T lymphocytes and NH cells in the asthmatic lung produce proteins named interleukin (IL)-5 and IL - 13 that are responsible for
severe airway inflammation.
Other researchers have shown that while the immune response Th1 is necessary to clear such viruses, another response, Th2, which is associated with
severe airway inflammation and the development of post-infection asthma, could arise after viral infection.
Not exact matches
Chronic
airway inflammation is characterized by the infiltration of EOS, an increase in serum immunoglobulin (Ig) E and excessive secretion of
airway mucus (4 — 6), which result in
airway structural changes and may even develop into refractory asthma or
severe asthma (7).
These serve to help with daily prevention and also manage more
severe episodes as they occur by reducing
inflammation and helping to relax the muscles of the
airway.