When the researchers injected myelin - deficient regions in the mice with a drug that prevents destruction of the AXIN2 protein, the mice grew myelin sheaths
faster than untreated mice, repairing the damage.
The experimental drug activated maternal PWS genes, and the treated mice had better growth and weight
gain than untreated mice, with 15 percent surviving to adulthood without serious side effects.
Examination showed that the TSA - treated mice also had larger neurons in the spinal cord, thicker muscle fibers, and more muscle
mass than untreated mice.
Mice who received the modified buckyballs had their symptoms delayed by 10 days and survived 8 to 10 days
longer than untreated mice.
Alzheimer's mice that received AF267B, they found, performed significantly better on this test
than untreated mice did.
That research showed that mice on a normal diet who were exposed to low doses of antibiotics throughout life, similar to what occurs in commercial livestock, packed on 10 to 15 percent more
fat than untreated mice and had a markedly altered metabolism in their liver.
The treated mice lived an average of 36 days longer
than untreated mice, and they were better at building nests, which reflects a combination of social behavior, cognitive performance and motor capabilities.
Furthermore, EV71 - infected mice given Roc - A lived longer
than untreated mice and had lower levels of the virus in their spinal cord and brain.
Goldman treated HD mice with virus - delivered instructions to make new neurons live longer
than untreated mice (Goldman's work will be published soon in the journal «Cell Stem Cell» and we'll definitely be writing an HDBuzz article on it!).