Sentences with phrase «reduce reactive oxygen»

In rodents, ketogenic diets reduce reactive oxygen species in the brain34 and reduce central inflammation and reactive oxygen species in a model of multiple sclerosis.35 Two clinical papers have found that ketogenic diet feeding of 12 weeks to 6 months reduced signs of liver inflammation in obese patients with nonalcoholic fatty liver disease (in addition to improving various other physiological and biochemical variables).36, 37 Unfortunately, basic research into non-alcoholic fatty liver disease has been hampered by species differences between mice and humans in their hepatic reaction to ketogenic diets.38

For this reason, it is very anti-inflammatory and can reduce reactive oxygen species by increasing oxygen levels.

Not exact matches

Finally, while the underlying mechanism is unclear, the team found that having reduced NOX2 - derived reactive oxygen species also raised the risk for these autoimmune diseases.

When potent oxygen radical scavengers such as cerium oxide nanoparticles (nanoceria) were combined with a highly charged polymer (polyacrylic acid) and incorporated into extracted chloroplasts using the LEEP process, damage to the chloroplasts from superoxides and other reactive oxygen species was dramatically reduced.

In medicine, they could also be used to encapsulate oxygen - sensitive active compounds and to reduce the inflammation caused by reactive oxygen species (ROS) in biomedical applications.

The Einstein team suspected that cysteine was helping to kill TB bacteria by acting as a «reducing agent» that triggers the production of reactive oxygen species (sometimes called free radicals), which can damage DNA.

Calorie restriction with proper nutrition seems to help extend lifespans and delay the onset of age - related disorders by reducing what are called reactive oxygen species in the body.

Also, in both cell culture and a mouse model of the condition, blocking the excessive splintering reduced production of reactive oxygen species, a potentially harmful byproduct of metabolism, as well as cell death.

Uroporphyrinogen I is a reduced porphyrin that can undergo spontaneous oxidation to form the potent photosensitizer uroporphyrin I. Intense light exposure causes porphyrin molecules to generate reactive oxygen species (ROS).

The prevailing theory is that this reduces damage to mitochondria occurring as a result of the reactive oxygen species generated within these organelles, with localized antioxidants soaking up reactive molecules before they can cause harm.

CLK1: Reduced CLK1 activity can extend life in mice due to altered mitochondrial function and consequently lowered generation of reactive oxygen species.

Moreover; ultra-violate radiation is known to reduce the immunity and stimulates formation of reactive oxygen species in melanocytes locally [27].

Research had shown that increased mitochondrial activity may be at least partly responsible for extending the life span of yeast, roundworms, fruit flies and some mammals — perhaps by reducing the production of disease - causing reactive oxygen species (ROS).

Specifically, the research demonstrates that grape seed not only damages cancer cells» DNA (by means of an increase of reactive oxygen species), but also prevents the pathways which allow repair (as seen by reduced amounts of the DNA repair molecules Rad51 and Brca1 as well as DNA repair foci).

Inflammation is also reduced during this process through the reduction of reactive oxygen species (ROS) by up to 73 percent.

Acai's seems to inhibit chemically reactive molecules that contain oxygen, reducing oxidative stress and reversing inflammatory processes.

They've also been shown to help decrease DNA damage by reducing inflammation (the underlying cause of many diseases) and reactive oxygen species.

All of these happen because the free radicals and reactive oxygen species produced caused significant damage to the thyroid and reduced thyroid hormone production.

It boosts the immune system, protects vision, stops the build - up of oxidised fats, and helps to convert carbohydrates into energy and most crucially reduces the damage done to important cell components caused by reactive oxygen molecules such as free radicals and peroxides7.

Pretreatment of cells with Cy -3-G reduced the intracellular production of reactive oxygen species, the activation of JNK, and attenuated H (2) O (2)- or TNF - alpha - induced insulin resistance in a dose - dependent manner.

Spectra, through research, has been reported to decrease ROS (reactive oxygen species, decrease extracellular H2O2 (hydrogen peroxide), and reduce TNFα - induced inflammatory response in humans.

Both antioxidant compounds work reducing the proliferation of reactive oxygen species in cells, preventing mutations and dead due to oxidative stress.

Reduce the harmful byproducts of carbohydrate metabolism called Reactive oxygen species (ROS).

Inflammation in the intestines produces damaging reactive oxygen species (ROS) which initiate DNA damage, reduces oxygen flow to tissue and results in the further generation of pro-inflammatory cytokines and injury.

For example, KBs were recently reported to act as neuroprotective agents by raising ATP levels and reducing the production of reactive oxygen species in neurological tissues, 80 together with increased mitochondrial biogenesis, which may help to enhance the regulation of synaptic function.80 Moreover, the increased synthesis of polyunsaturated fatty acids stimulated by a KD may have a role in the regulation of neuronal membrane excitability: it has been demonstrated, for example, that polyunsaturated fatty acids modulate the excitability of neurons by blocking voltage-gated sodium channels.81 Another possibility is that by reducing glucose metabolism, ketogenic diets may activate anticonvulsant mechanisms, as has been reported in a rat model.82 In addition, caloric restriction per se has been suggested to exert neuroprotective effects, including improved mitochondrial function, decreased oxidative stress and apoptosis, and inhibition of proinflammatory mediators, such as the cytokines tumour necrosis factor - α and interleukins.83 Although promising data have been collected (see below), at the present time the real clinical benefits of ketogenic diets in most neurological diseases remain largely speculative and uncertain, with the significant exception of its use in the treatment of convulsion diseases.

The hydrogen was lost to space leaving highly reactive oxygen to combust reduced carbon (C and CH4) and sulphur (inc sulphide) to acid gases as well as oxidise near surface elemental or ferrous iron to ferric (red) iron.