Sentences with phrase «oxygen species metabolism»

Among the most interesting ones identified were those associated with 1) the large category of transport, and the more specific categories of carbohydrate transport and cation: amino acid symport; 2) several related to calcium, including calcium ion binding, calmodulin binding, and voltage gated calcium channel activity; 3) DNA damage response and signal transduction; 4) response to oxidative stress; and 5) oxygen and reactive oxygen species metabolism.

Not exact matches

This approach allowed them to create a metabolic index for each species, which sets clearly defined limits for oxygen - breathing sea life: «Marine animals like eelpouts, rock crabs and Atlantic cod can only survive in environments with enough oxygen for them to increase their metabolism to between two and five times their resting metabolic rate if need be.

Free radicals, otherwise known as reactive oxygen species, are a byproduct of metabolism.

Also, in both cell culture and a mouse model of the condition, blocking the excessive splintering reduced production of reactive oxygen species, a potentially harmful byproduct of metabolism, as well as cell death.

Countering the prevailing theory that cellular hydrogen peroxide signaling is broad and non-specific, Whitehead Institute scientists have discovered that this reactive oxygen species (ROS) in fact triggers a distinct signal transduction cascade under control of the mitochondrial respiratory chain — the Syk pathway — that regulates transcription, translation, metabolism, and the cell cycle in diverse cell types.

The basic metabolism of a cell generates reactive oxygen species (ROS) which oxidize cellular lipids, proteins, and DNA leading to production of reactive electrophiles which can lead to deleterious consequences if not eliminated [1].

During the normal processes of metabolism the body generates what are called «reactive oxygen species» which are commonly referred to as «free radicals».

It has been thoroughly researched and shown that the overall health of a body is intrinsically linked to the health of its cells and their ability to metabolize oxidation.5 When oxidative metabolism's limits are exceeded free radicals and reactive oxygen species (ROS) are created and start to build up within the body.

Research suggests that Vitamin C is a highly effective antioxidant and that small amounts can protect essential molecules in the body from free radical damage and reactive oxygen species generated during normal metabolism, as well as through exposure to toxins and pollutants.

Reduce the harmful byproducts of carbohydrate metabolism called Reactive oxygen species (ROS).

Energy metabolism and the production of Reactive Oxygen Species (very small molecules that can result in significant damage to cell structures, of which include oxygen ions, free radicals and peroxides) are thought to underpin many nuerodegenerative disorders, and creatine is thought to enhance the brains ability to survive the metabolic and physical trauma associated with these condiOxygen Species (very small molecules that can result in significant damage to cell structures, of which include oxygen ions, free radicals and peroxides) are thought to underpin many nuerodegenerative disorders, and creatine is thought to enhance the brains ability to survive the metabolic and physical trauma associated with these condioxygen ions, free radicals and peroxides) are thought to underpin many nuerodegenerative disorders, and creatine is thought to enhance the brains ability to survive the metabolic and physical trauma associated with these conditions.

Compared to glucose metabolism, ketone metabolism produces fewer reactive oxygen species — which are known to contribute to inflammation.

* It is also a highly effective antioxidant that can protect the body's cells and molecules from damage by free radicals and reactive oxygen species generated during normal metabolism and exposure to environmental stressors.

In vitro, IF pancreatic islets had increased insulin secretion, glucose metabolism and net reactive oxygen species production, while decreased their mass.

Let me explain... Through normal metabolism of nutrients, your mitochondria release reactive oxygen species (ROS).

Generation of reactive oxygen species, or free radicals such as superoxide and hydrogen peroxide, is a normal byproduct of metabolism, but can damage cellular machinery when excessive and impair the production of cellular energy, which becomes a vicious cycle as energy - intensive repair processes become untenable (25, 26).

In an article reviewing the therapeutic potential of ketogenic diets, it was hypothesized that the metabolism of ketones and fatty acids changes the intracellular milieu to decrease the formation of reactive oxygen species (ROS)(87).

For example, KBs were recently reported to act as neuroprotective agents by raising ATP levels and reducing the production of reactive oxygen species in neurological tissues, 80 together with increased mitochondrial biogenesis, which may help to enhance the regulation of synaptic function.80 Moreover, the increased synthesis of polyunsaturated fatty acids stimulated by a KD may have a role in the regulation of neuronal membrane excitability: it has been demonstrated, for example, that polyunsaturated fatty acids modulate the excitability of neurons by blocking voltage-gated sodium channels.81 Another possibility is that by reducing glucose metabolism, ketogenic diets may activate anticonvulsant mechanisms, as has been reported in a rat model.82 In addition, caloric restriction per se has been suggested to exert neuroprotective effects, including improved mitochondrial function, decreased oxidative stress and apoptosis, and inhibition of proinflammatory mediators, such as the cytokines tumour necrosis factor - α and interleukins.83 Although promising data have been collected (see below), at the present time the real clinical benefits of ketogenic diets in most neurological diseases remain largely speculative and uncertain, with the significant exception of its use in the treatment of convulsion diseases.