Sentences with phrase «promotes cancer tumor growth»

One of his many discoveries was that casein, the protein found in milk products, promotes cancer tumor growth.

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This recipe is full of chickpeas which help promote bone health, improve blood sugar levels in diabetics, improve blood pressure, promote heart health, reduce bad cholesterol, and reduce risks of cancer and decreases tumor growth rates.

Mogroside V has been found in research to have the ability to inhibit tumor growth in pancreatic cancer by interfering with the rapid dividing of cancer cells, preventing angiogenesis (blood flow to the tumor), and even promoting cancer cell death (10).

Inflammatory breast cancer cells display a triple - negative breast cancer phenotype that lacks the receptors needed to promote tumor growth.

Researchers at the Bellvitge Biomedical Research Institute of Bellvitge, the Catalan Institute of Oncology and the University Hospital of Bellvitge have participated in an international study published in the journal Cancer Cell that describes how exosomes secreted by tumor cells contain protein and microRNA molecules capable of transform neighboring cells into tumoral cells promoting tumor growth.

Women with the KRAS - variant are also more susceptible to triple - negative breast cancer, tumors whose growth is not fueled by the hormones estrogen and progesterone, or by the presence of a particular genetic mutation known as HER2, which promotes cancer cell growth.

But recent evidence suggests that Nanog promotes tumor growth by stimulating the proliferation of cancer stem cells.

To demonstrate the potential for treating lung disease, the researchers used the nanoparticles to block two genes that have been implicated in lung cancer — VEGF receptor 1 and Dll4, which promote the growth of blood vessels that feed tumors.

A new study shows that stable microvasculature constitutes a dormant niche for disseminated breast cancer cells, whereas a sprouting neovasculature (green points) promotes breast tumor cell growth.

Their study, published in the ACS journal Chemical Research in Toxicology, found that triclosan, as well as another commercial substance called octylphenol, promoted the growth of human breast cancer cells in lab dishes and breast cancer tumors in mice.

They found that, by using math models to understand the complex dynamics within cancers, they could use small changes in the environment to promote the growth of cells that are less aggressive and thereby decrease tumor growth.

«Blocking known cancer driver unexpectedly reveals a new tumor - promoting pathway: Targeting both pathways with drug inhibitors more effective in stopping colorectal cancer growth.»

A widespread cancer - causing protein called MYC promotes the growth of tumor cells in part by ensuring that RNA transcripts are properly spliced, according to latest work from A * STAR researchers.

Due to the lack of oxygen in the cancer cells, VEGF - A (Vascular Endothelial Growth Factor) is formed and this promotes the formation of new blood vessels to supply the tumor.

«Researchers inhibit tumor growth in new subtype of lung cancer: Insight into tumor suppressing and tumor promoting mechanisms offers potential for new treatments.»

«Our study suggests that microRNAs are an important factor that modify bone remodeling and promote tumor growth in metastatic prostate cancer,» lead investigator Shingo Sato concludes.

The researchers found that cancers with a mutation in the BRAF gene require copper to promote tumor growth.

Scientists have known that LPA is secreted by many types of cancer cells, appears to promote the growth and spread of tumor cells, and that immune cells known as CD - 8 «killer» T cells have several receptors for LPA.

The analysis also found that a significant fraction of tumors contained rearrangements and mutations of a gene called PREX2, and experiments confirmed that cancer - associated mutations of PREX2 promoted the growth of human melanoma cells in mice.

More importantly, Zhang and his team for the first time found that treating the pancreatic tumor cells with MIR506 induced autophagy, a process that occurs as a normal and controlled part of an organism's growth or development and that could promote cancer cell death.

In recent years, my laboratory has discovered driver mutations, which confer a selective growth advantage and thus promote cancer development, for certain rare tumor types.

«Treatment of breast cancer remains difficult because there are multiple signaling pathways that promote tumor growth and develop resistance to treatment,» said Craig Meyers, Ph.D., Distinguished Professor of Microbiology and Immunology.

Anna Huttenlocher, University of Wisconsin, USA Neutrophils in the Tumor Microenvironment Neutrophils, Wounds, and Cancer Progression Stefan Kaufmann, Max Planck Institute, Germany Pathology and immune reactivity: understanding multidimensionality in pulmonary tuberculosis Constitutive BAK activation as a determinant of drug sensitivity in malignant lymphohematopoietic cells Kathryn Moore, New York University, USA MicroRNA -33-dependent regulation of macrophage metabolism directs immune cell polarization in atherosclerosis Lalita Ramakrishnan, University of Cambridge, UK Myeloid Growth Factors Promote Resistance to Mycobacterial Infection by Curtailing Granuloma Necrosis through Macrophage Replenishment Beth Stevens, Harvard University, USA Microglia: Dynamic Mediators of Synapse Development and Plasticity Do glia drive synaptic and cognitive impairment in disease?

Although mutations to the complex, dubbed GATOR1, are prevalent in cancer cells and promote tumor growth, they also render the cells susceptible to treatment with the drug rapamycin.

11/10/2008 Anti-Angiogenesis Drugs May Foil Tumors by Building Up Blood Vessels, Not Tearing Them Down, Moores UCSD Cancer Center Scientists Find Scientists have thought that one way to foil a tumor from generating blood vessels to feed its growth — a process called angiogenesis — was by creating drugs aimed at stopping a key vessel growth - promoting protein.

Scientists even think both decreases and increases in methylation (chemical bundles that enzymes attach to DNA) may cause cancer, by turning on too many growth - promoting genes or turning off tumor - suppressing genes.

Through its various targets, MMP1 promotes not only tumor invasion but also breast cancer colonization to bone by mechanisms that include the release of membrane - bound EGF - like growth factors from tumor cells, leading to activation of EGF receptor signaling and suppression of OPG expression in osteoblasts, which in turn promotes the differentiation and activation of osteoclasts required for bone destruction and enhanced tumor growth in the bone microenvironment (32).

When breast cancer cells invade the bone microenvironment, they produce molecules that activate osteoclastic bone resorption, leading to the release of growth factors stored in the bone matrix to promote tumor growth.

We also found that the EphB4 receptor expressed on the surface of breast cancer cells can promote tumor xenograft growth by enhancing blood vessel formation through interactions with its preferred ligand, ephrin - B2, present in tumor endothelial cells.

Important features of XMRV biology include (1) tropism for a variety of cell lines, including prostate cancer DU145 and LNCaP cells [27], [43], [48], and human neural cell types [57], (2) adaptations that promote growth in prostate epithelium and human - derived prostate cancer cell lines including an androgen response element in the promoter region [58] and downregulation of APOBEC3G [59], and (3) cellular effects with potential oncogenic properties including increased tumor aggressiveness mediated by downregulation of p27 [60] and differential regulation of several microRNAs [61].

Tumors thrive in a low oxygenated environment and this condition known as hypoxia promotes the invasion of cancer and its growth (1).

Some claim that it does promote the growth of breast cancer cells, while others say that it can halt the tumor.

Interleukin - 8 (IL - 8), a well - known tumor - growth promoting inflammatory cytokine104 is «substantially increased» in a number of different types of cancer cells.105 «We found for the first time that caprylic acid and MCT suppress IL - 8 secretion by Caco - 2 cells [colon cancer cells],» reported Japanese researchers in 2002.106

Additionally, corn oil can speed up the growth of tumor and promotes * prostate diseases, especially prostate cancer.

It can cause insulin to be released, promoting fat storage and elevated insulin - like growth factor that is associated with tumor growth and cancer.

Diabetics and prediabetics also appear to be at higher risk of cancer, likely because they have too much insulin in their bodies and insulin promotes tumor growth.

Since sterols are animal growth factors and phytosterols and plant growth factors which we, as animals don't particularly respond to, I would think that the (animal) sterols would set us up for cancer simply by virtue of the fact that as growth factors, they would promote growth of tumors.

In addition, they detoxify and neutralize carcinogens in the body, while inhibiting angiogenesis — the cancer - promoting growth of blood vessels that nourish tumors.

And, although this effect may help to prevent heart attacks and other forms of heart disease, it may have the potential to promote cancer as well by increasing the growth of blood vessels in cancerous tumors.

According to the article, sugar may fuel cancer cells, promoting tumor growth.

Also, recent research reveals that the inadequate supply of oxygen that characterizes sleep apnea may promote increased vascular and tumor growth in cancer patients.

Cancer researchers now consider that the problem with insulin resistance is that it leads us to secrete more insulin, and insulin actually promotes tumor growth.

But because metformin may stem cancer through several different channels — by lowering insulin, directly slowing tumor growth, or promoting suicide by cancer cells — it could potentially prove useful in many types of cancer.