Sentences with phrase «reduced use of glucose»

Follow - up tests a year later showed that the reduced use of glucose by the temporal lobe and posterior cingulate had been reversed in all six people (Annals of Neurology, DOI: 10.1002 / ana.22089).

Not exact matches

Using PET brain scans, they showed that those with impaired illness awareness also had reduced glucose uptake in specific brain regions, even when accounting for other factors linked to reduced glucose uptake, such as age and degree of memory loss.
PET scans demonstrated an increase in the brain's use of glucose, an indication of increased brain activity, while mental - status tests showed a slowing of the patients» rate of cognitive decline was reduced by 36 to 51 percent.
Metformin is a biguanide, a drug that reduces the amount of glucose that the liver makes and improves the body's use of insulin.
Talk to your doctor before taking aloe, especially if you use it as a laxative — it could lower blood glucose levels and reduce the effectiveness of medication when taken orally.
Many of those who have tried incorporating amla into their diet have already experienced reduced blood glucose and cholesterol levels within the first month of using Amla Green on a daily basis, and we are very excited to continue promoting improved cardiovascular health.
All doses significantly reduced blood glucose values - and using only 1 gram of amla per day was just as effective as Glimepiride.
If the excess blood sugar isn't used, shortly thereafter our pancreas reacts to reduce the high the blood sugar by increasing production of insulin and stores the extra glucose as fat.
A drastic reduction in your carbohydrate intake reduces the availability of glucose and initiates a chemical change with your body using fat instead of glucose for energy.
As a result, one could argue that things would run the opposite way than Adele proposes: reducing dietary glucose, which generally does not reduce blood glucose levels, will not affect cancer metabolism, but will limit availability of glucose to normal cells for structural use.
That hormone triggers the breakdown of fat for energy use, reducing insulin levels and other metabolic markers of glucose metabolism.
It improves the use of glucose within the body and reduces insulin resistance5.
Either (1) go on a starvation diet for the rest of your life or (2) make your body use fat as fuel by reducing its only other fuel, namely, glucose on most days of the week.
There are numerous studies reflecting the safety and superiority of a ketogenic diet and its use for reducing cardiovascular risk factors in adults and children including artery plaque, c - reactive protein, BMI (body mass index), blood glucose, triglycerides, LDL and HDL cholesterol, body fat and diastolic blood pressure without increases in inflammation or oxidation (8, 9, 10, 11, 12, 13, 14, 15).
The use of vitamins, minerals, omega - 3 oils, nutraceuticals, and botanicals is important as many patients are nutrient deficient, or have bodies that need help having more energy, lowering their glucose levels, having better moods, getting better sleep, reducing inflammation, enhancing their antioxidant status, controlling the appetite, helping the other Essentials, and so forth, actions that supplements can support.
In this case we are suggested to take about 200 mcg of chromium as it is used in insulin regulation of blood glucose and is important for balancing blood sugar levels thus helping reduce sugar cravings.
Eating up to 20 % of calories from carbs doesn't require the use of disposal pathways — glucose can be stored as glycogen and then released as needed, so the effect of dietary glucose is primarily to reduce the amount of gluconeogenesis.
Once the carbohydrate has worked its way through your digestive system, been reduced to a simple sugar such as glucose, and reached the bloodstream through the processes of absorption, it's taken to the liver where it's either distributed to the cells for energy, or stored for later use.
Adhering to these traditional concepts the US Department of Agriculture has concluded that diets, which reduce calories, will result in effective weight loss independent of the macronutrient composition, which is considered less important, even irrelevant.14 In contrast with these views, the majority of ad - libitum studies demonstrate that subjects who follow a low - carbohydrate diet lose more weight during the first 3 — 6 months compared with those who follow balanced diets.15, 16, 17 One hypothesis is that the use of energy from proteins in VLCKD is an «expensive» process for the body and so can lead to a «waste of calories», and therefore increased weight loss compared with other «less - expensive» diets.13, 18, 19 The average human body requires 60 — 65 g of glucose per day, and during the first phase of a diet very low in carbohydrates this is partially (16 %) obtained from glycerol, with the major part derived via gluconeogenesis from proteins of either dietary or tissue origin.12 The energy cost of gluconeogenesis has been confirmed in several studies7 and it has been calculated at ∼ 400 — 600 Kcal / day (due to both endogenous and food source proteins.18 Despite this, there is no direct experimental evidence to support this intriguing hypothesis; on the contrary, a recent study reported that there were no changes in resting energy expenditure after a VLCKD.20 A simpler, perhaps more likely, explanation for improved weight loss is a possible appetite - suppressant action of ketosis.
This data suggests a different effect of ketosis on glucose homeostasis in diabetic and non-diabetic individuals.21 Other studies support the long - term efficacy of ketogenic diets in managing complications of T2D.36, 37 Although significant reductions in fat mass often results when individuals restrict carbohydrate, the improvements in glycaemic control, haemoglobin A1c and lipid markers, as well as reduced use or withdrawal of insulin and other medications in many cases, occurs before significant weight loss occurs.
For example, KBs were recently reported to act as neuroprotective agents by raising ATP levels and reducing the production of reactive oxygen species in neurological tissues, 80 together with increased mitochondrial biogenesis, which may help to enhance the regulation of synaptic function.80 Moreover, the increased synthesis of polyunsaturated fatty acids stimulated by a KD may have a role in the regulation of neuronal membrane excitability: it has been demonstrated, for example, that polyunsaturated fatty acids modulate the excitability of neurons by blocking voltage-gated sodium channels.81 Another possibility is that by reducing glucose metabolism, ketogenic diets may activate anticonvulsant mechanisms, as has been reported in a rat model.82 In addition, caloric restriction per se has been suggested to exert neuroprotective effects, including improved mitochondrial function, decreased oxidative stress and apoptosis, and inhibition of proinflammatory mediators, such as the cytokines tumour necrosis factor - α and interleukins.83 Although promising data have been collected (see below), at the present time the real clinical benefits of ketogenic diets in most neurological diseases remain largely speculative and uncertain, with the significant exception of its use in the treatment of convulsion diseases.
In Alzheimer's patients, there appears to be a reduced ability to use glucose for energy in certain parts of the brain.
If a dog is chronically ill, then a short - acting insulin is used to reduce the risk of hypoglycemia (low levels of glucose in the blood).
ProZinc is a protamine zinc insulin, a type of long - acting insulin derived from recombinant human insulin used to reduce hyperglycemia (high blood glucose or sugar) in cats with diabetes mellitus.
The impact of the dietary carbohydrate source on food intake, glucose and insulin concentrations, and insulin sensitivity in overweight cats with reduced insulin sensitivity was assessed (57) using two diets formulated to contain similar starch content (33 %) from different cereal sources (sorghum and corn vs. rice).
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