For instance, greater vigilance to negative information may make one hesitant to express their thoughts and feelings in social contexts or behave in an assertive manner, making it difficult to form and maintain meaningful social relationships in individualistic societies, a social behaviour critical to reducing
the risk of affective disorders for genetically susceptible individuals.
Major depression in childhood or adolescence increases
the risk of affective disorder in adulthood.
Not exact matches
Postpartum
affective disorder (AD), including postpartum depression (PPD), affects more than one in two hundred women with no history
of prior psychiatric episodes, and raises the
risk of later
affective disorder for those women, according to a new study published in PLOS Medicine by Marie - Louise Rasmussen from Statens Serum Institut, Denmark, and colleagues.
have found that good levels
of vitamin D are associated with a lessened
risk for Seasonal
Affective Disorder (SAD) and depression.
Studies have found that good levels
of vitamin D are associated with a lessened
risk for Seasonal
Affective Disorder (SAD) and depression.
In the general population, the most frequent
of these is the combination
of alcohol use
disorder and depression and / or anxiety
disorder.5 — 7 Comorbidity
of alcohol abuse and dependence is two to three times higher for those who suffer from depression than for those in the general population.8 Moreover, risky alcohol use is associated with a higher probability
of developing
affective disorders than for not at -
risk users.9
Community approaches, such as home visitation, have been shown to be highly successful in changing the behavior
of parents at
risk for perpetrating maltreatment.18 Targeted programs for mothers with
affective disorders and substance abuse have also been shown to be useful in preventing psychological maltreatment.19, 20
Patients with psychiatric
disorders, particularly
affective disorders, had an increased
risk of developing dementia
The areas
of focus include: primary
risk factors (
affective disorders, previous suicide attempts, hopelessness); secondary
risk factors (substance abuse, personality
disorders); situational
risk factors (family functioning, social relationships, exposure to suicide, life stressors, sexual orientation); and protective factors or strengths (individual, family, social, and community resources).
The depressed group was at an increased
risk for
affective disorder in adult life and had elevated
risks of psychiatric hospitalization and psychiatric treatment.
Risk of dementia in people with depressive and bipolar
disorders increases with increasing number
of prior
affective episodes
Possibly, mania is a more purely biologically driven phenomenon than bipolar depression, with onsets more readily attributable to medication inconsistency, sleep deprivation, circadian disruption, or behavioral activation.21,22,84 - 86 In contrast, social and familial support has been found to protect against depression in bipolar and unipolar
affective disorders, but the role
of these variables in manic recurrences is unclear.86 - 88 An analysis
of laboratory interactional data from a subset
of 44 families in this sample revealed that treatment - related improvements in family communication skills were more closely associated with reductions in patients» depressive than manic symptoms.56 Thus, manic and depressive symptoms may be influenced by different constellations
of risk and protective factors.
Based on the literature in older children, it was hypothesized that preschoolers with a greater family history
of affective disorders, who experienced more stressful life events, or who had greater comorbidity would be at an increased
risk for recurrent and more severe depressive episodes during a 24 - month period.
While other
risk factors for later MDD were found, early MDD itself and family history
of affective disorder were the most powerful
risk factors for later MDD.
Preschool MDD as well as family history
of affective disorders emerged as the most robust predictors
of later MDD compared with other
risk factors considered simultaneously in the model.
Patients were excluded if they (a) were currently receiving psychotherapy or antidepressant drugs (unless they had been taking the same dose for at least three months without improvement); (b) were unwilling to accept randomisation or were unavailable for follow up; (c) met criteria for severe depression (melancholia) or had a history
of bipolar
affective disorder, schizophrenia, or substance misuse (as defined in the Diagnostic and Statistical Manual
of Mental Disorders, third edition, revised (DSM - III - R) 18); or (d) were at significant
risk of suicide or in need
of urgent psychiatric treatment.
Several studies
of inpatients have noted that depressed individuals are significantly less likely to be violent than individuals with other types
of disorders.8 - 10, 23 Moreover,
affective disorders were not found to be related to an increased
risk for homicide in a Finnish cohort study.15 By contrast, other studies have found a relationship between
affective disorders and homicide, 24 self - reported violent behavior, 2 and conduct
disorder in childhood and adolescence.25 One possible explanation for these conflicting results could be the potential moderating role
of alcohol abuse in this relationship.
Children
of parents with bipolar
disorder: A population at high
risk for major
affective disorders
Social influences
of early developing biological and behavioral systems related to
risk for
affective disorder
The impact
of high neuroticism in parents on children's psychosocial functioning in a population at high
risk for major
affective disorder: A family — environmental pathway
of intergenerational
risk