These guys sit in front of their sets and the gamma rays eat
the white cells of their brains out!
Not exact matches
My counterview proposes that we think
of the region
of the soul in its relation to the regions
of both the
brain cells, and the occasions in the interstices
of the
brain as
White - head thinks
of the
cell in its relation to the molecules and empty spaces within it.
Did it started off as a mass
of white ball, then it slowly started developing nerves, retina, a cornea, and essentially a complex apparatus for capturing light and transmitting it via nerve
cells to the
brain?
A study shows, for the first time, how these functional impairments arise: Social isolation during early life prevents the
cells that make up the
brain's
white matter from maturing and producing the right amount
of myelin, the fatty «insulation» on nerve fibers that helps them transmit long - distance messages within the
brain.
Specifically, they drew RNA from the hippocampus, which is the part
of the
brain that helps regulate learning and memory, and from leukocytes,
white blood
cells that play a key role in the immune system.
Furthermore,
brain imaging data for these very elderly animals shows a slight loss
of grey matter (neuronal
cell bodies), an effect that the researchers have not yet explained, as well as significantly slowed atrophy
of white matter (the neuronal fibers connecting different areas
of the
brain).
Myelin is a fatty microstructure
of the
brain's
white matter that allows electrical information between
brain cells to travel faster.
Small amounts
of glycogen are found in the kidneys, and even smaller amounts in certain glial
cells in the
brain and
white blood
cells.
Kipnis and his colleagues had previously shown that a type
of white blood
cell called a T
cell (shown above) in the meninges is associated with significant influence on cognition and hence were curious about the role
of meningeal immunity on
brain function.
The findings, published today in the journal
Cell, give new insights into how the
brain regulates body fat and may lead to more effective ways to lose weight and prevent obesity by promoting the conversion
of white fat to brown fat.
Surprisingly, methamphetamine addicts»
brains were about 10 percent larger than normal
brains, apparently due to inflammation
of the
brain's
white matter — the nerve
cells that link the different thinking centers
of the organ.
In neurons
of DIXDC1 mutant mice (center) the dendrites — neural antennae that receive input from other
brain cells — have fewer
of the dendritic spines (
white with red arrows)-- the receiving half
of most synaptic inputs — compared to dendrites in wild type mice (left).
These investigations take advantage
of white blood
cells, monocytes, and macrophages that can target an inflammation site, including inside the
brain.
Then they examined the density — the concentration
of functioning
cells — in the gray matter and the
white matter
of each subject's
brain.
Instead, the
brain is protected by the blood -
brain barrier, a highly selective filtration system which keeps out invaders and the army
of patrolling
white blood
cells.
«This study suggests that amyloid deposition in the gray matter affects the associated
white matter connections, which are essential for conducting messages across the billions
of nerve
cells in the
brain, allowing for all aspects
of mental function.»
White matter consists
of large bundles
of nerve
cells that connect different regions
of the
brain and enable communication between them.
The liver then instructs
white blood
cells to go to the site
of injury in the
brain.
Twenty - four hours after the injection, the researchers saw large numbers
of immune system
white blood
cells in tissue samples
of the rodent
brains near the site
of injury
of those mice injected with the cytokine IL - 1b, but not in the
brain tissue
of the control group
of mice.
These problems are caused by a type
of white blood
cells called T
cells that, after becoming activated, find their way into the
brain and attack the protective covering — myelin —
of neurons in the
brain and spinal cord, causing inflammation and damage to the central nervous system.
If the approach also works with human
cells, it could eventually lead to
cell therapies for diseases like inherited leukodystrophies — disorders
of the
brain's
white matter — and multiple sclerosis, as well as spinal cord injuries.
(E) Retinal ganglion
cell axons exit the eye as the optic nerve (
white arrow), pass under the
brain and out
of view then reappear on the contralateral side (arrowhead).
Health improvement (allowing to post - pone / escape the diseases and thus live, healthier / disease - free longer, but not above human MLSP
of around 122 years; thus these therapies do not affect epigenetic aging whatsoever, they are degenerative aging problems not regular healthy aging problem (except OncoSENS - only when you Already Have Cancer - which cancer increases epigenetic aging, but cancer removal thus does not change anything / makes no difference about what happens in the other
cells / about what happens in the normal epigenetic «aging» course in Normal non-cancerous healthy cells) Although there is not such thing as «healthy aging» all aging in «unhealthy» (as seen from elders who are «healthy enough» who show much damage), it's just «tolerable / liveable» enough (in terms of damage accumulating) that it does not affect their quality of life (enough yet), that is «healthy aging»: ApoptoSENS - Clearing Senescent Cells (this will have great impact to reduce diseases, the largest one, since it's all inflammation fueled by the inflammation secretory phenotype (SASP) of these senescent cells) AmyloSENS - Dissolving the Plaques (this will allow humans to evade Alzheimer's, Parkinsons and general brain degenerescence, allowing quite a boost; making people much more easily reach the big 100 - since the brain is causal to how long we live; keeping brain amyloid - free and keeping our memories / neuron sharp / means longer LongTerm Potentiation - means longer brain function means longer heavy brain mass (gray matter / white matter retention seen in «sharp - witted» Centenarians who show are younger brain for their age), and both are correlated to M
cells / about what happens in the normal epigenetic «aging» course in Normal non-cancerous healthy
cells) Although there is not such thing as «healthy aging» all aging in «unhealthy» (as seen from elders who are «healthy enough» who show much damage), it's just «tolerable / liveable» enough (in terms of damage accumulating) that it does not affect their quality of life (enough yet), that is «healthy aging»: ApoptoSENS - Clearing Senescent Cells (this will have great impact to reduce diseases, the largest one, since it's all inflammation fueled by the inflammation secretory phenotype (SASP) of these senescent cells) AmyloSENS - Dissolving the Plaques (this will allow humans to evade Alzheimer's, Parkinsons and general brain degenerescence, allowing quite a boost; making people much more easily reach the big 100 - since the brain is causal to how long we live; keeping brain amyloid - free and keeping our memories / neuron sharp / means longer LongTerm Potentiation - means longer brain function means longer heavy brain mass (gray matter / white matter retention seen in «sharp - witted» Centenarians who show are younger brain for their age), and both are correlated to M
cells) Although there is not such thing as «healthy aging» all aging in «unhealthy» (as seen from elders who are «healthy enough» who show much damage), it's just «tolerable / liveable» enough (in terms
of damage accumulating) that it does not affect their quality
of life (enough yet), that is «healthy aging»: ApoptoSENS - Clearing Senescent
Cells (this will have great impact to reduce diseases, the largest one, since it's all inflammation fueled by the inflammation secretory phenotype (SASP) of these senescent cells) AmyloSENS - Dissolving the Plaques (this will allow humans to evade Alzheimer's, Parkinsons and general brain degenerescence, allowing quite a boost; making people much more easily reach the big 100 - since the brain is causal to how long we live; keeping brain amyloid - free and keeping our memories / neuron sharp / means longer LongTerm Potentiation - means longer brain function means longer heavy brain mass (gray matter / white matter retention seen in «sharp - witted» Centenarians who show are younger brain for their age), and both are correlated to M
Cells (this will have great impact to reduce diseases, the largest one, since it's all inflammation fueled by the inflammation secretory phenotype (SASP)
of these senescent
cells) AmyloSENS - Dissolving the Plaques (this will allow humans to evade Alzheimer's, Parkinsons and general brain degenerescence, allowing quite a boost; making people much more easily reach the big 100 - since the brain is causal to how long we live; keeping brain amyloid - free and keeping our memories / neuron sharp / means longer LongTerm Potentiation - means longer brain function means longer heavy brain mass (gray matter / white matter retention seen in «sharp - witted» Centenarians who show are younger brain for their age), and both are correlated to M
cells) AmyloSENS - Dissolving the Plaques (this will allow humans to evade Alzheimer's, Parkinsons and general
brain degenerescence, allowing quite a boost; making people much more easily reach the big 100 - since the
brain is causal to how long we live; keeping
brain amyloid - free and keeping our memories / neuron sharp / means longer LongTerm Potentiation - means longer
brain function means longer heavy
brain mass (gray matter /
white matter retention seen in «sharp - witted» Centenarians who show are younger
brain for their age), and both are correlated to MLSP).
The Human Connectome Project, which is an international effort to map the connectomes
of 1,000 people on a macro scale — mostly just the
white matter, or active myelinated (insulated) nerve
cell bundles — using magnetic resonance imaging, this week announced its finding that
brain wiring patterns correlate with behavioral and demographic traits.
December 21, 2016 — Noteworthy NIH advances in basic research include an expanded map
of the human
brain, nanoparticles that convert
white fat to calorie - burning brown fat, and a 3 - D
cell culture model
of the placenta.
Using a mouse model for this disease, which in humans involves the destruction
of white matter in the
brain, a research team led by Albee Messing, director
of the UW — Madison Waisman Center, found that a protein behind the symptoms
of the disease, called GFAP, is broken down more rapidly in the body than researchers previously found in
cell culture studies.
He then describes a few more problems: organoids don't display
white matter (a prominent component
of human
brains), lack some
cells types and don't have sensory input.
According to an article in the NY Times, nerves were found that connect the
brain with the spleen and thymus, organs used in immune responses, and it was established that nerve
cells could affect the activity
of infection - fighting
white blood
cells.
One is Globoid
Cell Leukodystrophy (Krabbe's disease), a
brain disorder where the
white matter
of the
brain breaks down.
ACT - activated clotting time (bleeding disorders) ACTH - adrenocorticotropic hormone (adrenal gland function) Ag - antigen test for proteins specific to a disease causing organism or virus Alb - albumin (liver, kidney and intestinal disorders) Alk - Phos, ALP alkaline phosphatase (liver and adrenal disorders) Allergy Testing intradermal or blood antibody test for allergen hypersensitivity ALT - alanine aminotransferase (liver disorder) Amyl - amylase enzyme — non specific (pancreatitis) ANA - antinuclear antibody (systemic lupus erythematosus) Anaplasmosis Anaplasma spp. (tick - borne rickettsial disease) APTT - activated partial thromboplastin time (blood clotting ability) AST - aspartate aminotransferase (muscle and liver disorders) Band band
cell — type
of white blood
cell Baso basophil — type
of white blood
cell Bile Acids digestive acids produced in the liver and stored in the gall bladder (liver function) Bili bilirubin (bile pigment responsible for jaundice from liver disease or RBC destruction) BP - blood pressure measurement BUN - blood urea nitrogen (kidney and liver function) Bx biopsy C & S aerobic / anaerobic bacterial culture and antibiotic sensitivity test (infection, drug selection) Ca +2 calcium ion — unbound calcium (parathyroid gland function) CBC - complete blood count (all circulating
cells) Chol cholesterol (liver, thyroid disorders) CK, CPK creatine [phospho] kinase (muscle disease, heart disease) Cl - chloride ion — unbound chloride (hydration, blood pH) CO2 - carbon dioxide (blood pH) Contrast Radiograph x-ray image using injected radiopaque contrast media Cortisol hormone produced by the adrenal glands (adrenal gland function) Coomb's anti- red blood
cell antibody test (immune - mediated hemolytic anemia) Crea creatinine (kidney function) CRT - capillary refill time (blood pressure, tissue perfusion) DTM - dermatophyte test medium (ringworm — dermatophytosis) EEG - electroencephalogram (
brain function, epilepsy) Ehrlichia Ehrlichia spp. (tick - borne rickettsial disease) EKG, ECG - electrok [c] ardiogram (electrical heart activity, heart arryhthmia) Eos eosinophil — type
of white blood
cell Fecal, flotation, direct intestinal parasite exam FeLV Feline Leukemia Virus test FIA Feline Infectious Anemia: aka Feline Hemotrophic Mycoplasma, Haemobartonella felis test FIV Feline Immunodeficiency Virus test Fluorescein Stain fluorescein stain uptake
of cornea (corneal ulceration) fT4, fT4ed, freeT4ed thyroxine hormone unbound by protein measured by equilibrium dialysis (thyroid function) GGT gamma - glutamyltranferase (liver disorders) Glob globulin (liver, immune system) Glu blood or urine glucose (diabetes mellitus) Gran granulocytes — subgroup
of white blood
cells Hb, Hgb hemoglobin — iron rich protein bound to red blood
cells that carries oxygen (anemia, red
cell mass) HCO3 - bicarbonate ion (blood pH) HCT, PCV, MHCT hematocrit, packed -
cell volume, microhematocrit (hemoconcentration, dehydration, anemia) K + potassium ion — unbound potassium (kidney disorders, adrenal gland disorders) Lipa lipase enzyme — non specific (pancreatitis) LYME Borrelia spp. (tick - borne rickettsial disease) Lymph lymphocyte — type
of white blood
cell MCHC mean corpuscular hemoglobin concentration (anemia, iron deficiency) MCV mean corpuscular volume — average red
cell size (anemia, iron deficiency) Mg +2 magnesium ion — unbound magnesium (diabetes, parathyroid function, malnutrition) MHCT, HCT, PCV microhematocrit, hematocrit, packed -
cell volume (hemoconcentration, dehydration, anemia) MIC minimum inhibitory concentration — part
of the C&S that determines antimicrobial selection Mono monocyte — type
of white blood
cell MRI magnetic resonance imaging (advanced tissue imaging) Na + sodium ion — unbound sodium (dehydration, adrenal gland disease) nRBC nucleated red blood
cell — immature red blood
cell (bone marrow damage, lead toxicity) PCV, HCT, MHCT packed -
cell volume, hematocrit, microhematocrit (hemoconcentration, dehydration, anemia) PE physical examination pH urine pH (urinary tract infection, urolithiasis) Phos phosphorus (kidney disorders, ketoacidosis, parathyroid function) PLI pancreatic lipase immunoreactivity (pancreatitis) PLT platelet —
cells involved in clotting (bleeding disorders) PT prothrombin time (bleeding disorders) PTH parathyroid hormone, parathormone (parathyroid function) Radiograph x-ray image RBC red blood
cell count (anemia) REL Rocky Mountain Spotted Fever / Ehrlichia / Lyme combination test Retic reticulocyte — immature red blood
cell (regenerative vs. non-regenerative anemia) RMSF Rocky Mountain Spotted Fever SAP serum alkaline phosphatase (liver disorders) Schirmer Tear Test tear production test (keratoconjunctivitis sicca — dry eye,) Seg segmented neutrophil — type
of white blood
cell USG Urine specific gravity (urine concentration, kidney function) spec cPL specific canine pancreatic lipase (pancreatitis)-- replaces the PLI test spec fPL specific feline pancreatic lipase (pancreatitis)-- replaces the PLI test T4 thyroxine hormone — total (thyroid gland function) TLI trypsin - like immunoreactivity (exocrine pancreatic insufficiency) TP total protein (hydration, liver disorders) TPR temperature / pulse / respirations (physical exam vital signs) Trig triglycerides (fat metabolism, liver disorders) TSH thyroid stimulating hormone (thyroid gland function) UA urinalysis (kidney function, urinary tract infection, diabetes) Urine Cortisol - Crea Ratio urine cortisol - creatine ratio (screening test for adrenal gland disease) Urine Protein - Crea Ratio urine protein - creatinine ratio (kidney disorders) VWF VonWillebrands factor (bleeding disorder) WBC
white blood
cell count (infection, inflammation, bone marrow suppression)
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