Not exact matches
Glioblastoma multiforme is the most common and deadliest of the
glial tumors because the
cells reproduce
so rapidly.
Some recent studies have suggested that
glial cells can secrete chemical messengers such as glutamate and ATP; if
so, these «gliotransmitters» could influence neuron activity.
«The most obvious function of
glial cells has been related to their role in forming scar tissue to prevent the spread of injury and neuronal degeneration, but
so much about their role in the brain is unknown.»
«There are more reactive
glial cells and fewer functional neurons in the injury site,» Chen said, «
so we hypothesized that we might be able to convert
glial cells in the scar into functional neurons at the site of injury in the brain.
The protein, called
glial cell - line derived neurotrophic factor (GDNF), was first isolated in 1993 and has
so far only been tested in animals.
But while substantial data (including AFFiRiS» [15] and Prothena's [20,22] preclinical work) give good reason to think that early clearance of AS neuropathology will decelerate the rate of SN DA neuronal loss, astrocytosis, and possibly accumulation of senescent
glial cells in PD, there is no reason to think that doing
so will prevent these phenomena entirely, if for no other reason than because many other factors also contribute to their occurrence.
There are many more
glial cells in the brain than neurons, and astrocytes are the most abundant of the glia,
so if you take a sample of brain tissue, you're fairly sure to get some astrocytes as part of the bargain.
The second big surprise was that the support
cells,
so called
glial cells, these are the
cells that surround ganglion
cells from their — where their
cell body is in the eye all the way back to the brain.
Again, going back to those Opioid receptors and
Glial cell which can help — help control the information in the brain, we think that Anxiety depressionism is inflammation in the brain,
so if you can do — do anything to reduce that inflammation, uh — it — it seems to help uhm — So, uh — uh — so — so — The way it works, uhm — The mechanism of action for the low - dose Naltrexone, is that you — you — you temporarily block the opioid receptor
so if you can do — do anything to reduce that inflammation, uh — it — it seems to help uhm —
So, uh — uh — so — so — The way it works, uhm — The mechanism of action for the low - dose Naltrexone, is that you — you — you temporarily block the opioid receptor
So, uh — uh —
so — so — The way it works, uhm — The mechanism of action for the low - dose Naltrexone, is that you — you — you temporarily block the opioid receptor
so —
so — The way it works, uhm — The mechanism of action for the low - dose Naltrexone, is that you — you — you temporarily block the opioid receptor
so — The way it works, uhm — The mechanism of action for the low - dose Naltrexone, is that you — you — you temporarily block the opioid receptors.
In fact, cholesterol is
so important that it is manufactured by the
glial cells in the supportive tissues of the brain.