Antagonistic Basic Helix - Loop - Helix / bZIP Transcription Factors Form Transcriptional Modules That Integrate Light and Reactive
Oxygen Species Signaling in Arabidopsis
Serrano, F. and Klann, E. (2008) Reactive
oxygen species signaling in the hippocampus.
Not exact matches
Although reactive
oxygen species can damage cells when produced in high amounts, according to a study published online Sept. 5, 2017 by Science
Signaling and featured on the journal's cover, these oxidative
species are crucial
signals that start the process of repairing myofiber.
Among the most interesting ones identified were those associated with 1) the large category of transport, and the more specific categories of carbohydrate transport and cation: amino acid symport; 2) several related to calcium, including calcium ion binding, calmodulin binding, and voltage gated calcium channel activity; 3) DNA damage response and
signal transduction; 4) response to oxidative stress; and 5)
oxygen and reactive
oxygen species metabolism.
Ce - Duox1 / BLI - 3 Generated Reactive
Oxygen Species Trigger Protective SKN - 1 Activity via p38 MAPK
Signaling during Infection in C. elegans.
AÂ Emory News item on a helpful part of the microbiome focuses on how the same type of bacteria â $ «lactobacilli â $ «activates the same ancient
signaling pathway in intestinal cells in both insects and mammals. It continues a line of research from Rheinallt Jones and Andrew Neish on how beneficial bacteria stimulate wound healing by activating ROS (reactive
oxygen species).
Countering the prevailing theory that cellular hydrogen peroxide
signaling is broad and non-specific, Whitehead Institute scientists have discovered that this reactive
oxygen species (ROS) in fact triggers a distinct
signal transduction cascade under control of the mitochondrial respiratory chain — the Syk pathway — that regulates transcription, translation, metabolism, and the cell cycle in diverse cell types.
At the cellular level, p53 can either induce senescence or, of major interest here, inhibit senescence by controlling both reactive
oxygen species (ROS) levels and a
signaling pathway mediated by protein kinases AMPK and mammalian target of rapamycin (mTOR).
The expression of micro-RNA miR - 663 was induced by mitochondrial reactive
oxygen species production, an apoptotic
signal and oxidative stress response, and miR - 663 induced the expression of nuclear - encoded mitochondrial respiratory chain subunits.
Reactive
oxygen species (ROS) are not universally bad, and one of their important functions is to
signal the body to increase protein synthesis as part of the mTOR pathway.
Flavonoids have been shown to inhibit the production of ROS (Reactive
Oxygen Species), the activity of the ROS generating enzymes MPO and NADPH oxidase, and several
signal transducing enzymes involved in the cell activation in neutrophils.
Moreover, reactive nitrogen and
oxygen species (RNOS) are used extensively in cellular
signalling, and cells adaptively regulate endogenous antioxidants on short time scales to respond to deletorious spikes of RNOS faster than we could ever achieve with dietary antioxidants.
Reactive
oxygen species play an essential role in IGF - I
signaling and IGF - I - induced myocyte hypertrophy in C2C12 myocytes.
Reactive
oxygen species are
signaling molecules for skeletal muscle adaptation.
Reactive
oxygen species trigger cellular responses by activation of stress - responsive mitogen - activated protein kinase (MAPK)
signalling pathways.