Not exact matches
Writing
in Nature Neuroscience, David Rowitch at the University of California, San Francisco, and his colleagues point the way AXIN2 was expressed
in infants with
white -
matter brain
injuries.
«This is going to be important
in the future, both for multiple sclerosis and different types of newborn
white -
matter injury,» Fancy says.
Since most cognitive processes involve multiple parts of the brain working together,
injury to
white matter can impair the brain's communication network and may result
in cognitive problems.
Using neuropsychological testing to assess cognitive performance, the researchers found a relationship between the amount of
white matter injury and changes
in reaction time and the ability to switch between mental tasks.
The results showed that amyloid plaque levels
in the posterior cingulate cortex were related to the amount of
white matter damage, suggesting that
injury to the brain's wiring may be linked to the formation of amyloid plaques.
For the new study, researchers from the University of Pittsburgh Medical Center (UPMC)
in Pittsburgh used diffusion tensor imaging (DTI), an MRI technique that measures the integrity of
white matter — the brain's signal - transmitting nerve fibers — to see if
injuries to the nerves may be the root cause of these post-traumatic depression and anxiety symptoms.
«We now have
white matter tracts that allows for faster, more effective communication from one region
in the brain to another after
injury.»
Her work is designed to help people with traumatic brain
injury (TBI), an impairment caused by damage to critical
white matter in the brain.
Recent relevant publication: re authors of a recently published study, Genova HM, Rajagopalan V, Chiaravalloti N, Binder A, Deluca J, Lengenfelder J. Facial affect recognition linked to damage
in specific
white matter tracts
in traumatic brain
injury, Social Neuroscience, 2015; 10 (1): 27 - 34.
AST - OPC1, an oligodendrocyte progenitor cell population derived from human embryonic stem cells, has been shown
in preclinical testing
in animals and
in vitro to have three potentially reparative functions that address the complex pathologies observed
in demyelination disorders, such as spinal cord
injuries, and multiple neurodegenerative diseases, including multiple sclerosis and
white matter stroke.
A failure of normal astrocyte generation by CNS precursor cells has been discovered to be a consequence of the mutations that cause Vanishing
White Matter leukodystrophy [7], and dysfunction of astrocytes has also been suggested to be of importance
in models of amyotrophic lateral sclerosis [8], forebrain ischemic
injury [9], epileptic seizures [10], Huntington's disease [11], tuberous sclerosis [12] and Rett syndrome [13].
Migration of hGDAs
in white matter was more extensive than
in gray
matter, with both types of hGDAs showing extensive migration both rostral and caudal to the
injury site, with maximum rostral / caudal migration distances of 3.24 mm / 3.96 mm recorded for hGDAsCNTF and 2.52 mm / 2.16 mm recorded for hGDAsBMP respectively.
Montaged images of NeuN immuno - histochemistry at the C5 spinal level of normal (A) and untreated injured (control) spinal cords (B) show that the unilateral DLF transection
injury causes loss of NeuN + neurons
in multiple spinal cord laminae adjacent to the transected
white matter.
John B.
White Jr. has developed a diverse litigation practice, ranging from personal
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matters in both State and Federal Court.
««Daniel
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