There's
the pubertal age.
Schneider and her colleagues determined
pubertal age at first drink in 283 young adults (152 females, 131 males) that were part of a larger epidemiological study.
Scouts tend to be civil and respectful kids and most under
pubertal age, so I'm not sure why you'd presume they'd be interested in fooling around on a camping trip.
Not exact matches
In its 2012 Policy Statement on Baseball and Softball8, the AAP acknowledges the recent studies challenging the theory that the curveball and slider are stressful to the young elbow, but, on the basis of other studies showing increased injury among those who throw curve balls and sliders at early
ages, continues to recommend that introduction of the curve ball be delayed until after
age 14, or when
pubertal development has advanced to the stage when the athlete has started to shave, and that sliders not be thrown until
age 16.
Because some kids start to show signs of puberty as early as
age 7, your doctor will check
pubertal development.
Because some children start to show signs of puberty as early as
age 7, your pediatrician will check
pubertal development.
Since timing of puberty is not a simple function of chronological
age, and also greatly differs between the sexes, the
pubertal phase at first drink may therefore represent a stronger and better indicator for subsequent alcohol - related problems than simply the
age.»
At your
age it's
pubertal gynecomastia and it should go away on its own.
Pubertal gyno is pretty common at your
age, therefore doctors are prepared to deal with it.
At your
age, it could be a
pubertal gyno but usually, it starts earlier than 17.
Salivary cortisol levels throughout childhood and adolescence: Relation with
age,
pubertal stage, and weight
Circadian cortisol rhythms in healthy boys and girls: Relationship with
age, growth, body composition, and
pubertal development
Pearson correlations and t tests were conducted to explore variation in brain volumes related to children's sex,
age,
pubertal status, history of psychiatric disorders (yes / no), and children's history of psychotropic medication use (yes / no).
For analyses of white matter volume, children's
age and
pubertal status were also included as covariates.
We conducted a sensitivity analysis to determine whether the observed group differences were explained by differences in
pubertal development, assessed with a self - report questionnaire at the same
age as physiological reactivity was assessed.
However, the lack of a moderating effect of
pubertal status on
age - and sex - adjusted BMI z scores and any metabolic parameter indicates that the same caution is required when treating younger children and adolescents.
Using data from a sample of 478 males from a longitudinal birth cohort, we describe the calculations of three independent height - based markers of
pubertal timing:
Age at Peak Height Velocity (APHV), Height Difference in Standard Deviations (HDSDS), and Percent Achieved of Adult Stature (PAAS).
we describe the calculations of three independent height - based markers of
pubertal timing:
Age at Peak Height Velocity (APHV), Height Difference in Standard Deviations (HDSDS), and Percent Achieved of Adult Stature (PAAS).
Additional information will be collected from health and
pubertal maturation examinations at
ages 13.5, 14.5, 15.5, and 16.5; monitored physical activity at
age 15; analyses of middle school and high school transcripts; and surveys of middle school and high school personnel.
We further hypothesized that youth more advanced in
pubertal development would show increased neural response to peer rejection and acceptance (above and beyond the effects of
age) in regions involved in social and affective processing.
Pubertal timing was linked to depressive symptoms in both
age groups, and predicted an increase in depressive symptoms among the 7th graders.
The objective of this study was to test a comprehensive model of biologic (
pubertal status), family (communication and conflict), and psychological influences (behavioral autonomy) on diabetes management and glycemic control in a sample of youth (N = 226) with type 1 diabetes recruited during late childhood / early adolescence (
ages 9 — 11 years).
A whole - brain regression using AFNI's 3dRegana was conducted to identify areas showing a main effect of
pubertal status on response to rejection and / or acceptance controlling for the effects of chronological
age.
We examined whether sexual harassment, which has previously been linked to both
pubertal timing and depressive symptoms, mediates this link, using a two - wave longitudinal study including 454 girls in 7th (M
age = 13.42, SD =.53) and 8th grade (M
age = 14.42, SD =.55).
Future research should therefore focus on a more homogenous sample with regard to
age and
pubertal development, or it should perform subsequent sampling in girls during their adolescence.
Further, it was examined whether
pubertal status would be a more sensitive moderator in these models compared to
age.
When examining models of depression from a developmental perspective,
age may be preferred over
pubertal status, as
age is a less complex variable.
Additional analyses indicated that
pubertal timing moderated the association between synchrony and depressive symptoms at
age 20, such that girls who exhibited asynchronous development had the highest levels of depressive symptoms when they matured later than peers.
The two - way interaction
age (or
pubertal status) by gender (included in all models under test) was not significant; whereas it would be expected that girls report more depressive symptoms as level of maturation (
age /
pubertal status) increases compared to boys.
The analyses with
pubertal status instead of
age yielded different results with regard to the main models.
Results showed that although the four - way interaction between NCS, stressors, gender, and
age was significant while the four - way interaction with
pubertal status was marginally significant, the interpretation of these interactions was largely similar, i.e., NCS and depressive symptoms were significantly related only in the presence of many stressors in middle to late adolescent boys (or in boys reporting a high
pubertal status).
The purpose of this report is to provide evidence of an association between within - person variability in diurnal testosterone over 1 year, lifetime exposure to violence, and the manifestation of antisocial behavior in 135
pubertal -
aged adolescents across 1 year.
Moderation by
pubertal status instead of
age yielded slightly different results, that is, in the model with stress - reactive rumination, the relationship between negative cognitive style and depressive symptoms was stronger in adolescents who perceived their
pubertal status as high, whereas
age did not moderate this relationship.
Perceived
pubertal status, reflecting the subjective experience of morphological changes related to puberty (Angold and Costello 2006), may be a more sensitive moderator of NCS than
age.
However, contrary to
age, how
pubertal status is perceived and reported may also be influenced by depressive symptoms.
The role of autonomy and
pubertal status in understanding
age differences in diabetes responsibility across adolescence
Although there is no one established formula for determining a child's insulin requirement, insulin requirements are usually based on body weight,
age, and
pubertal status.
Developmental processes in early adolescence: Relations among chronologic
age,
pubertal stage, height, weight, and serum levels of gonadotropins, sex steroids, and adrenal androgens
These findings are consistent with those of Pieters et al. (2015) who found that sleep problems prospectively predicted increased substance use, internalizing symptoms, and externalizing problems 1 year later in a sample of 555 adolescents (
ages 11 — 16 years), even after controlling for baseline levels of adjustment,
age, sex, and
pubertal development.
The results for
pubertal status and
age are strikingly similar, indicating that after controlling for the effect of all the other variables in the regression model, the impact of life events on depression is significantly greater in the
pubertal girls (sex ×
pubertal status [
age] × life events interaction).
Because of the marked collinearity between
age and
pubertal status, we were unable to estimate the effects of these 2 variables simultaneously.
Under 2 separate regression models (1 for
pubertal status and 1 for
age), each variable's partial sums of squares and associated probability were used to examine these effects in the 2 sexes together (Table 1).
To address these issues in the present study, ADHD symptoms, IQ,
age,
pubertal stage and substance use will be included as covariates in order to control for their effects.
Due to the detailed characterization of our sample, we were able to control for various confounding variables such as comorbid ADHD symptoms,
age,
pubertal stage, IQ and substance use.
Age - independent effects of
pubertal status on behavioral constraint in healthy adolescents.
The effects of
age, IQ, ADHD symptoms,
pubertal stage and substance abuse were removed by fitting a linear model (with no interactions) and the residuals of the model were plotted against callous - unemotional traits
Thus, we controlled for three level 1 variables (
age,
pubertal status, and treatment delivery method), two level 2 variables (baseline social status and baseline BMI), and the interaction between
age and BMI in cross-sectional multilevel models.
Other potential confounding factors include: substance use and
age /
pubertal stage of the sample.