Subtitle: Reduction of physiological reactivity to
psychosocial stress by Taiji practice — who benefits the most?
Not exact matches
Effective parental / executive leadership and authority to nurture, protect, and socialize Organizational stability, with clarity, consistency and predictability Adaptability and flexibility — to better meet
stresses and change Open communication characterized
by clarity of rules and expectations, positive interactions, and a range of emotional expression and empathic responsiveness Effective problem - solving and conflict - resolution processes A shared belief system that enables trust, and promotes ethical values and concern for the larger human community Adequate resources for security and
psychosocial support
Although all new fathers, regardless of their youngest child's age, experienced a significant reduction in AM and / or PM T compared with nonfathers (Fig. 2 and Tables S5 and S6), fathers with newborns (1 mo old or less) at the time of follow - up hormone assessment showed significantly greater declines in AM (P = 0.023) and PM (P = 0.003) T compared with fathers whose youngest child was older than 1 y of age, which was not accounted for
by reports of
psychosocial stress, sleep quality, or involvement in caregiving (Tables S7 and S8).
Values are adjusted for time of saliva collection and usual wake time (AM) and are derived from regressing the change in T on fatherhood, stratified
by child age, with men who were not fathers in 2005 and 2009 as the comparison group, and controlling for sleep quality and
psychosocial stress (Tables S5 and S6).
The aim of the study, led
by PhD student, Helen Rockliff, from the University of Bristol's School of Clinical Sciences, was to find out what types of coping strategies, social circumstances and personality traits — called
psychosocial factors — help people through IVF treatment, and which types are linked to especially high
stress levels, and can lead to depression and anxiety disorders.
Psychosocial stress in adulthood is associated with a higher risk of type 2 diabetes, possibly mediated
by behavioral and physiological factors.
Instead, the growing consensus is that the health risks of low status are due in part to the chronic
psychosocial stress of the rat race itself, and of perceived social subordination, whether
by other individuals or
by institutions.
Psychosocial stress is caused
by mental, emotional, and social factors.
A previous meta - analysis
by the same first author focused on parent training using data on mothers found that parent training programmes improved mothers»
psychosocial well - being, including depression, anxiety /
stress, self - esteem and spouse / marital adjustment.1
Objective To understand why children exposed to adverse
psychosocial experiences are at elevated risk for age - related disease, such as cardiovascular disease,
by testing whether adverse childhood experiences predict enduring abnormalities in
stress - sensitive biological systems, namely, the nervous, immune, and endocrine / metabolic systems.
The study investigates whether among some adolescents with asthma,
psychosocial stress induces a shift in immunity that supports inflammatory processes in the lungs, and whether the degree of immune change is associated with the degree of
stress experienced
by the subject.
We test the hypothesis that this association is mediated
by concurrent
psychosocial stresses and maternal life - course risks including those that predate the child's birth.
OBJECTIVES: To examine the prenatal and postnatal mechanisms
by which maternal adverse childhood experiences (ACEs) predict the early development of their offspring, specifically via biological (maternal health risk in pregnancy, infant health risk at birth) and
psychosocial risk (maternal
stress during and after pregnancy, as well as hostile behavior in early infancy).
For example, N - acetyl cysteine, a mitochondrial agent, has been shown to improve depressive symptoms
by alleviating oxidative
stress in the brain.47 This may improve long - term cognitive,
psychosocial and functional impairment.
Workload, in particular tight deadlines, too much work and too much pressure or responsibility, a lack of managerial support, organisational changes at work, violence and role uncertainty are identified causes of work - related
stress.1 These factors are antecedents of sickness presenteeism which is mediated
by mental and physical health.2 At the individual level, chronic
stress produces long - term deleterious effects in health, namely, cardiovascular diseases, 3 burn - out, anxiety and depression.4 Sickness absence in Europe is associated with
psychosocial work factors.5 The link between work performance,
stress and health poses an important challenge to workers, employers and organisations in general, as
stress should be monitored and mitigation measures implemented accordingly.6
Their model proposes that the manifestation of the adverse effects of certain risk factors (e.g., parameters of the disease / disability, functional independence, and
psychosocial stressors) on children's
psychosocial adaptation (e.g., mental, physical, and social functioning) may be attenuated
by a variety of resistance factors (e.g., intrapersonal, social — ecological, and
stress - processing variables).
Their model proposes that manifestation of the adverse effects of certain Risk Factors (e.g., parameters of the disease / disability, functional independence, and
psychosocial stressors) on Adaptation (e.g., mental, physical, and social functioning) may be attenuated
by a variety of Resistance Factors (e.g., intrapersonal, social — ecological, and
stress processing variables).
These families may be helped
by early
psychosocial interventions to alleviate
stress and reduce children's emotional and behavioral problems.
However, one of the major proposed mechanisms to account for the relationship between prenatal
psychosocial stress and the offspring's susceptibility to infections is the secretion of cortisol, which is regulated
by the maternal HPA axis and the placenta [14,20].
Stress generation effects were moderated
by cortisol responses to a laboratory
psychosocial stressor, such that individuals with higher cortisol responses exhibited a pattern consistent with the depression autonomy model, whereas individuals with lower cortisol responses showed a pattern more consistent with the depression sensitization model.
Randomisation was
by minimisation controlling for baseline frequency of self harm, presence of conduct disorder, depressive disorder, and severity of
psychosocial stress.