Sentences with phrase «oxidative processes in»

This type of fatty acid protects the body cells from the effects of radicals and oxidative processes in the body.

Both omega - 3 and omega - 6 fatty acids are important in protecting body cells from destruction of circulating radicals and specific oxidative processes in the body.

Acetyl L Carnitine and Alpha lipoic acid can also prevent any harmful oxidative process in mitochondrial metabolism of penile cells.

Not exact matches

Without giving you the ol' biochemisty textbook definition, antioxidants are substances that counteract the process of oxidative stress in the body and oxidative stress is thought to be behind health issues like cancer, heart disease and other chronic disease in our body.

Now 24, he is a first - year graduate student in the department of cellular and structural biology at the University of Texas Health Science Center in San Antonio (UTHSCSA), where he is studying the role of oxidative damage — the wear and tear inflicted upon the cell by toxic molecules called free radicals — in the aging process.

Mitochondria are found in cells and they use oxygen to produce energy in a process called oxidative metabolism.

Drugs that enhance a process called oxidative stress were found to kill rhabdomyosarcoma tumor cells growing in the laboratory and possibly bolstered the effectiveness of chemotherapy against this aggressive tumor of muscle and other soft tissue.

This results in cells having less mitochondrial DNA, decreased mitochondrial membrane potential and a faulty process in cell metabolism called oxidative phosphorylation.

Mitochondria produce energy for cells through oxidative metabolism, but the process produces toxic byproducts that can accumulate and cause defects in the cell's mitochondria.

Although reactive oxygen species can damage cells when produced in high amounts, according to a study published online Sept. 5, 2017 by Science Signaling and featured on the journal's cover, these oxidative species are crucial signals that start the process of repairing myofiber.

Somatic cells generate their energy in an oxygen - fueled process called oxidative phosphorylation, which takes place in the mitochondria, also known as cellular powerhouses.

Taken together, the study's findings demonstrate that FOX01 plays an integral role in two key processes in wound healing: activation of TGF - β1 and protecting the cell against oxidative damage.

The hydrothermal conditions of the experiments, which also mimic early Earth settings (a proximity to volcanic activity and impact craters), released a complex mixture of oxygen - rich compounds, the probable result of oxidative processes that occurred in the parent body.

Fat cells produce excessive reactive oxygen species, which can damage cells in a process called oxidative stress.

Copper is an essential cofactor for enzymes involved in diverse cellular processes, including oxidative metabolism, neurotransmitter synthesis, free radical detoxification, and iron uptake.

Our work aims to investigate mechanistically the molecular processes through which dysregulation of factors controlling oxidative stress impairs RPE and photoreceptor cell metabolism and their survival in AMD.

«We then showed that this slower energy dissipation process is active in wild - type plants under stress conditions, such as cold and high light, and that it limits oxidative damage to lipids.

More recently, her lab started working on the mechanisms that link oxidative stress and photoreceptor loss and dysfunction, with the goal of identifying ways to target the underlying processes to prevent photoreceptor and RPE loss in dry AMD.

This work aims to investigate, mechanistically, the molecular processes related to dysregulation of factors that normally protect the eye against oxidative stress, thus impairing RPE and photoreceptor cellular metabolism and their survival in AMD.

Review: The role of operating parameters and oxidative damage mechanisms of Advanced Chemical Oxidation Processes in the combat against antibiotic - resistant bacteria and resistance genes present in urban wastewater — I. Michael - Kordatou — Water Research

This DNA becomes damaged in the course of normal cellular processes, and certain forms of mitochondrial DNA damage - to the thirteen genes needed for oxidative phosphorylation - produce malfunctioning mitochondria that can overtake their cells, either by replicating more readily or being more resistant to quality control mechanisms.

Over time however, mutations in these genes occur as a result of constant exposure to reactive oxygen species produced by oxidative phosphorylation, the mitochondrial energy generation process.

Enhanced amyloidogenic processing of APP by the ß - site APP cleaving enzyme (BACE) and the γ - secretase complex and reduced clearance lead to increased intracellular levels of soluble oligomeric Aß, resulting in cellular dysfunction comprising e.g., synaptic failure, mitochondrial dysfunction, enhanced oxidative stress, neurotransmitter and neurotrophin depletion, inflammation, and apoptosis which is reflected in patients as clinical symptoms such as cognitive deficits [2, 3].

Iron is useful for participating in lots of different chemical reactions in the body, as part of normal metabolism, but it's also important for caring oxygen to our tissues and oxygen is what we need in a process called oxidative phosphorylation, which gives us energy.

In fact, oxidative stress is so harmful to the body that many experts think of it as a key factor in the aging process itself.1 Caffeine and its catabolic products, theobromine and xanthine, are all capable antioxidants which are extremely useful to the body in fighting oxidative stressIn fact, oxidative stress is so harmful to the body that many experts think of it as a key factor in the aging process itself.1 Caffeine and its catabolic products, theobromine and xanthine, are all capable antioxidants which are extremely useful to the body in fighting oxidative stressin the aging process itself.1 Caffeine and its catabolic products, theobromine and xanthine, are all capable antioxidants which are extremely useful to the body in fighting oxidative stressin fighting oxidative stress.2

The correlation between oxidative stress and cortisol is basically this: to make steroid hormones, so it could be testosterone, estrogen, cortisol, to make steroids hormones actually generates a lot of free radicals in the process.

Stress resistance has not been assessed however and so the biological relevance of this finding is currently unknown.32 Several IER trials (75 - 85 % ER on restricted days) in overweight / obese populations have reported reductions in various markers of oxidative stress 37, 41, which in one study was accompanied by a complementary increase in the anti-oxidant uric acid.37 In a direct comparison of IER (75 % ER for two days / week) and CER, both ER strategies displayed equal efficacy in reducing levels of fast - acting advanced oxidation protein products (AOPP) after six months, which displayed a tendency to occur earlier (i.e. at three months) in the IER group.41 Levels of slow - acting (i.e. long term) AOPP tended to decrease in the IER group and increase in the CER group which the authors proposed may have resulted from IER - induced activation of autophagy, a key homeostatic cellular process in which dysfunctional or unnecessary cellular proteins are degraded and recycled.41 On the other hand, a follow - up study using similar IER / CER protocols demonstrated comparable reductions in AOPP in both groups after three months.48 Summary and Future Research Directioin overweight / obese populations have reported reductions in various markers of oxidative stress 37, 41, which in one study was accompanied by a complementary increase in the anti-oxidant uric acid.37 In a direct comparison of IER (75 % ER for two days / week) and CER, both ER strategies displayed equal efficacy in reducing levels of fast - acting advanced oxidation protein products (AOPP) after six months, which displayed a tendency to occur earlier (i.e. at three months) in the IER group.41 Levels of slow - acting (i.e. long term) AOPP tended to decrease in the IER group and increase in the CER group which the authors proposed may have resulted from IER - induced activation of autophagy, a key homeostatic cellular process in which dysfunctional or unnecessary cellular proteins are degraded and recycled.41 On the other hand, a follow - up study using similar IER / CER protocols demonstrated comparable reductions in AOPP in both groups after three months.48 Summary and Future Research Directioin various markers of oxidative stress 37, 41, which in one study was accompanied by a complementary increase in the anti-oxidant uric acid.37 In a direct comparison of IER (75 % ER for two days / week) and CER, both ER strategies displayed equal efficacy in reducing levels of fast - acting advanced oxidation protein products (AOPP) after six months, which displayed a tendency to occur earlier (i.e. at three months) in the IER group.41 Levels of slow - acting (i.e. long term) AOPP tended to decrease in the IER group and increase in the CER group which the authors proposed may have resulted from IER - induced activation of autophagy, a key homeostatic cellular process in which dysfunctional or unnecessary cellular proteins are degraded and recycled.41 On the other hand, a follow - up study using similar IER / CER protocols demonstrated comparable reductions in AOPP in both groups after three months.48 Summary and Future Research Directioin one study was accompanied by a complementary increase in the anti-oxidant uric acid.37 In a direct comparison of IER (75 % ER for two days / week) and CER, both ER strategies displayed equal efficacy in reducing levels of fast - acting advanced oxidation protein products (AOPP) after six months, which displayed a tendency to occur earlier (i.e. at three months) in the IER group.41 Levels of slow - acting (i.e. long term) AOPP tended to decrease in the IER group and increase in the CER group which the authors proposed may have resulted from IER - induced activation of autophagy, a key homeostatic cellular process in which dysfunctional or unnecessary cellular proteins are degraded and recycled.41 On the other hand, a follow - up study using similar IER / CER protocols demonstrated comparable reductions in AOPP in both groups after three months.48 Summary and Future Research Directioin the anti-oxidant uric acid.37 In a direct comparison of IER (75 % ER for two days / week) and CER, both ER strategies displayed equal efficacy in reducing levels of fast - acting advanced oxidation protein products (AOPP) after six months, which displayed a tendency to occur earlier (i.e. at three months) in the IER group.41 Levels of slow - acting (i.e. long term) AOPP tended to decrease in the IER group and increase in the CER group which the authors proposed may have resulted from IER - induced activation of autophagy, a key homeostatic cellular process in which dysfunctional or unnecessary cellular proteins are degraded and recycled.41 On the other hand, a follow - up study using similar IER / CER protocols demonstrated comparable reductions in AOPP in both groups after three months.48 Summary and Future Research DirectioIn a direct comparison of IER (75 % ER for two days / week) and CER, both ER strategies displayed equal efficacy in reducing levels of fast - acting advanced oxidation protein products (AOPP) after six months, which displayed a tendency to occur earlier (i.e. at three months) in the IER group.41 Levels of slow - acting (i.e. long term) AOPP tended to decrease in the IER group and increase in the CER group which the authors proposed may have resulted from IER - induced activation of autophagy, a key homeostatic cellular process in which dysfunctional or unnecessary cellular proteins are degraded and recycled.41 On the other hand, a follow - up study using similar IER / CER protocols demonstrated comparable reductions in AOPP in both groups after three months.48 Summary and Future Research Directioin reducing levels of fast - acting advanced oxidation protein products (AOPP) after six months, which displayed a tendency to occur earlier (i.e. at three months) in the IER group.41 Levels of slow - acting (i.e. long term) AOPP tended to decrease in the IER group and increase in the CER group which the authors proposed may have resulted from IER - induced activation of autophagy, a key homeostatic cellular process in which dysfunctional or unnecessary cellular proteins are degraded and recycled.41 On the other hand, a follow - up study using similar IER / CER protocols demonstrated comparable reductions in AOPP in both groups after three months.48 Summary and Future Research Directioin the IER group.41 Levels of slow - acting (i.e. long term) AOPP tended to decrease in the IER group and increase in the CER group which the authors proposed may have resulted from IER - induced activation of autophagy, a key homeostatic cellular process in which dysfunctional or unnecessary cellular proteins are degraded and recycled.41 On the other hand, a follow - up study using similar IER / CER protocols demonstrated comparable reductions in AOPP in both groups after three months.48 Summary and Future Research Directioin the IER group and increase in the CER group which the authors proposed may have resulted from IER - induced activation of autophagy, a key homeostatic cellular process in which dysfunctional or unnecessary cellular proteins are degraded and recycled.41 On the other hand, a follow - up study using similar IER / CER protocols demonstrated comparable reductions in AOPP in both groups after three months.48 Summary and Future Research Directioin the CER group which the authors proposed may have resulted from IER - induced activation of autophagy, a key homeostatic cellular process in which dysfunctional or unnecessary cellular proteins are degraded and recycled.41 On the other hand, a follow - up study using similar IER / CER protocols demonstrated comparable reductions in AOPP in both groups after three months.48 Summary and Future Research Directioin which dysfunctional or unnecessary cellular proteins are degraded and recycled.41 On the other hand, a follow - up study using similar IER / CER protocols demonstrated comparable reductions in AOPP in both groups after three months.48 Summary and Future Research Directioin AOPP in both groups after three months.48 Summary and Future Research Directioin both groups after three months.48 Summary and Future Research Directions

Leukotriene B4 (LTB4) plays an important role in several cellular processes involved in inflammation, including oxidative metabolism, enzyme release and stimulation of neutrophil migration and aggregation.

Processed, high fat, sugary foods create more oxidative damage and inflammation in the body.1, 2 This leads to greater fatigue, weakened immunity, mood imbalances, and disease.

Oxidative stress is harmful to the body at a cellular level, so much so that it is thought to be a central factor in the aging process itself.1 These suppositories contain epigallocatechin -3-gallate (EGCG), known for being one of the most potent catechins present in green tea.2

The study authors concluded that the Mediterranean diet plus CoQ10 «improves oxidative DNA damage in elderly subjects and reduces processes of cellular oxidation.

* It is also known to play a critical role in the body's defense against oxidative processes and is especially helpful for maintenance of healthy aging.

Free radicals are unstable molecules that damage or «oxidize» cells throughout the body in a process called oxidative stress.

A vicious cycle exists in which molecular mechanisms involved in the aging process, such as oxidative stress, DNA damage, senescence, and inflammation, lead to tissue NAD decline which subsequently exacerbates the processes that caused its decline in the first place.

Acai berries help in slowing down or reversing the aging process that occurs when oxidative damage is done to the cells, as they contain approximately 10 times more antioxidants than grapes and 2 times more antioxidants than berries.

According to an article published in the Journal of Clinical and Aesthetic Dermatology, rosacea «is linked through a variety of mechanisms to other processes including UV damage, vascular changes, and oxidative tissue damage.»

We live in a highly oxidative environment and many processes in our body produce more oxidants, also known as free radicals.

This sets off a chain reaction of imbalance and damage at the cellular level referred to as oxidative stress - a condition thought to be a core factor in the aging process itself!

Research has shown that oxidative damage contributes to many diseases including cancer, heart disease, Alzheimer, and most chronic diseases, and accumulation of oxidative damage plays an important role in the aging process.

These oils are crazy processed and prone to rancidity, which leads to oxidative stress and inflammation in the body.

PeakO2 ™ contains high levels of beta glucans and L - Ergothioneine, a «master» antioxidant that fights damage from oxidative stress in the body, helping athletes train longer, recover faster, and feel better throughout the process.

Oxidative damage has been implicated in the cause of many diseases such as cancer and Alzheimer's and has an impact on the body's aging process.

The health claims: Not only used for weight loss, intermittent fasting is purported to improve insulin sensitivity, build muscle, slow up the aging process, fight oxidative stress, and keep cholesterol in check.

In addition to disrupting fundamental biochemical processes, mercury promotes oxidative stress, depletes antioxidant defenses and destroys biological barriers.

A 2013 study published in Phytomedicine concluded that Lavender oil was able to increase the activity of the body's most powerful antioxidants — glutathione, catalase and Super Oxide Dismutase (SOD), which means that it can be very beneficial in helping prevent or reverse oxidative stress, thus reducing risk of numerous health problem and slowing down the ageing process.

From a paleo point of it would be oxidative stress or methylation issues or refined carbs that cause the start of cardiovascular disease, the cholesterol is merely plugging the hole or repairing the damage and the smaller ldl gets oxidised in the process.

Removing acidic processed foods, sugar, flour, high - glycemic foods, coffee, and alcohol can prevent alterations in membrane potential which compromises mitochondrial function by interfering with the electron transport chain integral to oxygen - based oxidative phosphorylation (55, 56).

For those suffering from lead or other heavy metal poisoning, a protective diet that avoids processed foods and emphasizes the central components of a nutrient - dense and antioxidant - rich WAPF - style diet is essential (see sidebar).21 Nutritional status shapes susceptibility to lead toxicity in important ways, affecting lead's intestinal absorption; its mobilization, distribution and retention in the body; and excretion.22 Because oxidative stress is a likely mechanism explaining some of lead's toxic actions, some investigators have begun to explore whether natural antioxidants and trace minerals may help mitigate lead - induced cell damage.17 Specifically, researchers in Spain found that administration of vitamins A, B6, C and E along with zinc modulated some of the negative effects of lead exposure in rat pups, confirming the influence of nutritional factors on health outcomes following lead exposure.17 Vitamin D also should be a major component of a protective diet.

Oxidative stress may be caused by exposure to environmental toxins, pollution, a poor diet, lack of sleep, lack of exercise, and other factors, including inflammatory processes in the body.

Studies have shown a diet rich in phytochemicals from colorful plant foods like vegetables, fruits, nuts, spices, and teas can reduce oxidative stress and slow the processes of telomere shortening.