We also don't want to down regulate an immune response at times with the body is
using reactive oxygen species to say fight a pathogen.
The cell envelops the foreign body and exterminates it specifically by
using reactive oxygen species (ozone, hydrogen peroxide, bleach), generated thanks to the enzyme NOX2.
Credit: ZEISS Microscopy Transient, specific bursts of reactive oxygen species can extend lifespan in C. elegans To many people it may sound counter-intuitive to
use reactive oxygen species (ROS) as a way to increase lifespan.
Not exact matches
When potent
oxygen radical scavengers such as cerium oxide nanoparticles (nanoceria) were combined with a highly charged polymer (polyacrylic acid) and incorporated into extracted chloroplasts
using the LEEP process, damage to the chloroplasts from superoxides and other
reactive oxygen species was dramatically reduced.
The team — which also included scientists from France (CNRS Marseille), Denmark (University of Copenhagen) and the UK (University of Cambridge)-- undertook a detailed investigation of a new class of LPMO enzymes
use oxygen from the air to initiate a highly
reactive oxidation process that allows a resistant form of starch to be broken down.
In medicine, they could also be
used to encapsulate
oxygen - sensitive active compounds and to reduce the inflammation caused by
reactive oxygen species (ROS) in biomedical applications.
At the Vienna University of Technology, it has now been possible to selectively switch individual
oxygen molecules sitting on a titanium oxide surface between a non-
reactive to a
reactive state
using a special force microscope.
Mitochondria help injured muscle cells (myofibers) repair by soaking up calcium that enters from the site of injury and
using it to trigger increased production of
reactive oxygen species.
Sunlight and toxins do much of the damage, but the biggest culprit may be highly
reactive byproducts created as cells
use oxygen to turn sugar into energy.
But organisms from humans to algae also have another clock that doesn't rely on rhythmic gene expression to keep time, but instead
uses the rise and fall of the
reactive oxygen molecules that are formed as natural byproducts of metabolism.
Technology such as this, scientists said, may have a promising future in the identification and surgical removal of malignant tumors, as well as
using near - infrared light therapies that can kill remaining cancer cells, both by mild heating of them and generating
reactive oxygen species that can also kill them.
Vitamin C can also help provide the antioxidants your immune system needs to
use ROS (
reactive oxygen species) to attack pathogens without destroying its own cells, so take a gram or so of vitamin C a few times a day if you think you are getting sick.
Free radicals are often
used in the same sentence with cancer, oxidative stress is a favorite buzzword for many nutritional supplement companies, and
reactive oxygen species are rarely mentioned because they're hard to pronounce.
P450
uses oxygen and NADH (the active form of the vitamin niacin) to add a
reactive «hydroxyl» group to the toxin to be excreted.
The immune defense against these infections is glucose - dependent (as it relies on production of
reactive oxygen species
using glucose) and thyroid hormone - dependent (as thyroid hormone drives not only glucose availability, but also the availability of iodine for the myeloperoxidase pathway).
Radiation was
used to generate
reactive oxygen species (free radicals) and tribulus terrestris was able to scavenge them.
Iodine along with the enzyme myeloperoxidase is needed to produce respiratory bursts — the burst of
reactive oxygen species (ROS) that white blood cells
use to kill pathogens.
The
oxygen - containing molecules the body
uses to produce energy can be highly
reactive and can inadvertently cause damage to the mitochondria and even the cells themselves.
Moreover,
reactive nitrogen and
oxygen species (RNOS) are
used extensively in cellular signalling, and cells adaptively regulate endogenous antioxidants on short time scales to respond to deletorious spikes of RNOS faster than we could ever achieve with dietary antioxidants.
For example, KBs were recently reported to act as neuroprotective agents by raising ATP levels and reducing the production of
reactive oxygen species in neurological tissues, 80 together with increased mitochondrial biogenesis, which may help to enhance the regulation of synaptic function.80 Moreover, the increased synthesis of polyunsaturated fatty acids stimulated by a KD may have a role in the regulation of neuronal membrane excitability: it has been demonstrated, for example, that polyunsaturated fatty acids modulate the excitability of neurons by blocking voltage-gated sodium channels.81 Another possibility is that by reducing glucose metabolism, ketogenic diets may activate anticonvulsant mechanisms, as has been reported in a rat model.82 In addition, caloric restriction per se has been suggested to exert neuroprotective effects, including improved mitochondrial function, decreased oxidative stress and apoptosis, and inhibition of proinflammatory mediators, such as the cytokines tumour necrosis factor - α and interleukins.83 Although promising data have been collected (see below), at the present time the real clinical benefits of ketogenic diets in most neurological diseases remain largely speculative and uncertain, with the significant exception of its
use in the treatment of convulsion diseases.
In earlier versions, the nickel - molybdenum - zinc alloy catalyst
used to produce the hydrogen also created
reactive oxygen species, which would attack and destroy the bacteria's DNA.
However, it is too chemically
reactive to remain a free element in Earth's atmosphere without being continuously replenished by the photosynthetic action of living organisms, which
use the energy of sunlight to produce elemental
oxygen from water.