The
study was supported by funds from the National Institutes of Health (R56 - AI089532 and RO1 - HL105704), an Abbott Viral Discovery Award, the QB3 Swartz Foundation Lyme Disease Grant, the National Research Fund for Tick - borne Diseases, a UCSF
Microbial Pathogenesis training grant and the CDC.
Published
studies have suggested that T1D results from environmental triggers acting on genetically susceptible individuals and that
microbial infection and their immunological consequences are suspected to take part in the
pathogenesis.