I am hoping so, because of its positive effects on glucose sensitivity, stability and
the second meal effect, but it's not necessarily so unless it's been tested in ketogenic subjects.
See Beans &
the Second Meal Effect.
How much lentils / beans / peas must one consume to experience
the second meal effect.
This second meal effect is really useful.
If you check out my 3 - min video Beans and
the Second Meal Effect, you can see that as the subjects» bean to rice ratio increases, cardiometabolic risk factors continually improve.
It has since been dubbed the «
second meal effect.»
Scientists call it «
the second meal effect,» where this biofilm slows glucose absorption from minutes to hours.
Could white potatoes (cooled), normally associated with spikes in blood sugar and insulin, have
any second meal effect >
Is there any kind of
second meal effect after tomatoes, raisins etc.?
At the time they called it the «lentil effect,» but chickpeas appear to work just as well, so it has since been dubbed the «
second meal effect.»
This study links the «
second meal effect» to the glycaemic index (the amount that food raises blood glucose) of the evening meal.
It also has a «
second meal effect» - meaning that if you eat resistant starch with breakfast, it will also lower the blood sugar spike at lunch (28).
I thought fibre - rich resistant starch was the most satiating, especially because of its «
second meal effect».
It also has a «
second meal effect» - meaning that if you eat resistant starch with breakfast, it will also lower the blood sugar spike at lunch (28).
First and
second meal effects of pulses on blood glucose, appetite, and food intake at a later meal.
Not exact matches
Second, I tend to agree with your assumptions about mandates and obesity — for example, the regulations require that fat calories be limited to 30 percent of the
meal but over the time period that's been in
effect, we've only seen an increase, not a reduction, in childhood obesity.
Interestingly, resistant starch even contributes to a «
second -
meal effect» whereby it has the potential to regulate blood sugar and insulin responses in a subsequent
meal.
A
meal's ability to diminish the glucose response to carbohydrates eaten during the following
meal is known as the «
second -
meal effect» (SME).
Conclusion: Fermentable carbohydrates, independent of their
effect on a food's glycemic index, have the potential to regulate postprandial responses to a
second meal by reducing NEFA competition for glucose disposal and, to a minor extent, by affecting intestinal motility.
The LGI breakfast rich in amylose may have exerted its
effect partly by ameliorating the preprandial metabolic status of the
second meal, as originally suggested by Jenkins et al (4) and Wolever et al (5), but this possibility is excluded in the case of the HGI - Lac breakfast.
This phenomenon was named the «
second -
meal effect.»
Jenkins et al (4) and Wolever et al (5) described this phenomenon as «the
second -
meal effect» (SME) and identified the GI of the
meal preceding the
second meal as the determinant of the improved glucose tolerance observed at the subsequent food consumption.
Second -
meal effect: Low - glycemic - index foods eaten at dinner improve subsequent breakfast glycemic response.
Second -
meal effect: low - glycemic - index foods eaten at dinner improve subsequent breakfast glycemic response