Treatment of rats with 6 - hydroxydopamine, a drug which destroys
sympathetic nerve terminals, leads to a decrease in serum dopamine - β - hydroxylase activity.
These data represent evidence that at least a portion of the circulating dopamine - β - hydroxylase activity arises from
sympathetic nerve terminals.
Not exact matches
Studies conducted by Hendry at the Australian National University and by K. Stockel and H. Thoenen at the Basel Institute for Immunology have demonstrated that NGF is taken up at the
terminal nerve endings of the
sympathetic fibers and transported back to the neuronal cell body along the axon.