This makes it a common target for antidepressants, which block serotonin from being
reabsorbed by neurons after it has dispatched its signal, so more of it stays floating around the brain.
Not exact matches
Many antidepressants that target serotonin work
by blocking serotonin transporters that
reabsorb the neurotransmitter into a
neuron, so it can be reused after it has sent a chemical signal.
Three basic mechanisms have been proposed over the years to explain the formation of specific neural circuitry: (1) an elaborate predetermined program encoded genetically in each
neuron that unfolds according to rigid and unmodifiable rules, (2) a random process of trial and error in which growing nerve fibers that make the right connections are consolidated and those that fail are
reabsorbed and (3) a general program of circuit formation that is brought to completion
by an interplay between genetic and extrinsic factors.
SSRIs are thought to reduce depressive symptoms
by increasing levels of serotonin, more specifically, making sure that serotonin stays between the
neurons and is not
reabsorbed to quickly
by the nerve cell.