Sentences with phrase «stress on cells»

Avoiding «wrong» training and creating too much stress on the cells is the other half of the picture.

Lower levels of antioxidants are likely to lead to damaging stress on cells and DNA throughout the body.

Oxidation — Stress on the cells and tissues cased by oxidation is one of the leading causes of aging.

Aging occurs chemically when we have too much oxidative stress on our cells, we don't have enough access to key micronutrients required for all parts of the body to function optimally, and when we start to make smaller concentrations of all of our hormones.

That could put stress on cells with weakened mitochondria, making them vulnerable to degenerative diseases such as Alzheimer's, geneticist Peter Larsen of Duke University and colleagues proposed February 24 in Alzheimer's & Dementia.

Of course one of the problems with it is there's a bit of shear stress on the cells which they have to resolve.

«It's possible that the culture conditions are causing increased stress on the cells, resulting in cell death.»

Unfolded proteins create stress on the cell, and you potentially create situations where you now put less assembled proteins in the plasma membrane.

Cellular functions create metabolic processes that generate oxidative stress on the cell.

Not exact matches

Doing so has a very direct positive effect on your biggest nerve [the sciatic nerve], which is your stress - reliever and what brings about cell regulation.»

Although much less research exists on monk fruit and its components, preliminary studies show promise for improving insulin signaling by protecting the pancreatic insulin - releasing cells from oxidative stress (3, 4).

DOn't give it too much sugar because it'll create too much stress on the yeast cells.

You can't stress enough the danger of texting or talking on a cell phone while behind the wheel of a car.

When cells become cancerous, tremendous stresses are placed on the cellular machinery responsible for maintaining protein equilibrium — and that machinery is the target of anti-cancer drugs called proteasome inhibitors.

The neurotransmitter acetylcholine is released by nerve cells in the brain when people or mice are under mild stress or concentrating on learning something new.

«By focusing on changing how infected cells respond to the resulting metabolic stress rather than targeting a component of the virus itself, there is less risk that the virus will become resistant to the drugs,» Thomas said.

The stress this places on cells leads to the overproduction of glucose, which when not used for energy transforms into lactic acid, which is difficult for the body to flush out.

Findings based on this new technology are likely to have a significant impact on basic cellular research and increase our knowledge of how cells work and respond to stresses, such as exposure to contamination.

In a study published today in Cell Metabolism, UT Southwestern researchers identify a hormone that acts on the brain to increase the desire to drink water in response to specific nutrient stresses that can cause dehydration.

«We hypothesize that older humans might be especially susceptible to the effects of sleep deprivation on the disruption of glucose homeostasis via cell stress.»

«Untangling the knots in cell stress: Study sheds new light on unfolded protein response.»

This metabolic demand makes brain cells particularly vulnerable to damage from oxidative stress, in which reactive oxygen species (ROS), sometimes called free radicals, exert toxic effects on cellular components.

The scientists zeroed in on the cells from the hippocampi of the cadavers, examining the switch for the stress hormone gene they had studied in rats.

«These findings build on a long history of work in our laboratory investigating mechanisms by which cancer cells respond to environmental stresses,» says Grant.

It's not feasible to study the effects of cosmic rays on real astronauts, such as those living in the International Space Station, because many variables, including the stress of living on a spaceship, can affect cognition, says Patric Stanton, a cell biologist at New York Medical College in Valhalla.

This activation causes additional stress which keeps the macrophages «turned on» in a cycle that amplifies and further damages the cells and surrounding tissues.

This gave them images showing how Bax proteins gathered on mitochondria of cells under stress in cell - death mode.

If a cell is under stress, Bax proteins deposit on the surface of mitochondria in symmetrical pairs.

Firstly, salt stress causes a delay in cell division, leading to synchronization of cell cycles; secondly, survival probability depends on the individual bacterial cell's position in the cell cycle at the time of the second exposure.

«I think what's needed for collective invasion will depend on the environmental stresses the cancer cells face, and it may shift with treatment,» Marcus says.

Sood and his team first studied the effects of stress hormones on human ovarian cancer cell anoikis in culture.

Their findings suggest that the protein plays a role in increasing stress on the endoplasmic reticulum, an organelle involved in transporting and processing materials within cells.

A study led by researchers at the University of Illinois at Chicago points blame at a regulatory molecule in cells called TRIP - Br2 that is produced in response to overeating's stress on the machinery cells use to produce proteins.

Now researchers reporting in the Cell Press journal Neuron on July 18 show in a study conducted in rats that even a single exposure to nicotine temporarily changes how the brain's reward system responds to alcohol and increases the reinforcing properties of alcohol via stress hormones.

To test whether the childhood infection and stem cell depletion had any effect on the adult heart, the researchers exposed infected mice to two different types of cardiac stress.

When enough RAP molecules adhere to enough target molecules, staph bacteria switch on their cell - to - cell communication and stress - response systems and begin producing the toxin that makes them so lethal.

Multiple components distributed on the chromosomes and megaplasmid that contribute to the ability of D. radiodurans to survive under conditions of starvation, oxidative stress, and high amounts of DNA damage were identified.Deinococcus radiodurans represents an organism in which all systems for DNA repair, DNA damage export, desiccation and starvation recovery, and genetic redundancy are present in one cell.

This fact makes the LCSB scientists» results of extraordinary interest to Parkinson's researchers worldwide, as Schwamborn stresses: «On our new cell cultures, we can study the mechanisms that lead to Parkinson's much better than was ever the case before.

Marson stressed that, while recent reports of CRISPR / Cas9 editing of human embryos have stirred up controversy, T cells are created anew in each individual, so modifications would not be passed on to future generations.

On larger surface areas, however, the performance of the cells falls considerably,» stressed Etxebarria, who has reached the following conclusion: to be able to manufacture cells with a large surface area it is necessary to build modules, to which cells with a smaller surface will be connected in series or in parallel, on the substrate itselOn larger surface areas, however, the performance of the cells falls considerably,» stressed Etxebarria, who has reached the following conclusion: to be able to manufacture cells with a large surface area it is necessary to build modules, to which cells with a smaller surface will be connected in series or in parallel, on the substrate itselon the substrate itself.

Scientists at the American Museum of Natural History and Gettysburg College found that this symbiosis, the only known example that includes a vertebrate species, puts stress on algal cells, changing the way they make energy, but does not seem to negatively impact salamander cells.

Even psychological stress can lead to counter-regulatory hormones, which lead to more insulin and more stress on the beta cells,» he says.

On the one hand, the researchers now understand which mechanisms the stressed T cells need to protect themselves against NK cells.

Other immune cells, the specific killer cells, which are also known as CD8 + T cells and multiply prodigiously and mature in response to an infection, can also exhibit stress symptoms and thereby potentially end up on the NK cells» hit list.

The demands of rapid cell division put a strain on cancer cells, and the approach works by tipping cell stress over the edge.

To coordinate their size and growth with current environmental conditions, cells rely on the mechanistic target of rapamycin complex 1 (mTORC1) pathway, which senses cellular stresses, growth factors, and the availability of nutrients, such as amino acids and glucose.

According to Whitehead Institute researchers, protein production or translation is tightly coupled to a highly conserved stress response — the heat shock response and its primary regulator, heat shock factor 1 (HSF1)-- that cancer cells rely on for survival and proliferation.

Her current work explores the effects of ER stress on intestinal stem cell function.

Researchers from immune biology professor Annette Oxenius's group have now discovered what keeps NK cells from killing off their «colleagues from the other department» of the immune system: healthy CD8 + cells are able to detect the immune messenger substance type 1 interferon, which binds to specific receptors on the surface of these immune cells and thereby conceals their stress.

CAMBRIDGE, Mass. — Protein production or translation is tightly coupled to a highly conserved stress response that cancer cells rely on for survival and proliferation, according to Whitehead Institute researchers.