Not exact matches
They found that the inactivation of POT1 caused by these
mutations leads to longer and
potentially unprotected telomeres, regions at the end of our chromosomes that protect chromosomes from
damage.
Fewer free radicals would decrease the rate of DNA
damage, which would curtail the rate of DNA
mutation, slowing the rate of aging and disease and
potentially increasing our lifespan.
So, the thinking is that lower methionine intake leads to less free radical production — the so - called «reactive oxygen species,» which slows the rate of DNA
damage, which then would slow the rate of DNA
mutation, slowing the rate of aging and disease — thereby
potentially increasing our lifespan.