Not exact matches
Three groups of middle - aged
mice (about a year old) were studied: one group ate a
normal diet,
in which fewer than 30 percent of calories came from fat,
while two others were fed high - calorie diets
in which 60 percent of the calories came from fat.
They destroyed the T cells
in 12
mice, five of which received marrow cells from
normal mice while seven received marrow from
mice with a defective Fas - ligand gene.
The hind paws of the
normal mice became hypersensitive and blistered
in response to the UVB exposure,
while those of the mutant
mice showed little sensitization and tissue injury.
Photoswitches inserted into retinal ganglion cells (RGC) of blind
mice produce much less variety of response (all evenly red means the cells fire at the same time),
while blind
mice with photoswitches inserted into bipolar cells (ON - BC driven) exhibit much more variety
in their retinal response to light, closer to that of
normal mice.
Some fell into a pattern of two to three separate cycles of sleep and activity per day,
in contrast to the single daily cycle found
in normal mice,
while others» rhythms were completely disorganized, Blackshaw says.
To test the effect of the change, Tsien and colleagues at the East China
Normal University
in Shanghai gave
mice a slight shock
in a training chamber
while playing a loud tone.
With thoughts of a jolt fresh
in their brain,
mice with
normal levels of α - CaMKII froze up when they returned to the chamber an hour later,
while mice with boosted levels remained calm.
The genetically altered
mice, however, had the same reaction when placed
in the altered cages,
while their
normal compeers showed nary a trace of fear.
In this study, the researchers demonstrated that when normal mice were given kynurenine, they displayed depressive behaviour, while mice with increased levels of PGC - 1a1 in muscle were not affecte
In this study, the researchers demonstrated that when
normal mice were given kynurenine, they displayed depressive behaviour,
while mice with increased levels of PGC - 1a1
in muscle were not affecte
in muscle were not affected.
At the advanced age of 20 to 22 months (roughly equivalent to 100 years
in humans), the Bax - deficient
mice still maintained hundreds of follicles
while normal mice had none.
The reason for Frizzled6
mice's messy hair soon became obvious: The mutant
mice are born with hair follicles that point
in random directions,
while normal mice have follicles pointing more or less
in the direction needed for the hair to lay flat.
It should be noted, however, that
while a study on senescent cell ablation
in genetically
normal mice would provide at least some evidence on the effect of senescent cells (and their ablation) on promoting cancer, even such a study would likely show less effect than could be anticipated
in a large mammal model, since even normally - aging
mice rarely suffer metastatic disease to the extent of aging humans, as sheer primary tumor volume is generally sufficient to be fatal to
mice.
Mice exposed to normal TB live for 161 days, while mice infected by bacteria that lack sigF lived for 246 days, according to findings that were reported in Infection and Immun
Mice exposed to
normal TB live for 161 days,
while mice infected by bacteria that lack sigF lived for 246 days, according to findings that were reported in Infection and Immun
mice infected by bacteria that lack sigF lived for 246 days, according to findings that were reported
in Infection and Immunity.
Skeletal analysis of the
mice deficient
in MT1 or MT2 melatonin receptors showed a low bone mass
in MT2 - / -
mice while MT1 - / -
mice had a
normal bone mass compared to the WT
mice.
Indeed, the inactivated form of GSK3β, pS9 - GSK3β, was readily detected by immunohistochemistry
in caErbb2 - initiated early lesions
in parous
mice while it was not found
in normal mammary cells (Figure 3E).
* Oddly enough, however, the experiments with fresh pancreatic islets and fat cells taken from the
mice suggested that the positive effects of uncarboxylated osteocalcin on insulin only occurred at concentrations extremely low compared to those of undercarboxylated osteocalcin normally present
in mice,
while the effects on adiponectin and energy expenditure only occurred at
normal to high concentrations.
this was the one thing with move that seemed to stand on its own... I don't mind the idea of HD wii sports either, as long as it really is 1:1... that was my only real complaint with the wii when it released... there was motion control, but it was gimmicky and registered «wiggles» into canned animations... not to mention the gamecube visuals... still not sold on Move though... for me to really want one, I want to see what they are doing with shooters... Socom 4 and killzone 3 could be very special for core gamers and motion controls if they are done right... if you can aim on screen
in true 1:1 fashion
while sitting comfortably at a «
normal» gaming distance... it could rearrange how I play first person shooters on a console... developers are saying the Move has input latency of 21ms, which is roughly half of a DS3... and second only to a wired
mouse / keyboard... need to see how it works though, as it is not always that simple... just saying that if it does what its supposed to... it could end up being the answer to shooters on a console... as much as I like playing shooters with 2 sticks... I can't argue that I miss the days of a
mouse and keyboard (as well as PC being the only platform to get the best shooters on... no longer the case by any means)... but with a first person shooter, there is no wiggle room... pun intended... it has to register every mm of movement on screen... and do it quickly... not sure if it can yet...