The high - fat aspect of these diets can make them more likely to cause
weight gain and obesity in pets and can cause pancreatitis in at - risk patients.
«We've known for some time that sleep deprivation is associated with
weight gain and obesity in the general population, but this study shows that getting enough sleep — even just two hours more — may be as important as a healthy diet and exercise for new mothers to return to their prepregnancy weight,» says Erica Gunderson, an investigator at Kaiser Permanente Divison of Research in Oakland, Calif., and lead author of the study published in the American Journal of Epidemiology.
Not exact matches
We chose to model the effects on body
weight because good evidence (from both trials
and epidemiological studies) links regular consumption of sugar sweetened drinks to
weight gain.8 10 12 Moreover, data from longitudinal studies support the idea that changes
in the price of sugar sweetened drinks are linked to changes
in body
weight.20 Other groups have used this form of modelling to estimate the effects of a sugar sweetened drink tax on
obesity.18 21 22
In this systematic review, the researchers investigate whether the disclosure of potential financial conflicts of interest (for example, research funding by a beverage company) has influenced the results of systematic reviews undertaken to examine the association between the consumption of highly lucrative sugar - sweetened beverages (SSBs)
and weight gain or
obesity.
Introducing solid food before your baby reaches four months raises her risk of increased
weight gain and obesity, both
in infancy
and early childhood.
The AAP states that «excessive fruit juice can lead to excessive
weight gain,»
and that consumption can
and does play a role
in obesity.
Poor sleep quality
and short sleep durations can lead to
weight gain and obesity, especially
in adolescents
and young adults.
While the benefits of breastfeeding are well known — breastfed babies tend to be better protected against rapid
weight gain and obesity, among other things — the reality is that 60 percent of American infants are exposed to infant formula by four months of age either as a sole source of nutrition or
in combination with breastmilk.
Rapid
weight gain in an infant's first six months of life is a risk factor for child -
and adulthood
obesity, according to researchers.
Protein
and total energy intake, as well as the amount of energy metabolised, are higher among formula - fed infants relative to breastfed (14,15), leading to increased body
weight during the neonatal period (10)
and data suggests that both higher protein intake (16)
and weight gain (17) early
in life is positively associated with the development of
obesity later
in childhood.
While the benefits of breastfeeding for a newborn alone are enough to persuade women to make that choice when feasible, knowing that it can prevent both childhood
obesity and later
weight gain in the moms may help persuade those who are on the fence.
By moderating the impact of physiological processes that promote
weight gain during infancy, breastfeeding might assist
in «programming» an individual to be at reduced risk for overweight
and obesity later
in life (20).
Studies to date have examined familial clustering of risk behaviors linked with accelerated
weight gain in children, psychosocial consequences of
obesity in children, parenting strategies that promote active lifestyles
in children,
and developmental
and contextual factors that explain declines
in adolescent girls» physical activity.
Breastfed babies can
gain «lots
and lots» of
weight in the first year, explains Gourley, but they'll still have a lower risk of
obesity later
in life, because of the powerful protective factors of breast milk.
The International Sweeteners Association (ISA) says it strongly refutes the claims made
in the study: «There is a broad body of scientific evidence which clearly demonstrates that low - calorie sweeteners are not associated with an increased risk of
obesity and diabetes as they do not have an effect on appetite, blood glucose levels or
weight gain.
In a new study published in the Journal of Health Psychology, researchers from the University of Surrey have found dieters who eat «on the go» may increase their food intake later in the day which could lead to weight gain and obesit
In a new study published
in the Journal of Health Psychology, researchers from the University of Surrey have found dieters who eat «on the go» may increase their food intake later in the day which could lead to weight gain and obesit
in the Journal of Health Psychology, researchers from the University of Surrey have found dieters who eat «on the go» may increase their food intake later
in the day which could lead to weight gain and obesit
in the day which could lead to
weight gain and obesity.
In fact, nutritionists recommend upping food intake by only about 300 calories a day during the last two trimesters, and excessive weight gain by women during pregnancy is linked to obesity in their kid
In fact, nutritionists recommend upping food intake by only about 300 calories a day during the last two trimesters,
and excessive
weight gain by women during pregnancy is linked to
obesity in their kid
in their kids.
In a new animal study, University of Missouri researchers examined how the development of
obesity and insulin resistance contribute to bone - fracture risk
and whether exercise prevents
weight gain and diabetes
and protects bone health.
This pattern of
weight gain and insulin resistance parallels the development of
obesity and Type 2 diabetes
in humans, Hinton said.
Sleeping
in the dark
and consuming these foodstuffs could help control
weight gain and prevent cardiovascular diseases associated with
obesity and dyslipidemia.
The researchers determined that excessive pregnancy
weight gain was associated with greater overall
and abdominal body fat
in children
and obesity at age seven.
Mice that lost
weight and then
gained back more than they lost maintained an
obesity - type microbiome that affected biochemicals involved
in either burning or adding fat — suggesting interventions.
Maternal diet during pregnancy
and lactation may prime offspring for
weight gain and obesity later
in life, according to Penn State College of Medicine researchers, who looked at rats whose mothers consumed a high - fat diet
and found that the offspring's feeding controls
and feelings of fullness did not function normally.
The results provide further insight into the potential role of sugar - sweetened drinks — the largest single source of sugar
in the American diet —
in weight gain and obesity.»
«These new data are an important first step
in determining the amount of
weight gain (or loss) that is appropriate for women who enter pregnancy with
obesity — data which are sorely needed by obstetric providers to better serve their patients,» says Sharon Herring, MD, MPH, an expert in this area who is a member of The Obesity Society and Assistant Professor at Temple Univ
obesity — data which are sorely needed by obstetric providers to better serve their patients,» says Sharon Herring, MD, MPH, an expert
in this area who is a member of The
Obesity Society and Assistant Professor at Temple Univ
Obesity Society
and Assistant Professor at Temple University.
This new study provides data that have the potential to aid
in the development of national reference values for optimal gestational
weight gain for pregnancy among women who have higher classes of
obesity including class II
and class III.
A new Kaiser Permanente study, published
in Pediatric
Obesity, found that pre-pregnancy obesity and excessive weight gain during pregnancy was associated with an increased risk of the child becoming overweight at
Obesity, found that pre-pregnancy
obesity and excessive weight gain during pregnancy was associated with an increased risk of the child becoming overweight at
obesity and excessive
weight gain during pregnancy was associated with an increased risk of the child becoming overweight at age 2.
«Gestational
weight gain greater than the IOM recommendations has long - term implications for
weight - related health,» said Elizabeth Widen, PhD, RD, postdoctoral fellow at the New York
Obesity Research Center
in the Department of Medicine, Department of Epidemiology,
and Institute of Human Nutrition.
Unhealthy
weight gain in pregnancy has been linked with postpartum
weight retention but until now its long - term effects had been understudied
in low - income
and minority populations who are at high risk for
obesity.
Rapid
weight gain in an infant's first six months of life is a risk factor for child -
and adulthood
obesity, according to researchers.
«The high protein diet that has been used increasingly
in recent years to control
weight gain and obesity may have deleterious impacts on kidney health
in the long term,» said Kalantar - Zadeh, director of the Harold Simmons Center of Kidney Disease Research
and Epidemiology,
and chief of the Division of Nephrology
and Hypertension, UC Irvine School of Medicine.
Obesity in these mice resembles several important clinical features of human obesity such as weight gain and disturbance of metabolism, and this mouse model was ideal for unraveling any underlying biological mechanisms of pancreas cancer that are put in motion by o
Obesity in these mice resembles several important clinical features of human
obesity such as weight gain and disturbance of metabolism, and this mouse model was ideal for unraveling any underlying biological mechanisms of pancreas cancer that are put in motion by o
obesity such as
weight gain and disturbance of metabolism,
and this mouse model was ideal for unraveling any underlying biological mechanisms of pancreas cancer that are put
in motion by
obesityobesity.
«Although parents» perception that their children are overweight has been presumed to be important to management of childhood
obesity, recent studies have suggested the opposite; when a parent identifies a child as being overweight, that child is at increased risk of future
weight gain,» psychology researchers Eric Robinson (University of Liverpool)
and Angelina Sutin (Florida State University College of Medicine) write
in their paper.
The researchers tested their theory by orally administering a drug that inhibits DNA - PK
and found that,
in addition to preventing
weight gain in the mice, the inhibitor drug boosted mitochondrial content
in skeletal muscle, increased aerobic fitness
in obese
and middle aged mice,
and reduced the incidence of
obesity and type - 2 diabetes.
Despite the common perception that good - tasting food is unhealthy because it causes
obesity, new research from the Monell Center using a mouse model suggests that desirable taste
in and of itself does not lead to
weight gain.
The endocannabinoid system regulates energy homeostasis through G protein — coupled cannabinoid - 1 receptors5, 6 located
in the central nervous system and in various peripheral tissues, including adipose tissue, muscle, the gastrointestinal tract, and the liver.7 While peripheral cannabinoid - 1 receptor activation decreases adiponectin production in adipocytes, 8 central cannabinoid - 1 receptor activation in preclinical studies stimulates eating, decreases muscle, and stimulates hepatic and adipose tissue lipogenic pathways in animal models of obesity.9 In genetic and diet - induced obesity, rimonabant, a selective cannabinoid - 1 receptor blocker, reduces overactivation of the central8, 10 and peripheral11, 12 endocannabinoid system8, 10,13 and prevents weight gain and associated metabolic disorders, thus revealing a novel strategy for the treatment of obesity and related cardiometabolic disorder
in the central nervous system
and in various peripheral tissues, including adipose tissue, muscle, the gastrointestinal tract, and the liver.7 While peripheral cannabinoid - 1 receptor activation decreases adiponectin production in adipocytes, 8 central cannabinoid - 1 receptor activation in preclinical studies stimulates eating, decreases muscle, and stimulates hepatic and adipose tissue lipogenic pathways in animal models of obesity.9 In genetic and diet - induced obesity, rimonabant, a selective cannabinoid - 1 receptor blocker, reduces overactivation of the central8, 10 and peripheral11, 12 endocannabinoid system8, 10,13 and prevents weight gain and associated metabolic disorders, thus revealing a novel strategy for the treatment of obesity and related cardiometabolic disorder
in various peripheral tissues, including adipose tissue, muscle, the gastrointestinal tract,
and the liver.7 While peripheral cannabinoid - 1 receptor activation decreases adiponectin production
in adipocytes, 8 central cannabinoid - 1 receptor activation in preclinical studies stimulates eating, decreases muscle, and stimulates hepatic and adipose tissue lipogenic pathways in animal models of obesity.9 In genetic and diet - induced obesity, rimonabant, a selective cannabinoid - 1 receptor blocker, reduces overactivation of the central8, 10 and peripheral11, 12 endocannabinoid system8, 10,13 and prevents weight gain and associated metabolic disorders, thus revealing a novel strategy for the treatment of obesity and related cardiometabolic disorder
in adipocytes, 8 central cannabinoid - 1 receptor activation
in preclinical studies stimulates eating, decreases muscle, and stimulates hepatic and adipose tissue lipogenic pathways in animal models of obesity.9 In genetic and diet - induced obesity, rimonabant, a selective cannabinoid - 1 receptor blocker, reduces overactivation of the central8, 10 and peripheral11, 12 endocannabinoid system8, 10,13 and prevents weight gain and associated metabolic disorders, thus revealing a novel strategy for the treatment of obesity and related cardiometabolic disorder
in preclinical studies stimulates eating, decreases muscle,
and stimulates hepatic
and adipose tissue lipogenic pathways
in animal models of obesity.9 In genetic and diet - induced obesity, rimonabant, a selective cannabinoid - 1 receptor blocker, reduces overactivation of the central8, 10 and peripheral11, 12 endocannabinoid system8, 10,13 and prevents weight gain and associated metabolic disorders, thus revealing a novel strategy for the treatment of obesity and related cardiometabolic disorder
in animal models of
obesity.9
In genetic and diet - induced obesity, rimonabant, a selective cannabinoid - 1 receptor blocker, reduces overactivation of the central8, 10 and peripheral11, 12 endocannabinoid system8, 10,13 and prevents weight gain and associated metabolic disorders, thus revealing a novel strategy for the treatment of obesity and related cardiometabolic disorder
In genetic
and diet - induced
obesity, rimonabant, a selective cannabinoid - 1 receptor blocker, reduces overactivation of the central8, 10
and peripheral11, 12 endocannabinoid system8, 10,13
and prevents
weight gain and associated metabolic disorders, thus revealing a novel strategy for the treatment of
obesity and related cardiometabolic disorders.
What's known is that rapid
weight gain or
obesity,
and standing for long periods of time can worsen any vein issues that exist;
and getting regular exercise may help prevent vascular problems from arising
in the first place.
Research suggests a strong link between excess sugar intake,
weight gain,
and obesity,
and beverages are the largest source of added sugar
in the American diet, says Brownell, who is also a professor of psychology, epidemiology,
and public health.
So while monitoring
and curbing excess
weight gain in pregnancy is important,
obesity «may have affected the mother's cardiovascular
and other vital systems long before [the woman] becomes pregnant,» said Rabin.
Not only can
obesity itself trigger inflammation; so can some of the the eating behaviors that lead to
weight gain in the first place — like high - sugar
and high - fat diets.
And although a 2008 study discovered that eating out doesn't necessarily cause
weight gain, people who live
in neighborhoods with more fast - food restaurants have a higher risk of
obesity than individuals who have a higher per capita of sit - down options.
Sugar: * suppresses your immune system, LINKED TO CANCERS * promotes
weight gain and obesity * disrupts the mineral balance (causing stress
in the body) * contributes to depression, anxiety
and mood swings * promotes gut dysbiosis or overgrowth of bad bacteria
in the gut, such as candida * contributes to insulin resistance
and diabetes * can cause hormonal imbalances * increases risk for Crohn's disease
and ulcerative colitis * disrupt the body's pH balance * can cause hypoglycemia * will reduce good HDL cholesterol
and increase bad LDL cholesterol * prohibits
weight loss
So,
weight gain in menopause
and PCOS, both low E conditions, further supports the overall link between low estrogen
and obesity.
Berkemeyer S. Acid - base balance
and weight gain: are there crucial links via protein
and organic acids
in understanding
obesity?
Of course caving
in to too many cravings
and overdoing our calorie intake over time contributes to
weight gain and obesity.
Meanwhile, high protein definitely causes
obesity in infants — the
weight gain is not muscle
and bone.
This will not only contribute to your
weight gain, but also increase your risk of many chronic illnesses, as leptin resistance plays a significant, if not primary, role
in heart disease,
obesity, diabetes, osteoporosis, autoimmune diseases, reproductive disorders,
and perhaps the rate of aging itself.
Changes
in gut bacteria have even been linked to
weight gain and obesity.
Objective: We investigated the prevalence of RLPV
and BRR
in research about the proposition that skipping breakfast causes
weight gain, which is called the proposed effect of breakfast on
obesity (PEBO)
in this article.
After all, I've spent the last 60 + blog posts arguing that insulin
and insulin resistance are the key factors
in obesity and weight gain.