Each of these cells, called naive,
pre-implantation embryonic cells, has the capacity to develop into any cell type in the human body, an ability called pluripotency.
Not exact matches
Benedict argued that non-conjugal reproduction such as in vitro fertilization had created «new problems» ¯ the freezing of human embryos, for instance, and the selective abortion of medically implanted embryos, together with
pre-implantation diagnosis,
embryonic stem -
cell research, and attempts at human cloning.
Although primed, post-implantation
embryonic stem
cells can still turn into any type of human
cell, they are more difficult to work with than the
pre-implantation, naive
cells.
Human
embryonic stem
cells derived from affected embryos during a
pre-implantation diagnostic (PGD), as well as the conversion of somatic
cells, such as skin fibroblasts, into induced pluripotent stem
cells by genetic manipulation, offer the unique opportunity to have access to a large spectrum of disease - specific
cell models.
Benedict argued that non-conjugal reproduction such as in vitro fertilization had created «new problems» ¯ the freezing of human embryos, for instance, and the selective abortion of medically implanted embryos, together with
pre-implantation diagnosis,
embryonic stem -
cell research, and attempts at human cloning.
For the first time, through the use of human
embryonic stem
cells (hES) sourced from
pre-implantation diagnosis, researchers from Inserm's Institute for Stem
Cell Therapy and Exploration of Monogenic Diseases (I - Stem) have successfully identified the previously unknown mechanisms involved in Steinert» disease, also known as type 1 myotonic dystrophy.
Using
pre-implantation genetic diagnosis derived human
embryonic stem
cell lines (hES) or human induced pluripotent stem
cells carrying the causal mutation for neuro - muscular diseases, our objective is double: identify new physiopathogical mechanisms and develop new therapeutic strategies.