For women contemplating having a baby, new research adds to the evidence suggesting that starting
a pregnancy at a normal weight is best.
About 46 percent of women started
their pregnancies at a normal weight.
Not exact matches
By 4 days after surgery I was back to
normal, and
at 3 weeks postpartum today I have lost double the
weight I gained during
pregnancy!
Therefore, if you have gained a
normal weight range during your
pregnancy, you don't need to worry much about losing it
at this point.
A woman being obese (BMI of 30.0 or higher) prior to getting pregnant increased the odds of her child being overweight
at age 2 by more than two-fold compared to women who had a
normal pre-
pregnancy weight (BMI between 18.5 and 25), after adjusting for
weight gain during
pregnancy, gestational diabetes and breastfeeding.
The results show that mothers with excessive
weight gain during
pregnancy weighed more and had greater body fat seven years after delivery if they began
pregnancy at normal or slight overweight.
The current Institute of Medicine recommendation is to gain 25 to 35 pounds during
pregnancy if already
at a
normal weight and 15 - 25 pounds if overweight.
This could be a problem in patients with congestive heart failure or other patients who require sodium restriction May stunt growth if used in young, growing animals May lower seizure threshold and alter mood and behavior
At high doses, this medication can cause birth defects early in
pregnancy, be irritating to the stomach or cause higher than
normal blood sugar levels If your pet has received high doses, it should not be vaccinated without your veterinarian's advice as the vaccine may not work or it may actually give your pet the disease you are trying to prevent Less common side effects include
weight gain, insomnia, panting, diarrhea, vomiting, elevated liver enzymes, pancreatitis and behavior changes Serious side effects are not expected with routine use.
A covariate was included in the multivariate analyses if theoretical or empirical evidence supported its role as a risk factor for obesity, if it was a significant predictor of obesity in univariate regression models, or if including it in the full multivariate model led to a 5 % or greater change in the OR.48 Model 1 includes maternal IPV exposure, race / ethnicity (black, white, Hispanic, other / unknown), child sex (male, female), maternal age (20 - 25, 26 - 28, 29 - 33, 34 - 50 years), maternal education (less than high school, high school graduation, beyond high school), maternal nativity (US born, yes or no), child age in months, relationship with father (yes or no), maternal smoking during
pregnancy (yes or no), maternal depression (as measured by a CIDI - SF cutoff score ≥ 0.5), maternal BMI (
normal / underweight, overweight, obese), low birth
weight (< 2500 g, ≥ 2500 g), whether the child takes a bottle to bed
at age 3 years (yes or no), and average hours of child television viewing per day
at age 3 years (< 2 h / d, ≥ 2 h / d).
Regarding the child, the importance of the intrauterine and early postnatal environments for metabolic programming and modifications of the epigenome is increasingly recognised, 12 — 14 particularly for metabolic diseases such as obesity and diabetes.15 Thus, GDM is related to macrosomia
at birth (> 4 kg), to excess body fat and (central) obesity and to insulin secretion in infants and children, the obesity being in part mediated by maternal body mass index (BMI) or birth
weight.16 — 23 Intrauterine exposure to GDM also doubles the risk for subsequent type 2 diabetes in offspring compared with offspring of mothers with a high genetic predisposition for type 2 diabetes, but with
normal glucose tolerance during the index
pregnancy.24 Maternal prepregnancy overweight and excessive gestational
weight gain also predict high birth
weight and adiposity during infancy.12 25 This is highly relevant, as up to 60 % — 70 % of women with GDM are overweight or obese before
pregnancy.26 Finally, maternal lifestyle behaviour such as a high fat diet or lack of physical activity during
pregnancy can influence offspring adiposity independent of maternal obesity.12 27