An extensive review of the unique and interactive functions across neurobiological regions is beyond the scope of this brief report, and thus we highlight several specific and robust examples in which emotion
regulation difficulties manifest as neurobiological differences and help explain risk for comorbid disorders among youths.
Not exact matches
These observed differences in neurological activity, consistent with the dual systems model, contribute to increased emotional volatility and
difficulty with emotion
regulation that increases during adolescence, which ultimately
manifests as increased risk for SUDs and comorbid psychopathology (e.g., [29 • •, 36]-RRB-.
Indeed, disordered and dysregulated mood defines many forms of psychopathology, and
difficulty with emotion
regulation has been described as a core deficit that emerges across psychiatric disorders and
manifests as dysregulation across multiple levels of analysis — biology, physiology, and behavior [15].