When these enzymes are inhibited, SMN2 (and
presumably other genes) remains accessible, and is therefore transcribed more often.
Often this is attributed to mysterious «background effects,»
presumably other genes that were not properly accounted for.
Not exact matches
The mutation in this case can be compatible either with male or female development,
presumably due to
other genes or chance events.
In its healthy form the
gene encodes a protein that unwinds DNA,
presumably so
other proteins that manipulate DNA can wriggle between the strands to do their work.
«
Presumably,
genes governing male reproduction are under continuous pressure to evolve ways to out - compete
other males when it comes to fathering offspring.»