Sentences with phrase «tuberculosis pathogens»

EZ120 works in very small doses, is outstanding at entering tuberculosis pathogens, and has low toxicity toward human cells.

The evidence for Adrian Williams and Robin Dunbar's hypothesis that the tuberculosis pathogen could provide essential nicotinamide to humans is...

Adrian Williams and Robin Dunbar make a persuasive case for the probiotic effect of the tuberculosis pathogen in our evolutionary...

The mycomembrane of the tuberculosis pathogen Mycobacterium tuberculosis consists of a lipid double layer that encapsulates the cell wall, forming an exterior barrier.

«Going undercover to fight tuberculosis: Antibacterial beta - lactone infiltrates mycomembrane biosynthesis and kills tuberculosis pathogen.»

When the mammal died, she determined the animal was infected with a novel tuberculosis pathogen, Mycobacterium mungi, closely related to the TB pathogen infecting humans in West Africa.

Not exact matches

There, I got to study tuberculosis not only on the level of macroscopic bone changes as before, but also look at the molecular level and search for the pathogen's DNA.

And the skeptics point out that vaccine developers have had little success against pathogens like HIV that routinely outwit the immune system — the malaria parasite, hepatitis C virus, and the tuberculosis bacillus are prime examples.

Studies in mice and in tissue cultures suggest that giving vitamin C with tuberculosis drugs could reduce the unusually long time it takes these drugs to eradicate this pathogen.

However, because Mycobacterium tuberculosis has developed many ways to evade the normal immune response, infections become chronic because a stalemate develops between the pathogen and the host.

Now, in a novel twist, researchers have found a way to recruit help from none other than Mycobacterium tuberculosis itself to make the deadly pathogen susceptible to an existing TB drug that it has learned to dodge.

Now, in a novel twist, researchers have found a way to recruit help from none other than Mycobacterium tuberculosis itself to make the deadly pathogen susceptible to an existing tuberculosis (TB) drug that it has learned to dodge.

The compounds have been shown to be effective in killing many species of bacterial pathogens but are generally less effective against the bacterium that causes tuberculosis.

A level - three lab is designed to contain potentially lethal airborne pathogens for which a cure is available, such as tuberculosis.

The approach used in this research «holds great potential to generate more rifamycin analogs to combat the threat of MDR strains of M. tuberculosis, and / or other life - threatening pathogens,» the researchers wrote in their conclusion.

Tuberculosis bacteria belong to the mycobacterial pathogens.

In addition, because the investigators have data indicating that other important airway pathogens that cause acute bacterial pneumonia also may use M - cells to cause severe disease, they are extending their work beyond tuberculosis.

Shortly after the introduction of the BCG vaccine it was noticed that not only tuberculosis occurred less frequently but that young children also died less due to other pathogens.

And unlike for most other drug - resistant pathogens, we have evidence that, with a comprehensive response, drug - resistant tuberculosis epidemics can be rapidly reversed.

The difference between these two outcomes lies less with the pathogen and more with us as a global tuberculosis control community and whether we have the political will to prioritise a specific response to the disease.

This could lead to the development of active tuberculosis and perhaps drug resistant forms of the pathogen in some patients.

Washington, DC — January 3, 2018 — Studies in mice and in tissue cultures suggest that giving vitamin C with tuberculosis drugs could reduce the unusually long time it takes these drugs to eradicate this pathogen.

Researchers increasingly recognize that latent TB infection includes diverse responses to infection with Mycobacterium tuberculosis (the pathogen that causes TB) and thus variable outcomes.

In addition, tuberculosis provides one example in which pathogen and host genomics can be used in combination to identify those at increased risk and to implement measures to control the spread of disease.

Proteins targeted for structure determination by SSGCID are selected for their biomedical relevance in human pathogens such as Ebola and Zika, as well as those responsible for tuberculosis, leprosy, malaria, and influenza.

For example, NIAID - supported researchers have completed hundreds of genomic sequences of disease - causing organisms, including pathogens responsible for malaria, tuberculosis, chlamydia, and seasonal and pandemic influenza.

Sarah's recent work links Genome - Wide Association Studies (GWAS) with genomic studies of the pathogen, and investigates the interaction of both host and pathogen genomes in Vietnamese tuberculosis and enteric fever patients.

Mycobacterium tuberculosis remains the leading bacterial cause of death globally because, like other successful pathogens (e.g. HIV), it goes beyond evasion to take over functions of immune cells.

Sarah is using genomics to understand host - pathogen interactions of a number of infectious diseases including tuberculosis, malaria and enteric fever.

In this area, Sette's disease focus has shifted over the years from HIV, HBV and HCV to emerging diseases and diseases of potential biodefense concern to, most recently, diseases and pathogens relevant to worldwide global health, including dengue viruses, malaria, tuberculosis, and trypanosome infections.

She is completing her postdoctoral training at IDRI and is excited to use her background in adaptive immunity to help optimize vaccine strategies for pathogens like Mycobacterium tuberculosis.

Along with the Centers for Disease Control and Prevention and other organizations around the world, the ASBMB is observing Tuberculosis Day 2018 by sharing research news from scientists expanding our understanding of Mycobacterium tuberculosis and a variety of perspectives on how the pathogen continues to affecTuberculosis Day 2018 by sharing research news from scientists expanding our understanding of Mycobacterium tuberculosis and a variety of perspectives on how the pathogen continues to affectuberculosis and a variety of perspectives on how the pathogen continues to affect our world.

Research interests include Mycobacterium tuberculosis pathogenesis, host - pathogen interactions, and chemical biology.

The complete genome sequence of the best - characterized strain of Mycobacterium tuberculosis, H37Rv, has been determined and analysed in order to improve our understanding of the biology of this slow - growing pathogen and to help the conception of new prophylactic and therapeutic interventions.

These natural antimicrobial peptides (AMPs) are active against small and large bugs alike, even pathogens such as tuberculosis, Mycobacterium tuberculosis, and the dastardly Staphylococcus aureus.

For instance, recent evidence suggests that cultural values of collectivism also serve an «anti-pathogen defence» whereby behavioural manifestations of collectivism, such as conformity and parochialism, function as buffers against the transmission and increased prevalence of disease - causing pathogens (e.g. malaria, typhus and tuberculosis)(Fincher et al. 2008).