that
oxidative damage contributes
to brain aging in dogs.
Several candidate mechanisms of CRCI have been proposed, including inflammation and cytokine dysregulation, chemotherapy - induced epigenetic changes, blood —
brain barrier disruption, hormone deficiencies,
oxidative DNA
damage and shortened telomere length, and genetic susceptibility.21 — 26 However, the heterogeneity and design limitations within the emerging body of evidence have led
to recommendations for harmonising study methodologies and moving towards multisite, longitudinal research.3 27 Such approaches would support the demonstration of robust relationships between measurable biological processes and cognitive outcomes.