Sentences with phrase «than cancer cells»

Generally, normal cells are better able to repair themselves than cancer cells.

One, known as MIRA - 1, turned out to kill more than cancer cells: It was toxic in mice.

Not exact matches

This feat is possible because cancer cells emit a different kind of metabolic waste than healthy cells.

BioNTech, which has around 700 employees at sites in Germany — more than any other unlisted biotech firm in Europe — is also working on other cancer - fighting technologies, including antibodies, cell therapies and small molecules.

They'll also jointly market Pfizer's drug Xalkori, which is approved in more than 75 countries for treating non-small cell lung cancer in patients with a certain genetic mutation.

We have no more final answers for the flowering of evil than we do for the multiplying of cancer cells.

There is evidence to suggest that cancer cells are softer than normal cells (although tumours are stiffer).

These mogrosides are 300 times sweeter than sugar and act as anti-oxidants that have shown abilities to inhibit cancer cell formation (5, 6).

The body wears out, mutations accumulate faster than can be repaired just through the natural process of cell division, cancer grows, arthritis wracks the world worn joints, the child born with tetralogy of Fallot away from surgical care dies.

But cell phone use has now been linked to cancer, and children are even more susceptible than adults.

Researchers at Tufts Medical School noticed that cancer cells being grown in the lab multiplied more quickly in polyester test tubes than in glass.

A 2013 study by Cheryl Watson at The University of Texas Medical Branch at Galveston found that even picomolar concentrations (less than one part per trillion) of BPS can disrupt a cell's normal functioning, which could potentially lead to metabolic disorders such as diabetes and obesity, asthma, birth defects or even cancer.

Preclinical trials indicate potential for noscapine as a cancer drug with less toxicity to healthy cells than currently available chemotherapies.

Residual tumors, spawned from the remaining cancer cells, were 3.5 times smaller in the treated mice than in untreated mice.

And using cells from someone other than the cancer patient being treated might trigger an immune response against the foreign cells.

PARP inhibitors prevent DNA repair causing cancer cells to die rather than repair.

«These chemicals may lead to new cancer treatments if we can find ones that are more potent to cancer cells than normal healthy cells.

Because they multiply quickly and spread throughout the body, cancer cells require more energy than normal cells.

«The fine particles of this drug allow for it to be released slowly and stay in the abdomen,» said Katherine Roby, Ph.D., research associate professor in the Department of Anatomy and Cell Biology at KU Medical Cancer, who started her pre-clinical work on Nanotax more than a decade ago.

The overabundance of receptors causes the cancer cells to divide rapidly and the tumor grows faster than average.

RARE but pernicious cancer «stem» cells, blamed for the spread and invincibility of some tumours, may be more vulnerable than we thought.

Protein expression in these glioblastoma cells more closely mimicked that in real cancer cells than in 2D cultures of cells, indicating that this method could be used to study cancer (Nature Nanotechnology, DOI: 10.1038 / nnano.2010.23).

The hope is that 3BP specifically kills certain cancer cells — while leaving normal cells alone — because they rely more on glucose metabolism than on an alternative pathway called oxidative phosphorylation.

In a head - to - head clinical trial comparing standard chemotherapy with the immunotherapy drug nivolumab, researchers found that people with squamous - non-small cell lung cancer who received nivolumab lived, on average, 3.2 months longer than those receiving chemotherapy.

The researchers studied triple - negative breast cancer cells, which grow and spread faster than most other types of cancer cells.

The study also suggests that targeting the machinery that makes cells mobile, rather than targeting the tissue - clearing process — which has been tested in patients but has not been very effective — may be a better treatment strategy to stop cancers from spreading.

We know that they are under stress when they are fighting cancer or other diseases, so I wondered whether anything measureable could be seen if we put them under further stress with UVA light.We found that people with cancer have DNA which is more easily damaged by ultraviolet light than other people, so the test shows the sensitivity to damage of all the DNA — the genome — in a cell.»

But T - cells appear to be even more promising, because they can react to abnormalities inside cancer cells rather than just on their surfaces.

The team led by Andreas Plückthun, Director of the Department of Biochemistry at the University of Zurich, involving postdoc Rastik Tamaskovic and PhD student Martin Schwill, has now found out why these antibodies merely slow tumor growth rather than killing off the cancer cells.

This is how treatments based on a type of white blood cell called T - cells are curing some cancers, rather than just slowing their advance (see «Cancer meets its nemesis in reprogrammed blood cells «-RRB-.

«This is important because some cancer cells are more resistant to one type of treatment than another.

Their analysis of more than 4,000 individual tumor cells, the largest effort to date in brain tumors, finds three developmental categories of cancer cells — one resembling neural stem cells and two characterized by sets of genes indicting paths towards differentiation.

With that knowledge, they screened more than four dozen monoclonal antibodies — unique agents that can stop cells from growing or forming tumors and can be mass produced — before finding two that block tumor creation in both types of cancer.

Vitamin C is up to ten times more effective at stopping cancer cell growth than pharmaceuticals such as 2 - DG, according to scientists in Salford, UK.

Cancer stem cells will need to be identified and treated in a different manner than the bulk tumor.»

Non-melanoma skin cancers — specifically, basal cell carcinoma and squamous cell carcinoma — are more likely than melanoma to involve itch or pain, the study found.

When injected with cancer cells, animals housed there developed tumors 80 % smaller than those in control mice, or no tumors at all.

Patients were considered free of MRD if less than one cancer cell could be detected in 1,000 normal bone marrow cells.

«We discovered that the aggressive cancer cells that are spreading in colon, breast, and skin cancer contained a much higher portion of the protein PITPNC1, than the non-aggressive cancer cells,» says researcher Nils Halberg of the CELLNET Group at the Department of Biomedicine at UiB.

More than 80 % of all skin cancers are BCC, arising from the basal cells (i.e., small, round cells found in the lower layer of the epidermis).

In another clue that cancer cells persist because they maintain their telomeres, those cells that started out with longer telomeres in the experiment lived longer than those with shorter telomeres.

But a study of mice shows that breast cancer cells decamp in groups, and the clumps of cells have a better chance of establishing a colony than loners do, Kevin Cheung of Johns Hopkins University reported December 7 at the annual meeting of the American Society for Cell Biology.

«This means we can predict which of the cancer cells are getting aggressive and spread, at a much earlier stage than today.»

«We have to hit cancer cells from more than one angle, and that's made it important to learn how to combine drugs that hit the right combination of pathways,» says Anne Le, M.D., H.D.R., assistant professor of pathology at the Johns Hopkins University School of Medicine and member of the Johns Hopkins Kimmel Cancer Ccancer cells from more than one angle, and that's made it important to learn how to combine drugs that hit the right combination of pathways,» says Anne Le, M.D., H.D.R., assistant professor of pathology at the Johns Hopkins University School of Medicine and member of the Johns Hopkins Kimmel Cancer CCancer Center.

The study published in Cancer Cell shows that exosomes from tumor cells of breast cancer (and other tumor types such as ovarian and endometrial) are different in size and composition than those of healthy Cancer Cell shows that exosomes from tumor cells of breast cancer (and other tumor types such as ovarian and endometrial) are different in size and composition than those of healthy cancer (and other tumor types such as ovarian and endometrial) are different in size and composition than those of healthy cells.

Fat cells cultured from the body mass index of a morbidly obese patient cause multiple myeloma cells to anchor to a much greater extent than normal cells and produce a significantly larger number of blood vessels to sustain the cancer cells.

The nanoparticle, according to the scientists, helps the drug slip through capillaries near cancer cells and remain within the tumor longer than it would otherwise.

Many cancers get energy from glycolysis, which occurs in the liquid inside cells, rather than via aerobic respiration from mitochondria.

More than 2 million units of platelets — the blood cells responsible for clotting — are transfused each year, with cancer patients receiving an important share of them.

And because mouse embryo cells with inactivated copies of BRCA2 are more sensitive to ionizing radiation than normal cells are, «it's a reasonable extrapolation» that breast cancers with mutated copies of the gene may be especially good candidates for radiation therapy.