Some amount of inflammation is generally beneficial, as it ramps
up production of cytokines, proteins that help us heal and protect us from the effects of overexertion.
Not exact matches
The third approach turns
up production of T regulatory cells, which prevent specific immune cells from copying themselves and can also suppress rejection by secreting biochemicals called
cytokines that direct the immune orchestra to change its tune.
Recently, some other
cytokines (such as tumor necrosis factor) or chemical drugs were also found to
up - regulate several IFN - inducible gene family members (including RIG - G) through
production of type I IFNs (37, 38), further indicating that the induction
of RIG - G by ATRA in NB4 cells was tightly related with an IFNα autocrine pathway.
• Effects
of soy isoflavones on the make -
up of the gut microflora • Bacterial transformation
of genetically modified soy DNA into gut bacteria capable
of expressing toxic proteins that damage the intestinal lining • Effects
of chemical contamination from glyphosate residues on neurological and gut function • Soy - induced activation
of the immune system and altered
production of inflammatory
cytokines associated with febrile seizures
However, consistent with the differences between visits in wound healing,
production of IL - 6, IL - 1β, and TNF - α increased more steeply between 4 and 22 hours following the social support interaction than after the conflict interaction, ending
up higher at 22 hours at the first visit for all 3
cytokines (IL - 6, F2, 81 = 3.55; P =.03; IL - 1β, F2, 81 = 9.12; P <.001; TNF - α, F2, 81 = 3.56; P =.03).