Sentences with phrase «tumor growth after»

Cats and other species have been affected by chronic inflammation and tumor growth after receiving some vaccines.

«We wanted to see whether these newly generated neurons could result in tumor growth after transplanting them into mouse brains,» said Karen Ring, UCSF Biomedical Sciences graduate student and the paper's lead author.

Not exact matches

According to Irving Weissman of the Stanford University School of Medicine in Palo Alto, California: «We showed that even after the tumor has taken hold, the antibody can either cure the tumor or slow its growth and prevent metastasis.»

Model of tumor growth in a rat brain before radiation treatment (left) and after one session of radiotherapy (right).

After EZH2 enzymes rise, their levels taper off, and then, the scientists found two to three-fold increases in a protein called DNMT1, which maintains DNA methylation in the «start» location of a variety of tumor suppressor genes that normally suppress cell growth.

A raft of studies in laboratory animals, molecular models and cancer patients suggest that pain drugs given during and after cancer surgery stimulate the growth and spread of certain tumors.

The researchers confirmed these findings in a mammary carcinoma mouse model — treatment with dasatinib just a few days after administering two high doses of chemotherapy prevented tumor growth and increased survival rates.

With these in vitro test methods, the KU researchers have shown that anti-CD44s antibody can reduce pancreatic cancer cell growth, metastasis and ability of the tumors to recur after radiation therapy.

The study, which followed four individuals for a year after they were treated with embryonic stem cell - derived retinal pigment epithelial cells for macular degeneration, observed no serious side effects (tumor growth or other unexpected effects) related to the therapy.

On the safety side, the panelists delved into the possible risks of injecting genetically modified cells into patients, including the potential for deadly viral infections, brain toxicity, and, paradoxically, the growth of new tumors brought about by CAR - T cells years after treatment.

Researchers were able to complete the first genome - wide screen of a mouse by injecting small hairpin RNAs into the embryos of 100 pregnant mice and analyzing the animals after birth to look for genes that contributed to tumor growth.

The researchers from the study group were able to show that upregulation of the growth factor VEGF - B, which is related to VEGF - A, in the primary tumor in the bowel prior to treatment was associated with a poorer prognosis, whereas upregulation of VEGF - B in the liver metastases after treatment was associated with a better prognosis.

Four months after receiving the treatment, the patients have not developed any tumors or abnormal growths that have sometimes been seen in animals receiving hESC - derived cells, the team reports online today in The Lancet.

After 5 weeks, metastatic tumor growth in the brain was prominent in all mice that received CSCs carrying vector only.

After four cycles of therapy (42 days), all mice receiving the combined regimen were alive, whereas mice in the other groups had to be euthanized due to tumor growth.

Type I tumors are often treated with radiation therapy, which helps stop or slow cancer growth, given in addition to or after the primary treatment.

After enrolling in a clinical trial for genetically based drug therapy, Victor's tumor has shrunk and shows no signs of growth.

More than 90 percent of patients with either high nm23, a protein that prevents the cancer from spreading, or low angiogenesis, minimal growth of new blood vessels to supply the budding tumor, were alive an average of 14 years after treatment, compared to only 70 percent of those with low nm23 or high vessel density.

The recently discovered protein NUDT5 is now presented as a candidate target for development of breast cancer treatment after being demonstrated to stop breast tumor cell growth in laboratory experiments.

Slow down lymphoma growth modestly and show no effect on tumor regression; delay tumor recurrence with activation of p53; enhance tumor regression and inhibit tumor recurrence after alkylating drug therapy.

After 30 days, there was a reduced tumor growth markers from 30 percent to 71 percent.

After discussing similar effects in other animal models of cancer and a few studies showing the ability of antioxidants to prevent cancer growth, Campbell concluded that «a pattern was beginning to emerge: nutrients from animal foods increased tumor development while nutrients from plant - based foods decreased tumor development.»

Extrapolating the tumor growth curves, it looks like the mice in experiment (B) would be sacrificed 5 weeks after implantation on the high protein diet, or 8 weeks after implantation on the low protein diet; in experiment (G), mice on the high protein diet would be sacrificed about 9 weeks after implantation, while mice on low protein diets would have been sacrificed about 11 weeks after implantation.

In study after study, the rats eating only 5 percent of their total calories as casein remained tumor - free, while the rats eating 20 percent of their calories as casein developed abnormal growths that marked the beginning of liver cancer.

However, a vaccine has been developed that may be able to control the growth of a tumor after surgery.