Sentences with phrase «retinal cells called»

Vision in low light is dependent on retinal cells called rods.

Working with researchers at the Friedrich Miescher Institute for Biomedical Research in Switzerland, the MIT team also tested Jaws's ability to restore the light sensitivity of retinal cells called cones.

Retinitis pigmentosa is an inherited retinal degenerative disease that causes slow but progressive vision loss due to a gradual loss of the light - sensitive retinal cells called rods and cones.

Working with rats and mice, Xi and colleagues found that specialized retinal cells called Müller glial cells release IL - 33 in response to retinal injury.

«Overlooked cell key player in preventing age - related vision loss: Tree - shaped retinal cells called Müller glia may provide a new therapeutic target for treating degenerative eye diseases.»

The retinal cells called cones come in three varieties.

Two of his math professors were collaborating with physiologist Joshua Singer, also at Northwestern, who was keen to model the biology of a retinal cell called the AII amacrine interneuron.

Not exact matches

From the embryonic stem cells, the researchers produced a type of tissue called retinal pigment epithelium (RPE).

A layer of cells called the retinal pigment epithelium (RPE) is essential for supporting and nourishing the retina cells that capture light for vision.

They further demonstrated that the cells — called intrinsically photosensitive retinal ganglion cells, or ipRGCs — could detect light.

In a developing fetus, she surmised, those phone calls would reach groups of nerve cells in the LGN; as their synaptic connections with the retinal neurons strengthened, the LGN neurons would begin forming «area codes» of their own.

Now, an Australian team reports evidence that the computations take place in retinal neurons called ganglion cells (Science, 29 September, p. 2347).

Next, they used a technique called fluorescence - activated cell sorting to separate out the newly differentiated retinal ganglion cells from a mixture of different cells into a highly purified cell population for study.

In an experimental setup that allowed the two types of cells to bathe in the same fluid without coming into physical contact, retinal neurons in a bath with hUTCs formed new connections between neurons called synapses, and they sprouted new «neurites» — tiny branches that lead to additional connections.

It first targets a protective lining called the retinal pigment epithelium (RPE), which shuttles nutrients to the photoreceptor cells and is vital for their survival.

Scientists have known of the existence of these nerve cells, called melanopsin - containing retinal ganglion cells (mRGCs), since 2000.

A new gene therapy treatment has restored some sight in a handful of blind patients suffering from Leber's congenital amaurosis, a syndrome in which, because of a broken or missing gene called RPE65, retinal photoreceptor cells malfunction and eventually die.

When these photoreceptors detect light, they send a signal to specialized neurons in the retina called retinal ganglion cells, or RGCs, which then transmit visual information to the brain by firing electrical pulses along the optic nerve.

In vertebrates, nerve cells called retinal ganglion cells send information from the retina to vision - processing centers in the brain.

That news alone was an exciting breakthrough, but there's more: In lab mice, damaged retinal ganglion cells survived longer and were able to regenerate when excess zinc was removed through a chemical process called chelation.

DHA in brain and retinal cells also builds reservoirs for molecules called into action when normal functions are disrupted, resulting in such conditions as retinal degeneration, Parkinson's or Alzheimer's disease.

Here the so - called bipolar cells play a central role, as the first retinal layer to process the output of the light - sensitive cells in the eye.

As neuroscientists David Hubel and Torsten Wiesel of Harvard University discovered in a series of groundbreaking experiments in the 1960s, individual cells in the visual cortex, so - called binocular cells, receive input from both eyes, specifically from corresponding retinal locations, thus providing a mechanism for perceptual fusion.

First, researchers found a pigment called melanopsin in a small subset of retinal ganglion cells (RGCs) in the eyes of rats.

The technique developed is called DARC, which stands for detection of apoptosing retinal cells.

In research published this week in the Journal of Neuroscience, University at Buffalo scientists and colleagues focused on a particular protein, called a transcription factor, that regulates gene activity necessary for the development of one type of retinal neuron, the horizontal cells.

As you mentioned, in dry AMD we basically have atrophy in the back of the retina in cells that are called retinal pigment epithelial cells.

What we discovered was that was in fact there are a certain group of cells in the retina, retinal ganglion cells, and a particular group of those ganglion cells which we call OFF cells.

In addition to differentiated retinal cells, the amphibian eye contains a population of self - renewing retinal stem cells located in the retinal periphery in a region called the ciliary marginal zone or CMZ.

We hypothesize that upon retinal neuronal damage MG or RPE cells undergo defined and controlled changes in cellular and molecular phenotype towards a cell with progenitor properties — this process that we are studying we call regenerative reprogramming.

Those are understandably called the «on» retinal ganglion cells and «off» retinal ganglion called.

Behind the photoreceptors is another layer of cells called retinal pigment epithelium (RPE), which support the rods and cones by delivering nutrients from the bloodstream and removing waste that the rods and cones generate.

The treatment could help people who have a fault in a gene called RPE65, which causes problems in the retinal pigment epithelium (RPE), a thin layer of cells that support and nourish photoreceptors.

The researchers wanted to understand how retinal cells survive the initial vascular damage, and found that they go into a self - preservation mode called senescence.

Eventually the paintings emerge, but the little bubbles, as we call them, are stimulating specific types of retinal ganglion cells in very specific ways, which is necessary, we know from other studies, to encourage their ongoing health by transmitting electrical signals down their axons to the brain.

The stem cell approach I am most excited about involves a treatment to replace cells called retinal pigment epithelial cells.

The retinal cells that help us see in bright light are called cones, and these are not destroyed by the disease itself, but by the toxic by - products released by the rod cells as they die.