In this mouse model, mutations in Kras and
p53 genes resulted in the formation of individual tumor cell populations that were labeled with different colors.
Not exact matches
The
results showed that all nine patients lost identical regions of chromosome 17, where the cancer - linked
p53 gene is located, in each of the cancer samples, including the early - stage STIC lesions, suggesting that the «misprinted» or flawed
p53 gene is an early step in ovarian cancer development.
Combining iPSC lines, and bone cancer driven by
p53 mutations in LFS patients, the research team revealed for the first time that the LFS bone cancer
results from an overactive
p53 gene.
Serum albumin high concentration > 47g / l stabilizes the entire genome, includinfg
P53 anti-cancer
gene from constant mutations that
result in cancer.