Although we are now living in a drastically different world, the body hasn't yet learnt to differentiate between physical and emotional or mental threats; it still
releases excess glucose into the bloodstream to provide energy for running or fighting, despite the fact you're likely dealing with a demanding email and downing your third caffeine hit for the day.
Not exact matches
This guarantees three things: there is no
excess glucose that can be stored as body fats, there is a reduction in the levels of insulin
released, and there is no possibility of developing a resistance to insulin.
Your pancreas
releases the hormone Insulin, which takes the
excess glucose and (1) puts it in needy muscle cells, or (2) puts in empty liver cells, or (3) takes all the leftovers and remakes the
glucose into TRIGLYCERIDES.
Insulin resistance can cause the liver to produce
excess glucose, which then causes even more insulin insensitivity and can eventually result in type II diabetes, and IGF - 1 can decrease the need for this type excessive insulin
release.
Excess glucose is stored in the liver; when needed to sustain blood sugar between meals, the liver
releases sugar and the pancreas responds with more insulin to help it enter cells.
The pancreas has to produce more insulin to dispose of the
excess glucose that the liver is
releasing.
When that reserve becomes full both the muscles and the liver send a signal to stop insulin production and
excess glucose from dietary carbs begins to build up in the bloodstream, calling for more and more insulin to be
released to remove it.
The loss of insulin sensitivity contributes to
excess insulin
release — as the body tries to force serum
glucose into cells.
Determined that the product of the pathway iL1b was
released with
excess concentrations of
glucose as well as Palmitate.