Eriksson, B.O., Karlsson, J. and Saltin, B. (1971) Muscle metabolism during exercise in
pubertal boys.
In addition, only
pubertal boys had lower IGF - 1 concentrations, which were also related to various urinary phthalate metabolites.
Previous studies have looked at the effect of smoking marijuana in adult rats and humans but this is the first time that the effects have been looked at in
pubertal boys.
Not exact matches
Obesity and the
pubertal transition in
boys and girls.
Oral Abstract Presentations Amy Shealy — «Novel Microduplication of 12q13.12 Including TUBA1A and DHH Detected in a
Boy with Abnormal Brain MRI Findings, Cryptorchidism, Urethral Stricture and
Pubertal Delay» Marissa Smith — «Expanding Genetic Counseling Services to an Executive Health Program: Identifying Risk for Genetic Disease in a Low - Risk Population» Jessi Moline — «Approach to Systematic Screening of Endometrial Cancers for Lynch Syndrome: the Cleveland Clinic Experience» Brandie Leach — «A Time Study of Genetic Counselor Only versus Traditional Care Genetic Counselor / Geneticist Patient Care in a Cancer Genetics Setting» Jill Polk — «Ethico - legal Obligations in Hereditary Diffuse Gastric Cancer Kindred with Novel CDH1 Mutation»
Circadian cortisol rhythms in healthy
boys and girls: Relationship with age, growth, body composition, and
pubertal development
The current study examined the joint contributions of
pubertal maturation, parental monitoring, involvement in older peer groups, peer dating, and peer delinquency on dating in a sample of early adolescent
boys and girls.
The physical changes of
pubertal growth typically initiate a redefinition of self among
boys and girls, with an emphasis on body image.
Summary of hierarchical regression analyses testing peer - and teacher - reported popularity among
boys as a mediator of the link between
pubertal timing and rumors
Depression was associated with more mature
pubertal status and early timing (both actual and perceived) in girls, but with less mature
pubertal status and late timing (actual and perceived) in
boys.
While off - time
pubertal development has emerged as a potential risk factor for both symptoms of depression and anxiety in youth, the literature is mixed and inconsistent as to (1) how early versus late
pubertal timing confers risk for both
boys and girls, (2) if the conferred risk is distinct between symptoms of anxiety and depression, and (3) under what social contexts (e.g., family environment, peer relationships) off - time
pubertal development may emerge as a potent risk factor for these symptoms.
Results suggest that the contextual amplification process of early
pubertal timing may occur in both high stress family and peer environments and impact both girls and
boys.
The first model (dashed arrows) predicts that popularity among
boys at the start of middle school will partially mediate the link between earlier
pubertal development and rumor nominations received from peers in the spring of 6th grade.
We examined whether — with progressing
pubertal development — a stronger increase in body dissatisfaction could still be found for girls than for
boys.
In this longitudinal study (three family visits; time interval: 1 year),
pubertal development, body satisfaction, and desired body changes of 106
boys and 108 girls were assessed.
In this longitudinal study (three family visits; time interval: 1 year),
pubertal development, body satisfaction, and desired body changes of 106
boys and 108
In
boys who reported low
pubertal status, both NCS (β =.48, p =.001) and stressors (β =.27, p =.002) were significantly associated with depressive symptoms.
The two - way interaction age (or
pubertal status) by gender (included in all models under test) was not significant; whereas it would be expected that girls report more depressive symptoms as level of maturation (age /
pubertal status) increases compared to
boys.
Early
pubertal timing is only positively associated with baseline levels for
boys (p <.01).
Results showed that although the four - way interaction between NCS, stressors, gender, and age was significant while the four - way interaction with
pubertal status was marginally significant, the interpretation of these interactions was largely similar, i.e., NCS and depressive symptoms were significantly related only in the presence of many stressors in middle to late adolescent
boys (or in
boys reporting a high
pubertal status).
NCS was more strongly related to depressive symptoms in
boys with high
pubertal status that reported many (mean +1 SD) stressors (β =.73, p =.16) compared to
boys with high
pubertal status reporting few stressors (β =.21, p =.74).
The four - way interaction between NCS, stressors,
pubertal status, and gender approached significance (β = −.10, p =.08), indicating that the interaction between NCS and stressors was only significant in
boys who perceived their
pubertal status as high (β =.42, p =.007).
Our goals were (1) to compare the trajectory of depressive symptoms among
boys and girls from childhood into adolescence; (2) to analyze the role of genetic, shared, and unique environmental factors in depression among prepubertal and
pubertal male and female twins; and (3) to investigate a possible common etiology between liability to depression and one salient index of the child's environment: past - year life events.