Some scientists might explore how and why the neural circuits that detect threats and
store fearful memories sometimes behave in unusual ways after traumatic events — the kinds of changes that are partially responsible for post-traumatic stress disorder.
Not exact matches
Now, though, at the very moment in which a rat remembered the shock, the scientists injected an antibiotic that inhibits the synthesis of new proteins into its amygdala — a part of the brain long known to
store lasting
memories of
fearful experiences.
Pointing to the change in amygdala activity, which is central to the brain's system of
storing and recalling
fearful memories (see How Fear Works to learn about this process), the researchers say the
memory was not simply disconnected from fear, but that it was actually erased in its entirety.