SM may result in other neurological deficits such as thoracic limb weakness and muscle atrophy (due to ventral horn cell damage) and
pelvic limb ataxia and weakness (due to white matter damage or involvement of the lumbar spinal cord by the syrinx).
Gait abnormalities, such as
ataxia, paresis, or paralysis, may be present if secondary spinal cord or nerve root compression occurs.2 When neurologic signs are present, they correlate to the location of the discospondylitis lesion; cervical vertebral column lesions may cause tetraparesis and neck pain; thoracolumbar lesions may cause
pelvic limb paresis, proprioceptive
ataxia, and back pain; and lumbosacral lesions may cause a stiff, stilted
pelvic limb gait.1, 2 Discospondylitis can affect any area of the vertebral column, but the most commonly affected sites are L7 to S1, caudal cervical, mid-thoracic, and the thoracolumbar spine.