Sentences with phrase «term synaptic»

Neonatal isolation stress alters bidirectional long - term synaptic plasticity in amygdalo - hippocampal synapses in freely behaving adult rats

Describing the association of morphological changes in dendritic spines with long - term synaptic plasticity

A Gain - of - Function Mutation in the α9 Nicotinic Acetylcholine Receptor Alters Medial Olivocochlear Efferent Short - Term Synaptic Plasticity

There is no specific time at which neurons are active, instead dynamic interactions or time - dependent changes between neurons within the network provide information about lapsed time (e.g., short - term synaptic plasticity, inhibitory feedback, etc.).

By observing these natural stimuli, professor Holtmaat's team was able to demonstrate that sensory stimulus alone can generate long - term synaptic strengthening without the neuron discharging either an induced or natural electrical pulse.

This rapid, transmitter - mediated down - regulation of a cell adhesion molecule in the sensory neurons may be one of the early molecular changes in long - term synaptic facilitation.

Modulation of an NCAM - related adhesion molecule with long - term synaptic plasticity in Aplysia

Nitric oxide and carbon monoxide produce activity - dependent long - term synaptic enhancement in hippocampus

«The appropriate balance of synapses strengthening and weakening, collectively termed synaptic plasticity, is critical for appropriate brain function.

Not exact matches

Thus, both the structural changes and the synaptic facilitation of Aplysia sensorimotor synapses accompanying long - term behavioral sensitization can be produced in vitro by applying a single facilitating transmitter repeatedly.

More recent work has revealed that the establishment of long - lasting synaptic plasticity and long - term memory also requires the removal of inhibitory constraints.

Repeated applications of serotonin, a facilitating transmitter important in behavioral dishabituation and sensitization, produced growth of the sensory neurons that paralleled the long - term enhancement of synaptic strength.

These properties, which are believed to be important for the induction of long - term changes in synaptic strength, are imparted by asparagine residues in a putative channel - forming segment of the protein, transmembrane 2 (TM2).

A potential bridge is a process called long - term potentiation (LTP), a type of synaptic strengthening that has been scrutinized in slices of rodent hippocampus and is widely considered a likely physiological basis for memory.

A form of learning in the marine mollusk Aplysia, long - term sensitization of the gill - and siphon - withdrawal reflex, results in the formation of new synaptic connections between the presynaptic siphon sensory neurons and their target cells.

In the realm of pure research, we are steadily gaining a better understanding of how galaxies form; how a single fertilized egg turns into a fruit fly or a congressman; how synaptic growth supports long - term memory.

In fact, compared with ethanol, which enhances synaptic plasticity only after long - term use, «stress can do the same job more rapidly,» Morikawa says.

He and his team have now gathered that data by exploring the impact of long - term BACE1 inhibition on dendritic spine dynamics, synaptic functions, and cognitive performance in adult mice.

Moser demonstrated a number of changes in the strength of connections between nerve cells — a phenomenon called synaptic plasticity — in the hippocampus as rats stored information in their long - term memory.

Previous studies have shown for different brain regions that the metabotropic glutamate receptor 5 (mGlu5 receptor) plays a crucial role in this long - term form of synaptic plasticity.

Long - term memories likely form when selective alterations in synaptic connections occur within patterns of neurons called neuronal ensembles.

It manifests itself in two forms: as long - term potentiation, LTP, a strengthening of synaptic efficacy, and as long - term depression, LTD, a weakening of synaptic efficacy.

The long - term facilitation of synaptic efficacy that is induced by serotonin in dissociated cell cultures of sensory and motor neurons of Aplysia is accompanied by the growth of new synaptic connections.

«Although the two types of inhibitory neurons are very similar to each other in terms of morphology, synaptic connectivity, and the kinds of small molecules they use for signaling, they're very different in terms of the methylation pattern.

Moreover, in conjunction with these results, they found decreased protein synthesis, known to be required for long - term potentiation — a form of long - lasting synaptic plasticity — and for long - term memory.

It has been known for decades that triggering new protein synthesis is vital to the formation of long - term memories as well as for long - lasting synaptic plasticity — the ability of the neurons to change the collective strength of their connections with other neurons.

Previous work had shown that spines grow when neurons undergo long - term potentiation, a persistent strengthening of synaptic connectivity that happens naturally in the brain but can also be artificially induced through stimulation.

The findings offer support for Tonegawa's new hypothesis that the strengthening of synaptic connections, while necessary for a memory to be initially encoded, is not necessary for its subsequent long - term storage.

The hippocampus is critical for episodic memory, and synaptic changes induced by long - term potentiation (LTP) are thought to underlie memory formation.

Long - term potentiation (LTP), a phenomenon in which brief repetitive activity causes a long lasting (many weeks) enhancement in the strength of synaptic transmission, is generally accepted to be a key cellular substrate for learning and memory.

Endocannabinoids work as retrograde messengers and contribute to short - term and long - term modulation of synaptic transmission via presynaptic cannabinoid receptors.

This strengthened the synaptic connections without overstimulating them — thus translating learning into long - term memories.

Egr proteins play a role in the regulation of synaptic plasticity, learning and memory [76, 77] and Egr3 in particular is very important for the processing of both short term and long term hippocampal dependent memory [78].

Plenary Address: R. Bourtchouladze Columbia University and New York State Psychiatric Institute, New York, New York, USA Genes important for long - term memory and synaptic plasticity

It has been known for many years that long - lasting forms of synaptic plasticity and long - term memory require new protein synthesis.

Long - term BACE1 inhibition in the neurodevelopmental stage caused the loss of synaptic proteins but failed to alter synaptic proteins in the already developed maintenance stage.

The freezing process is far too slow to stop such dynamic processes as action potentials and synaptic transmission; short - term memory, however, is suspected to involve chemical modification of the neurons, and long - term memory is believed to involve the growth and modification of neuronal structures, particularly synapses (15).

Studies show that nitric oxide signaling and the blood flow increases it stimulates play a central role in nerve cell maintenance, growth and repair.,, Most pertinent to anyone looking to enhance their aptitude for learning, nitric oxide - induced blood flow also makes forming new memories physically possible as it plays a key role in what neurologists call long - term potentiation, a process required for assembling and reinforcing new synaptic connections throughout the entire cerebral cortex, striatum, and hippocampus.

Most pertinent to anyone looking to enhance their aptitude for learning, nitric oxide - induced blood flow also makes forming new memories physically possible as it plays a key role in what neurologists call long - term potentiation, a process required for assembling and reinforcing new synaptic connections throughout the entire cerebral cortex, striatum

BDNF is vital for cognitive performance in the short - term and for promoting synaptic plasticity in the long - term.

Paradoxical terms such as these came up repeatedly in conversations with painter Jim Butler as he discussed his new «Synaptic Reverb» series on view at New York's Tibor de Nagy Gallery through December 22, 2017.

Considering that Samsung and Synaptics have an ongoing long - term partnership and collaboration, it was not far fetched to believe that the Galaxy S8 could sport something similar.

Synaptic does state the term «Infinity Display,» when referring to the mystery device's slim bezels, a branding term that's typically associated with Samsung.

While the «top five OEM» part is vague enough, Synaptics used the term «infinity displays» three times, including once with a specific reference to OLED panels.

It dramatically shortens attention spans, diffusing concentration and causing what Baroness Susan Greenfield, professor of synaptic pharmacology at Oxford University, termed the «infantilization of the human mind.»