Sentences with phrase «plasma membrane into»

After stimulation (right), PtdIns (4,5) P2 is hydrolyzed by PLC - beta and the YFP - tagged domain is released from the plasma membrane into the cytosol.

PLC - beta is a 150 kDa enzyme that catalyzes the hydrolysis of phosphoinositide PtdIns (4,5) P2 located in the plasma membrane into diacylglcyerol (DAG) and Ins (1,4,5) P3.

Not exact matches

Of course, if it's a fat - soluble drug they can get more into breast milk than in plasma because they can get and cross that fatty membrane.

This allows synaptic vesicles filled with neurotransmitter to fuse with the plasma membrane and release the neurotransmitters into the synaptic cleft.

The link between endocytosis and generation of cortical actin waves may provide deeper insight into how cells maintain the composition and dynamics of the plasma membrane.

Here we used this approach to show that newly synthesized connexin43 was transported predominantly in 100 - to 150 - nanometer vesicles to the plasma membrane and incorporated at the periphery of existing gap junctions, whereas older connexins were removed from the center of the plaques into pleiomorphic vesicles of widely varying sizes.

This project aims to elucidate the role of membrane recycling for the lateral segregation of integral membrane proteins into dynamic microcompartments in the budding yeast plasma membrane (PM).

They thought it was a protein that functions on the plasma membrane to transport inorganic phosphate into a neuron.

Compartmentalized cyclic adenosine 3 ′, 5 ′ - monophosphate at the plasma membrane clusters PDE3A and cystic fibrosis transmembrane conductance regulator into microdomains.

Nicola Bauer (Corbeil, TUD)-- «New insights into the plasma membrane dynamics of rare stem cells by studying the prominin molecule» (2007)

A Highlights from MBoC Selection: Compartmentalized Cyclic Adenosine 3 ′, 5 ′ - Monophosphate at the Plasma Membrane Clusters PDE3A and Cystic Fibrosis Transmembrane Conductance Regulator into Microdomains.

We delve into very fundamental problems such as «how does a malaria red blood cell attach itself to a blood vessel» or «how does binding of a ligand to a cell surface receptor or contact of a crystalline surface with the plasma membrane drive lipid sorting and how will this lead to signalling» but then immediately apply it to a practical problem such as «how does contact of uric acid crystals with dendritic cells cause gout in affected joints and how can we prevent this occurrence?»

«The plasma membrane also dynamically changes shape during cell migration, cancer cell invasion, cell division, nutrient uptake, and entry of pathogens and viruses into cells,» Shin explains.

During an action potential, calcium influx into the presynaptic terminal triggers the fusion of synaptic vesicles with the plasma membrane, leading to the release of transmitter through the process of exocytosis.

The data suggest that the shell formation mechanism can be divided into three different main parts, namely 1) transport of ions across plasma membranes, 2) secretion of shell matrix proteins and 3) production of protease inhibitors.

This morphology prevents at least some FB - MO proteins from diffusing into the plasma membrane [44], and it may prevent diffusion of PEEL - 1 as well.

EPA and DHA are more rapidly incorporated into plasma and membrane lipids and produce effects more rapidly than does ALA..