Sentences with phrase «release reactive oxygen»

Let me explain... Through normal metabolism of nutrients, your mitochondria release reactive oxygen species (ROS).

Scientists have long assumed that wounded mitochondria release reactive oxygen molecules, which then damage DNA and proteins, increasing disease risk.

Apparently, a side reaction in the respiratory system in the mitochondria causes them to steadily release reactive oxygen species that can damage DNA, RNA and proteins.

The immune system may kick in when animals eat, releasing reactive oxygen molecules to kill microbes on food, helping to protect from disease.

This is a key component of the immune system, when the body releases reactive oxygen species to fight against invading bacteria.

Not exact matches

The oxygen species released in the process «won't be super reactive,» she said.

Oxidative states are generally considered to be indicative of cellular stress; however, cells inherently release harmful reactive oxygen species during energy production, neutralized by intracellular antioxidative buffering systems.

The finding suggests that microbes with the ability to produce oxygen were prolific at least locally around 3.46 billion years ago, releasing large quantities of this reactive molecular gas into the oceans and eventually the atmosphere by the end of this period (more).

Now in a study published in the journal Scientific Reports, Alexander Zaika, Ph.D. and coworkers show that DNA damage in the esophageal cells caused by acidic bile reflux (BA / A) activates enzymes called NADPH oxidases in the mitochondria, the cell's power house, to release highly reactive - oxygen species (ROS).

This primary response involves the production of reactive oxygen species (ROS), which are molecules that can act like tiny bombs when released by the plant cell at the offending pathogen.

Furthermore, the manganoporphyrin was stably contained within the microcapsule without release, and researchers showed that both manganoporphyrin and tannic acid were required for the synergistic scavenging of reactive oxygen species.

The release of reactive oxygen species is also common in inflammaging, and these substances cause oxidative damage to cells and tissues, releasing debris that further elicits inflammation.

However, the latter is preferred because it is a cleaner, healthier fuel, as it releases far fewer reactive oxygen species (ROS) and secondary free radicals.

Free radicals are released by cellular mechanisms responding to impact and cardio - muscular stress - specifically reactive oxygen and nitrogen species (RONS).

The reactive oxygen species and free radicals released from such reactions like lipid peroxidation can progressively damage neurons.

Cadmium interacts with cells of the immune system (neutrophils and macrophages) and promotes the release of free radicals such as reactive oxygen species.

LTB4 increases edema and chemotaxis, induces release of lysosomal enzymes, increases reactive oxygen species, and enhances production of the cytokines TNF - α, IL - 1, and IL - 6.»

The mitochondria in our cells are responsible for releasing energy from the molecules in our food, but they also unleash electron - stealing free radicals like reactive oxygen and reactive nitrogen species.

Some of the proven mechanisms of action include release of endogenous opioids; stimulation of angiogenesis; reduction of the formation of fibrosis; reduction of inflammation; blockage of the effects of reactive oxygen species (ROS) release; neuronal regeneration and functional recovery; enhancement of wound closure; improvement of wound epithelialization, cellular content, granulation tissue formation, and collagen deposition; and bactericidal effects.