Now Cecilia Lai, Simon Fisher and colleagues, reporting in todays Nature, show that a gene on chromosome 7, which they call FOXP2, is mutated in the affected members of the KE family, as well as in an unrelated patient with
a similar language deficit.
Reading research, he observes, has long shown that a key aspect of dyslexia is the child's difficulty in understanding the sounds of
language — a
deficit that is
similar to the faulty number sense seen in dyscalculia.