Sentences with phrase «part by insulin»

The animal's nutritional state, as sensed in part by insulin signaling, influences ISC division and whether divisions are symmetric or asymmetric (Choi et al., 2011; O'Brien et al., 2011).

Not exact matches

In large part, this is because Type II Diabetes is a state of insulin resistance; Your body is no longer listening to the signals sent by that particular hormone.

In the vernacular of the science, calorie restriction appears to increase life span and health span at least in part by «reducing signaling in the insulin / IGF pathway.»

• Although dietary fats do not cause a surge in insulin levels, they could still play a part in diabetes — and thus Alzheimer's — by contributing to obesity.

«We know that amlexanox works to reverse obesity and insulin resistance in part by resolving chronic inflammation and increasing energy expenditure, but that's not the whole story of the drug's effects,» said Shannon Reilly, first author of the study.

These trends may be explained in part by the yo - yo effects that high glycemic - index carbohydrates have on blood glucose, which can stimulate fat production and inflammation, increase overall caloric intake and lower insulin sensitivity, says David Ludwig, director of the obesity program at Children's Hospital Boston.

Both glucose transport and glycogen synthesis are stimulated by insulin, and defects in both processes have been postulated to take part in the development of insulin resistance in type 2 diabetic subjects in vivo.

(17) Slow - aging growth hormone receptor knockout (GHRKO) mice are obese, but highly insulin sensitive: in such animals, surgical removal of visceral adipose tissue impairs insulin secretion and peripheral insulin action, in part by reducing adiponectin production.

Well watch this fabulous Tedx talk by Dr Sarah Hallberg and see how insulin resistance can be playing a part in all the above conditions.

Just today I've watched very interesting and thought provoking interview with Ori Hofmekler, who among other things basically says that the way to increase health and longevity is to lower insulin as much as possible (beans immediately spanng to my mind after hearing this), which is done, in part, by correct food combining (or rather separation).

By referring to this condition as CI, an opportunity emerges: Key underlying problems (insulin resistance, chronic inflammation, increased body fat) and the full spectrum of their associated conditions, and signs and symptoms, can be presented as part of one integrated model.

It may also reduce insulin levels by slowing the movement of food from the stomach into the small intestine (a part of digestion called «gastric emptying») which also slows the breakdown of carbohydrates.

A 2013 study published in The Journal of Nutritional Biochemistry found that the anthocyanins in purple sweet potatoes helped suppress the spike in blood sugar after meals, in part by weakening insulin resistance.

For more information on CAH, see: A Commonly Missed Cause of Infertility, NonClassical CAH by Jeffrey Dach MD (78) Articles with Related Interest PCOS Part Two PCOS Part three Jeffrey Dach MD 7450 Griffin Rd Suite 180/190 Davie, FL 33314 Phone: [phone] Facebook Blog REFERENCES (1) http://www.amazon.com/Clinical-Gynecologic-Endocrinology-Infertility-Editorial/dp/0781747953 The Clinical Gynecologic Endocrinology and Infertility: Leon Speroff MD (2) http://jcem.endojournals.org/cgi/content/full/88/5/1927 A Modern Medical Quandary: Polycystic Ovary Syndrome, Insulin Resistance, and Oral Contraceptive Pills, The Journal of Clinical Endocrinology & Metabolism Vol.

I don't understand what you mean by «steroids are more prone to insulin resistance...» Changes in steroid levels occur in part due to changes in autonomic function.

MI and DCI treat PCOS infertility by improving insulin resistance, androgen levels as well as many of the features of this metabolic disorder and are well worth consideration as part of any fertility treatment plan if you have PCOS.

It is now well established that the adipocyte - derived hormone leptin, which is well known for its central role in body weight regulation in part via its control over thermogenesis, 52 — 55 also plays an important role in blood glucose homeostasis and in the protection of insulin - sensitive tissues against excessive ectopic lipid storage by regulating the partitioning of fatty acid away from storage towards oxidation.

Even critics of the Insulin Hypothesis (like Stephan Guyenet) would agree with some of what Taubes is saying — it's just that they believe CICO is an important part of the equation, the over eating is driven by the brain and changes to the brain and the hormone of interest is leptin.

And our insulin levels, for the most part, are determined by the carb - content of our diet — the quantity and quality of the carbohydrates consumed.

Diet can play an important part in diabetes, since this disease is caused by the imbalance of glucose (sugar) in the body and the insulin the body uses to break down the glucose.

Regarding the child, the importance of the intrauterine and early postnatal environments for metabolic programming and modifications of the epigenome is increasingly recognised, 12 — 14 particularly for metabolic diseases such as obesity and diabetes.15 Thus, GDM is related to macrosomia at birth (> 4 kg), to excess body fat and (central) obesity and to insulin secretion in infants and children, the obesity being in part mediated by maternal body mass index (BMI) or birth weight.16 — 23 Intrauterine exposure to GDM also doubles the risk for subsequent type 2 diabetes in offspring compared with offspring of mothers with a high genetic predisposition for type 2 diabetes, but with normal glucose tolerance during the index pregnancy.24 Maternal prepregnancy overweight and excessive gestational weight gain also predict high birth weight and adiposity during infancy.12 25 This is highly relevant, as up to 60 % — 70 % of women with GDM are overweight or obese before pregnancy.26 Finally, maternal lifestyle behaviour such as a high fat diet or lack of physical activity during pregnancy can influence offspring adiposity independent of maternal obesity.12 27

Part of the deterioration in metabolic control during adolescence has been attributed to the difficulty in keeping up with increasing insulin requirements caused by hormonal changes associated with puberty (Goran & Gower, 2001; Moran et al., 1999), and part has been attributed to a decline in self - care behavPart of the deterioration in metabolic control during adolescence has been attributed to the difficulty in keeping up with increasing insulin requirements caused by hormonal changes associated with puberty (Goran & Gower, 2001; Moran et al., 1999), and part has been attributed to a decline in self - care behavpart has been attributed to a decline in self - care behavior.