Not exact matches
Past studies have shown that ET - 1 is the most potent, longest - lasting vasoconstrictor
in humans, and it
promotes the development of
plaques inside the
arteries.
Though the UPR usually
promotes healthy endoplasmic reticulum function, sustained UPR activation sometimes results
in diseases such as atherosclerosis, the deposition of fatty
plaques on
artery walls, among other conditions.
In men, as noted above, DHEA is a 5AR agonist, thus
promoting conversion of testosterone to DHT, and has also been shown to exert anti-atherogenic effects; higher serum levels of DHEA - S correlate inversely with carotid
artery intima - media thickness,
plaque and blood flow volume.94
This process eventually causes blockage to occur
in the form of
plaque -
promoting inflammation of the
artery walls.
Lp (a) is known to accumulate
in plaques and
promote atherosclerosis
in the walls of the
arteries.
Sugar, which is inflammatory,
promotes plaque formation
in arteries, and raises stress hormones.
This means that consumption
promotes the development of fatty
plaques in the
arteries.
This evidence includes findings from a mouse - based study published
in the journal Pathobiology
in 2005,
in which allicin appeared to
promote the breakdown of LDL cholesterol and reduce
plaque buildup
in the animals»
arteries.