Sentences with phrase «way is a tumor»

One protein that keeps healthy cells from behaving this way is a tumor suppressor named p53.

Not exact matches

«In the long term, we want to be able to send energy to and communicate with implants all over the body, to record data from a variety of organs in many different ways, maybe even report on the conditions of tumors or cancer therapies,» Maharbiz says.

Right now, that data is locked up in electronic health records, which tend to be organized to help bill insurers, rather than detail, say, the rate at which the tumor is shrinking, in a way that could be pulled out for a study.

Do you think that the best way to treat a tumor is blood letting and leaches, it was in the 18th century?

Glenn Beck is one sick man... the things he says are indicative of someone with a brain tumor... the way he twists things to fit his teachings... or rather... preachings... is very scary.

Also, eating in a comparable manner to our precursors has been demonstrated on numerous occasions to offer stunning medical advantages, including counteractive action of most maladies of human progress, for example, tumor, coronary illness, Alzheimers, and other interminable conditions that are generally created by horrible eating routine and way of life.

With his kind of cancer, there were no tumors to X-ray, no reliable way to chart the course of the disease.

What's the best way to give it to minimize the number of tumor cells for the longest amount of time?»»

A research team at the University of California, Riverside has discovered a way for chemotherapy drug paclitaxel to target migrating, or circulating, cancer cells, which are responsible for the development of tumor metastases.

In this special section of Science, expert contributors retrace the long and tortuous path leading to the mapping and identification of the BRCA1 gene; discuss the ways in which BRCA mutation status has been integrated into the clinical management of patients in high - risk families; and highlight the role of the BRCA proteins in preserving the structural and numerical integrity of chromosomes throughout the cell cycle, a function that may explain their tumor suppressor activity.

For some years now, a new class of drugs called antibody - drug conjugates (ADCs) have been used, which work in two ways: they consist of an antibody that binds selectively to the tumor cell receptor and interrupts the signal to propagate; they also act as a transport vehicle for a chemical substance that enters the cancer cells with the antibody and triggers their death.

The goal is to create ways of detecting that stiffness while a tumor is still too small to be felt by human hands.

Lo's team set out to find ways to further weaken the tumors, since the drug addiction response (which can range from a mere slow down of the cancer's growth rate to cancer cell death), can be used to improve clinical outcomes.

Conventional radiation with photons gave way to intensity - modulated radiation therapy, or IMRT, in which more precise beams of photons could be moved dozens or hundreds of times with varying intensities, attacking tumors in three dimensions with safer high doses.

The study's 31 samples of dog tumors was compared to 40 normal canine tissues samples as a way of estimating the variance in gene expression.

«The traditional way to deliver drugs to tumors is to put the drug inside some type of nanoparticle and inject those particles into the bloodstream,» said Jian Yang, professor of biomedical engineering, Penn State.

«We are looking for novel ways of preventing cancer cells of the primary tumor from spreading to other parts of the body.

Some researchers are working to discover new, safer ways to deliver cancer - fighting drugs to tumors without damaging healthy cells.

«What we may be looking at,» he adds, «is a future way to prevent metastasis to many organs simultaneously» using drugs that make tumor cells let go of the blood vessels they cling to.

«The molecular biology of the tumor is really leading the way over the tissue of origin.»

«If we can identify the point at which tumor cells acquire the characteristics of stem cells, it will be possible to look for ways to interrupt the process and avoid progression of the disease.»

There are two ways it's been dealt with: one is looking at the tumor again at a different time point.

Cells suspended in a stiff matrix were more likely to work their way through the matrix to other side of a serum gradient, analogous to how metastasizing cancer cells break free from their tumors.

«Studies like the current one involving rhabdomyosarcoma are giving us a close - up look at the way cancer evolves in response to treatment,» said study co-author Richard K. Wilson, Ph.D., director of The Genome Institute at Washington University School of Medicine in St. Louis, where scientists have extensive expertise analyzing tumor recurrence using whole - genome sequencing.

Along with finding that the tumor suppressor protein SIRT6 is inactive in around 30 percent of cases of pancreatic ductal adenocarcinoma (PDAC), the team identified the precise pathway by which SIRT6 suppresses PDAC development, a mechanism different from the way it suppresses colorectal cancer.

Then again, the Web is also making us sicker, or at least making us feel that way: A 2008 study by Microsoft Research showed that Internet searchers tend to focus on only the top few results, typically highlighting rarer, more serious diagnoses of common ailments such as headache (brain tumor!)

Dr. Folkman's War: Angiogenesis and the Struggle to Defeat Cancer, Robert Cooke (Random House) Judah Folkman is convinced the best way to kill tumors is to cut off their blood supply.

Another version, the CTC - iChip, rapidly isolates CTCs in a way that does not rely on preidentified tumor antigens, allowing capture of cells with gene expression patterns that may be missed by the antibodies used in the HBCTC - Chip.

«It's being given to patients as a way of stopping the growth of tumors.

However, tumor cells are smart and have developed ways to avoid immune detection.

Scientists knew Apc was involved in stifling tumor formation because most colon cancers find a way to turn the gene off.

However, because of the aggressive way glioblastomas invade surrounding brain tissue, it is impossible to remove all parts of the tumors, and the cancer eventually returns and spreads.

There is no way to directly target the loss of the tumor suppressor, but Ler et al. found another strategy to effectively treat tumors with this mutation.

«We had a hypothesis about how these treatments would work together, and when we did biopsies of patients» tumors we found that they were cooperating in just the way we thought they would,» says lead author Antoni Ribas, director of the Immunology Program at the UCLA Jonsson Comprehensive Cancer Center.

So making cells in the primary tumor softer might be a way to prevent metastasis,» he said.

In human cells as well, if Nbs1 and ATM function in the same way to ensure repair of DNA damage, tumor formation may be prevented.

They are designed to get around one of the ways that cancer protects itself from the immune system: tumors can activate the body's natural protective response from autoimmunity, called a checkpoint, and thereby thwart cytotoxic T cells.

When the protein is present, these cells that start out round and stuck together in a pattern resembling cobblestones become irregularly shaped and tend to detach from the tumor site in an uncoordinated way — hallmarks of metastasis.

Von Maltzahn and Bhatia are developing ways to use nanobots nearly 500 times smaller than Montréal Polytechnic's microbots that can find their way to cancerous tumors without needing to be guided from outside the body.

«The macrophage is migrating into a site and doing what we want it to do rather than driving tumor development in a normal way,» Lewis says.

«The best way to apply immunotherapy as cancer prevention is during tumor dormancy to prevent advanced stage disease.»

Using a fluorescent protein to detect Rgs16 expression, the investigators found that this gene is induced by pancreatic tumor formation starting from its earliest manifestation as ductal neoplasm all the way to advanced solid tumor in a spatially and temporally coincidental manner.

Moffitt Cancer Center, a leader in molecular cancer research, and a research team led by Jia Fang, Ph.D., assistant member of the Tumor Biology Department, has discovered a new way to control the activity of SETDB1, a protein that is often upregulated in cancer.

In this way, metastases may be formed, even after the main tumor was treated successfully.

A team led by neuroscientist Khalid Shah, MS, PhD, who recently demonstrated the value of stem cells loaded with cancer - killing herpes viruses, now has a way to genetically engineer stem cells so that they can produce and secrete tumor - killing toxins.

Now, Ludwig researchers have shown that one way to override the growth - promoting effects of PTEN deletion is, surprisingly, to inhibit a separate tumor suppressor gene.

Given that breast cancer cells traveling through the bloodstream on their way to secondary sites where breast tumors metastasize most often — lung, bone marrow, brain and liver — must first pass through the basement membrane microvasculature, Ghajar and Bissell suspected that the basement membrane could be a major component of the dormant niche in distant organs.

«This is one way to try to train the immune system to attack metastatic tumors that may not be recognized yet.»

The treatment manipulates our natural defenses to fight off the tumor in the same way it has been trained to attack other foreign invaders in our body.»

There's a need for ways to find these cells and to study them, and importantly, to develop drugs that target them, because these cancer stem cells are resistant to chemotherapy drugs that target the main tumor.