Sentences with phrase «pattern of an allele»

The graph shows the global frequency pattern of an allele adaptive to vegetarian diets.

Patterns of allele - frequency change across the rabbit hybrid zone.

Not exact matches

The decision whether the maternal or the paternal version is shut down occurs early in embryonic development — one reason, why for long it was thought that the pattern of active alleles is nearly homogeneous in the various tissues of the organism.

They soon noticed a strange pattern: Stretches of chromosomes inherited from Neandertals also carried ancient alleles, or mutations, found in all the Africans they studied, including the Yoruba, Esan, and Mende peoples.

The team looked for patterns associated with two COLIA1 alleles, called S and s. Women who had one copy of the s allele had bone density in the thighbone and lower spine that was 2 % less than that of women with no s alleles.

With the advent of more and more sophisticated ways to measure genetic variation and relatedness using molecular markers, such as allozyme polymorphisms and DNA - based markers, not only can individuals be tracked as to their parentage, but changes in allele patterns over time and thus the effects of evolution on populations can be «seen» in the genetic information.

A similar pattern has been observed in humans, where alleles associated with lactase persistence in Europe did not rise to significant frequencies until at least the Bronze Age, that is, 3,000 years after the introduction of pastoral livestock35.

This approach revealed coat color variation as one early result of domestication, by showing that the selection of multiple alleles driving diverse coloration patterns was already ongoing in the Early Bronze Age (14).

ONE - PARENT SAMPLE SET PARENTAL GUIDANCE RULES: Maternally methylated (red dots) and paternally methylated (blue dots) regions across the human autosomal chromosomes, based on studies of uniparental disomy samplesGENOME RES, 24:554 - 69, 2014 RESEARCHERS Kazuhiko Nakabayashi, Division Chief, Department of Maternal - Fetal Biology, National Research Institute for Child Health and Development, Tokyo, Japan; David Monk, Principal Investigator, Epigenetics and Cancer Biology Program, Bellvitge Institute for Biomedical Research ORGANISM Human METHODS Bisulfite - seq; bisulfite - chip Methylation is typically associated with the silencing of the nonexpressed allele, making it a convenient marker for imprinted genes, though it's possible for patterns of differential methylation to exist in tissues where both alleles are expressed.

Evolutionary theory predicts what patterns of genetic variation should look like around a positively selected allele.

We also found the same pattern in APOE 34 carriers: carriers of the long and very long alleles had a slightly higher, but not statistically significantly different, AAO than did carriers of the short alleles (Supplemental Figure 4; http://neuroscienceresearch.wustl.edu/Pages/cruchaga2011.aspx).

A - Contrasting patterns of introgression between subspecies of the European rabbit summarized with pie charts of allele frequency data across the rabbit hybrid zone and shape of genealogies for four representative loci (DIAPH2, HPRT1, FMR1, and MSN; data from [21], [26]--[27].

Distinct patterns of brain activity in young carriers of the APOE - epsilon4 allele.

The early Anglo - Saxon samples from Oakington are more diverse with O1 and O2 being closer to the middle Anglo - Saxon samples, O4 exhibiting the same pattern as the Iron Age samples, and O3 showing an intermediate level of allele sharing, suggesting mixed ancestry.

SIX engaging worksheet activities will help your students learn inheritance terminology, construct and analyze a variety of Punnett Squares and develop deeper understandings of Mendel's Principles as well as more advanced patterns of inheritance such as incomplete dominance, codominance, sex - linked inheritance, multiple alleles, and polygenic inheritance.

This provides a logical genetic indicator and explains why it is likely that certain dilute or patterned dogs, such as extreme piebalds, or other types of homozygous dilutes common in the APBT, as well as those that may be carrying the Merle pattern are prone to psychological, neurological and / or immunological problems found in other breeds that carry these alleles.

To avoid producing «double merle» (M / M) puppies, dogs with a copy of M (particularly those with M alleles near the size which is likely to produce the classic merle coat color / pattern) should only be bred to dogs that do not have a copy of the M allele.

Although some coat color / pattern variations have been associated with specific sizes of the M allele in certain breeds, referred to here as a «bin», the size of the M allele does not guarantee a specific outcome.

Specific sizes of the M Allele have been associated with the potential to produce «classic» merle patterning or other M - associated coat color variations.

Merle is inherited in a dominant fashion, meaning that only one copy of an M allele is necessary for a dog to display some variation of the merle coat color / pattern, which is marked by random dilution of eumelanin (black pigment) leaving patches of normal coat color within areas of diluted pigmentation.

There is no direct test for the Wild type (think wolf pattern) allele which is designated aw.Any dog which has at least one copy of Ay (and no KB) will be fawn or sable, either with or without brindling.Any dog that is «aa» (and no KB) will be black.

The recessive allele, ky, allows the basic patterns of the A locus to be expressed.

The dog could be a carrier with a false allele (but because of how markers are inherited it will never be a Pattern B; it's still a Pattern C).

Through the examination of SNP allele frequencies in the Labrador Retriever family, we identified 2 chromosomes harboring CMS candidate genes that showed an inheritance pattern consistent with autosomal recessive transmission.

A weakness of the correlational approach used here is the degree to which this pattern of results could occur as a result of random processes such as genetic drift (fluctuations in allele frequency due to chance; Keinan et al., 2007).

Children who manifest higher levels of shyness or have 1 or 2 copies of the short allele of the serotonin transporter promoter gene appear to have a different pattern of processing affective stimuli of interpersonal hostility.

Conclusion Children who manifest higher levels of shyness or have 1 or 2 copies of the short allele of the serotonin transporter promoter gene appear to have a different pattern of processing affective stimuli of interpersonal hostility.