The researchers noticed that control mice treated with N - methyl -2-pyrrolidone (NMP) survived
longer than control mice treated with other drug delivery vehicles.
Instead mice injected with stem cells developed a far greater number of synapses, or connections between neurons, at the damaged site
than control mice did.
When scrapie prion protein was later injected into their abdomen, mice treated with venom came down with scrapie an average of 24 days
later than control mice did.
Nondiabetic mice that received the betatrophin gene injections were able to control their blood sugar even more
effectively than control mice — performing better on the glucose tolerance test that is used to diagnose diabetes.
The increased rBAT was sufficient to maintain normal body temperature and also protect against diet - induced obesity: When the control and mutant mice were fed a high - fat diet, the mutant mice did not gain more
weight than the control mice.
Further research showed that the mice produced lower levels of immune signaling molecules called cytokines after
treatment than control mice.
The rats who ate non palatable chow and were on normal «diets» were perfectly fine; the rats who had eaten sweets but were on a normal diet ate about 20 % more; the rats who had been in restricted cycling patterns and refed on sweets ate 80 % more
than control mice on normal diets.
The vaccine slowed the progression of prion disease in the remaining 70 % of the experimental mice, allowing them to live
longer than control mice, which did not receive the vaccine and died within about 200 days following infection.
Mice that received extra copies of a protein during fetal development produced more of a key hearing cell (bottom)
than control mice did.
All the mice died within 96 hours, but those with human amyloid lost less weight, had fewer bacteria in their brains, and lived up to roughly 30 hours longer
than the control mice, the team reports today in Science Translational Medicine.
lost less weight, had fewer bacteria in their brains, and lived up to roughly 30 hours longer than the control mice
The mice lived longer
than control mice; 30 percent lived longer than 80 days compared to mice treated with only imatinib, all of which died within 60 days.
The mice whose mTOR was suppressed lived 20 percent longer
than the control mice — which in human terms, equates to an additional 15 years of life!
These trials caused no harm, discomfort or suffering to the mice — in fact, the mice receiving the antioxidants lived longer, healthier lives
than the control mice.